Ubiquitin and a charged loop regulate the ubiquitin E3 ligase activity of Ark2C.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
04 03 2022
Historique:
received: 29 06 2021
accepted: 03 02 2022
entrez: 5 3 2022
pubmed: 6 3 2022
medline: 14 4 2022
Statut: epublish

Résumé

A large family of E3 ligases that contain both substrate recruitment and RING domains confer specificity within the ubiquitylation cascade. Regulation of RING E3s depends on modulating their ability to stabilise the RING bound E2~ubiquitin conjugate in the activated (or closed) conformation. Here we report the structure of the Ark2C RING bound to both a regulatory ubiquitin molecule and an activated E2~ubiquitin conjugate. The structure shows that the RING domain and non-covalently bound ubiquitin molecule together make contacts that stabilise the activated conformation of the conjugate, revealing why ubiquitin is a key regulator of Ark2C activity. We also identify a charged loop N-terminal to the RING domain that enhances activity by interacting with both the regulatory ubiquitin and ubiquitin conjugated to the E2. In addition, the structure suggests how Lys48-linked ubiquitin chains might be assembled by Ark2C and UbcH5b. Together this study identifies features common to RING E3s, as well elements that are unique to Ark2C and related E3s, which enhance assembly of ubiquitin chains.

Identifiants

pubmed: 35246518
doi: 10.1038/s41467-022-28782-y
pii: 10.1038/s41467-022-28782-y
pmc: PMC8897509
doi:

Substances chimiques

Ubiquitin 0
Ubiquitin-Conjugating Enzymes EC 2.3.2.23
Ubiquitin-Protein Ligases EC 2.3.2.27

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1181

Informations de copyright

© 2022. The Author(s).

