Construction and validation of safe Clostridium botulinum Group II surrogate strain producing inactive botulinum neurotoxin type E toxoid.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
02 02 2022
Historique:
received: 18 10 2021
accepted: 24 12 2021
entrez: 3 2 2022
pubmed: 4 2 2022
medline: 11 3 2022
Statut: epublish

Résumé

Botulinum neurotoxins (BoNTs), produced by the spore-forming bacterium Clostridium botulinum, cause botulism, a rare but fatal illness affecting humans and animals. Despite causing a life-threatening disease, BoNT is a multipurpose therapeutic. Nevertheless, as the most potent natural toxin, BoNT is classified as a Select Agent in the US, placing C. botulinum research under stringent governmental regulations. The extreme toxicity of BoNT, its impact on public safety, and its diverse therapeutic applications urge to devise safe solutions to expand C. botulinum research. Accordingly, we exploited CRISPR/Cas9-mediated genome editing to introduce inactivating point mutations into chromosomal bont/e gene of C. botulinum Beluga E. The resulting Beluga Ei strain displays unchanged physiology and produces inactive BoNT (BoNT/Ei) recognized in serological assays, but lacking biological activity detectable ex- and in vivo. Neither native single-chain, nor trypsinized di-chain form of BoNT/Ei show in vivo toxicity, even if isolated from Beluga Ei sub-cultured for 25 generations. Beluga Ei strain constitutes a safe alternative for the BoNT research necessary for public health risk management, the development of food preservation strategies, understanding toxinogenesis, and for structural BoNT studies. The example of Beluga Ei generation serves as template for future development of C. botulinum producing different inactive BoNT serotypes.

Identifiants

pubmed: 35110559
doi: 10.1038/s41598-022-05008-1
pii: 10.1038/s41598-022-05008-1
pmc: PMC8810926
doi:

Substances chimiques

CRISPR-Associated Protein 9 EC 3.1.-
Botulinum Toxins EC 3.4.24.69
botulinum toxin type E T579M564JY

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1790

Subventions

Organisme : NIAID NIH HHS
ID : R01 AI125704
Pays : United States

Informations de copyright

© 2022. The Author(s).

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Auteurs

Maria B Nowakowska (MB)

Department of Food Hygiene and Environmental Health, Faculty of Veterinary Medicine, University of Helsinki, Helsinki, Finland.

Katja Selby (K)

Department of Food Hygiene and Environmental Health, Faculty of Veterinary Medicine, University of Helsinki, Helsinki, Finland.

Adina Przykopanski (A)

Institut Für Toxikologie, Medizinische Hochschule Hannover, Hannover, Germany.

Maren Krüger (M)

Biological Toxins, Centre for Biological Threats and Special Pathogens, Robert Koch Institute, Berlin, Germany.

Nadja Krez (N)

Institut Für Toxikologie, Medizinische Hochschule Hannover, Hannover, Germany.

Brigitte G Dorner (BG)

Biological Toxins, Centre for Biological Threats and Special Pathogens, Robert Koch Institute, Berlin, Germany.

Martin B Dorner (MB)

Biological Toxins, Centre for Biological Threats and Special Pathogens, Robert Koch Institute, Berlin, Germany.

Rongsheng Jin (R)

Department of Physiology and Biophysics, University of California, Irvine, CA, USA.

Nigel P Minton (NP)

Clostridia Research Group, BBSRC/EPSRC Synthetic Biology Research Centre (SBRC), School of Life Sciences, Biodiscovery Institute, University of Nottingham, Nottingham, UK.

Andreas Rummel (A)

Institut Für Toxikologie, Medizinische Hochschule Hannover, Hannover, Germany.

Miia Lindström (M)

Department of Food Hygiene and Environmental Health, Faculty of Veterinary Medicine, University of Helsinki, Helsinki, Finland. miia.lindstrom@helsinki.fi.

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Classifications MeSH