ULK3-dependent activation of GLI1 promotes DNMT3A expression upon autophagy induction.
Autophagy
DNMT3A
GLI1
ULK3
transcription
Journal
Autophagy
ISSN: 1554-8635
Titre abrégé: Autophagy
Pays: United States
ID NLM: 101265188
Informations de publication
Date de publication:
12 2022
12 2022
Historique:
pubmed:
1
3
2022
medline:
19
11
2022
entrez:
28
2
2022
Statut:
ppublish
Résumé
Macroautophagy/autophagy is a tightly regulated catabolic process, which contributes at baseline level to cellular homeostasis, and upon its stimulation to the adaptive cellular response to intra- and extracellular stress stimuli. Decrease of autophagy activity is occurring upon aging and thought to contribute to age-related-diseases. Recently, we uncovered, upon autophagy induction, the role of
Identifiants
pubmed: 35226587
doi: 10.1080/15548627.2022.2039993
pmc: PMC9673947
doi:
Substances chimiques
Zinc Finger Protein GLI1
0
Protein Serine-Threonine Kinases
EC 2.7.11.1
RNA, Small Interfering
0
Microtubule-Associated Proteins
0
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
2769-2780Références
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