Autoantibodies Targeting AT
Autoantibodies
/ pharmacology
Blood Coagulation
/ drug effects
Cell Proliferation
/ drug effects
Endothelial Cells
/ cytology
Humans
Immunoglobulin G
/ metabolism
MAP Kinase Signaling System
/ drug effects
Models, Biological
Promoter Regions, Genetic
/ genetics
Protein Binding
/ drug effects
Proto-Oncogene Protein c-ets-1
/ metabolism
Receptor, Angiotensin, Type 1
/ metabolism
Receptor, Endothelin A
/ metabolism
Thromboplastin
/ metabolism
angiotensin
autoantibodies
coagulation
endothelin-1
renal crisis
renin–angiotensin system
systemic sclerosis
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
27 Dec 2021
27 Dec 2021
Historique:
received:
23
11
2021
revised:
20
12
2021
accepted:
23
12
2021
entrez:
11
1
2022
pubmed:
12
1
2022
medline:
2
2
2022
Statut:
epublish
Résumé
Scleroderma renal crisis (SRC) is an acute life-threatening manifestation of systemic sclerosis (SSc) caused by obliterative vasculopathy and thrombotic microangiopathy. Evidence suggests a pathogenic role of immunoglobulin G (IgG) targeting G-protein coupled receptors (GPCR). We therefore dissected SRC-associated vascular obliteration and investigated the specific effects of patient-derived IgG directed against angiotensin II type 1 (AT
Identifiants
pubmed: 35008670
pii: ijms23010244
doi: 10.3390/ijms23010244
pmc: PMC8745726
pii:
doi:
Substances chimiques
Autoantibodies
0
Immunoglobulin G
0
Proto-Oncogene Protein c-ets-1
0
Receptor, Angiotensin, Type 1
0
Receptor, Endothelin A
0
Thromboplastin
9035-58-9
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Deutsche Forschungsgemeinschaft
ID : 394046635
Organisme : Federal Ministry of Education and Research
ID : 01 GM 0310
Organisme : Deutsche Forschungsgemeinschaft
ID : 421152132
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