Références

Rape, M. Ubiquitylation at the crossroads of development and disease. Nat. Rev. Mol. Cell Biol. 19, 59–70 (2017).
pubmed: 28928488 doi: 10.1038/nrm.2017.83
Oh, E., Akopian, D. & Rape, M. Principles of ubiquitin-dependent signaling. Annu. Rev. Cell Dev. Biol. 34, 137–162 (2018).
pubmed: 30110556 doi: 10.1146/annurev-cellbio-100617-062802
Buetow, L. & Huang, D. T. Structural insights into the catalysis and regulation of E3 ubiquitin ligases. Nat. Rev. Mol. Cell Biol. 626–642 (2016).
Yau, R. & Rape, M. The increasing complexity of the ubiquitin code. Nat. Cell Biol. 18, 579–586 (2016).
pubmed: 27230526 doi: 10.1038/ncb3358
Zheng, N. & Shabek, N. Ubiquitin ligases: structure, function, and regulation. Annu. Rev. Biochem. 86, 129–157 (2017).
pubmed: 28375744 doi: 10.1146/annurev-biochem-060815-014922
Deshaies, R. J. & Joazeiro, C. A. P. RING domain E3 ubiquitin ligases. Annu. Rev. Biochem. 78, 399–434 (2009).
pubmed: 19489725 doi: 10.1146/annurev.biochem.78.101807.093809
Budhidarmo, R., Nakatani, Y. & Day, C. L. RINGs hold the key to ubiquitin transfer. Trends Biochem. Sci. 37, 58–65 (2012).
pubmed: 22154517 doi: 10.1016/j.tibs.2011.11.001
Dou, H., Buetow, L., Sibbet, G. J., Cameron, K. & Huang, D. T. Essentiality of a non-RING element in priming donor ubiquitin for catalysis by a monomeric E3. Nat. Struct. Mol. Biol. 20, 982–986 (2013).
pubmed: 23851457 pmcid: 4471106 doi: 10.1038/nsmb.2621
Middleton, A. J. et al. The activity of TRAF RING homo- and heterodimers is regulated by zinc finger 1. Nat. Commun. 8, 1788 (2017).
pubmed: 29176576 pmcid: 5702613 doi: 10.1038/s41467-017-01665-3
Plechanovova, A., Jaffray, E. G., Tatham, M. H., Naismith, J. H. & Hay, R. T. Structure of a RING E3 ligase and ubiquitin-loaded E2 primed for catalysis. Nature 489, 115–120 (2012).
pubmed: 22842904 pmcid: 3442243 doi: 10.1038/nature11376
Dou, H., Buetow, L., Sibbet, G. J., Cameron, K. & Huang, D. T. BIRC7–E2 ubiquitin conjugate structure reveals the mechanism of ubiquitin transfer by a RING dimer. Nat. Struct. Mol. Biol. 19, 876–883 (2012).
pubmed: 22902369 doi: 10.1038/nsmb.2379
Branigan, E., Penedo, J. C. & Hay, R. T. Ubiquitin transfer by a RING E3 ligase occurs from a closed E2~ubiquitin conformation. Nat. Commun. 11, 1–11 (2020).
doi: 10.1038/s41467-020-16666-y
Koinuma, D. et al. Arkadia amplifies TGF-β superfamily signalling through degradation of Smad7. EMBO J. 22, 6458–6470 (2003).
pubmed: 14657019 pmcid: 291827 doi: 10.1093/emboj/cdg632
Kelly, C. E. et al. Rnf165/Ark2C enhances BMP-Smad signaling to mediate motor axon extension. PLoS Biol. 11, e1001538 (2013).
pubmed: 23610558 pmcid: 3627648 doi: 10.1371/journal.pbio.1001538
Zhou, F. et al. Nuclear receptor NR4A1 promotes breast cancer invasion and metastasis by activating TGF-β; signalling. Nat. Commun. 5, 1–13 (2014).
doi: 10.1038/ncomms4388
Niederlander, C., Walsh, J. J., Episkopou, V. & Jones, C. M. Arkadia enhances nodal-related signalling to induce mesendoderm. Nature 410, 830–834 (2001).
pubmed: 11298453 doi: 10.1038/35071103
Mavrakis, K. J. et al. Arkadia enhances nodal/TGF-β signaling by coupling phospho-Smad2/3 activity and turnover. PLoS Biol. 5, 586–603 (2007).
doi: 10.1371/journal.pbio.0050067
Poulsen, S. L. et al. RNF111/Arkadia is a SUMO-targeted ubiquitin ligase that facilitates the DNA damage response. J. Cell Biol. 201, 797–807 (2013).
pubmed: 23751493 pmcid: 3678163 doi: 10.1083/jcb.201212075
van Cuijk, L. et al. SUMO and ubiquitin-dependent XPC exchange drives nucleotide excision repair. Nat. Commun. 6, 7499 (2015).
pubmed: 26151477 doi: 10.1038/ncomms8499
Erker, Y. et al. Arkadia, a novel SUMO-targeted ubiquitin ligase involved in PML degradation. Mol. Cell. Biol. 33, 2163–2177 (2013).
pubmed: 23530056 pmcid: 3648077 doi: 10.1128/MCB.01019-12
Sriramachandran, A. M. et al. Arkadia/RNF111 is a SUMO-targeted ubiquitin ligase with preference for substrates marked with SUMO1-capped SUMO2/3 chain. Nat. Commun. 10, 1–12 (2019).
doi: 10.1038/s41467-019-11549-3
Jansen, N. S. & Vertegaal, A. C. O. A chain of events: regulating target proteins by SUMO polymers. Trends Biochem. Sci. 46, 113–123 (2021).
pubmed: 33008689 doi: 10.1016/j.tibs.2020.09.002
Wright, J. D., Mace, P. D. & Day, C. L. Secondary ubiquitin-RING docking enhances Arkadia and Ark2C E3 ligase activity. Nat. Sruct. Mol. Biol. 23, 45–52 (2015).
doi: 10.1038/nsmb.3142
Kiss, L., Clift, D., Renner, N., Neuhaus, D. & James, L. C. RING domains act as both substrate and enzyme in a catalytic arrangement to drive self-anchored ubiquitination. Nat. Commun. 12, 1220 (2021).
pubmed: 33619271 pmcid: 7900206 doi: 10.1038/s41467-021-21443-6
Bosanac, I. et al. Modulation of K11-linkage formation by variable loop residues within UbcH5A. J. Mol. Biol. 408, 420–431 (2011).
pubmed: 21396940 doi: 10.1016/j.jmb.2011.03.011
David, Y., Ziv, T., Admon, A. & Navon, A. The E2 ubiquitin-conjugating enzymes direct polyubiquitination to preferred lysines. J. Biol. Chem. 285, 8595–8604 (2010).
pubmed: 20061386 pmcid: 2838281 doi: 10.1074/jbc.M109.089003
Friedland, G. D., Lakomek, N. A., Griesinger, C., Meiler, J. & Kortemme, T. A correspondence between solution-state dynamics of an individual protein and the sequence and conformational diversity of its family. PLoS Comput. Biol. 5, e1000393 (2009).
pubmed: 19478996 pmcid: 2682763 doi: 10.1371/journal.pcbi.1000393
Rennie, M. L., Chaugule, V. K. & Walden, H. Modes of allosteric regulation of the ubiquitination machinery. Curr. Opin. Struct. Biol. 62, 189–196 (2020).
pubmed: 32305021 doi: 10.1016/j.sbi.2020.02.003
Brzovic, P. S., Lissounov, A., Christensen, D. E., Hoyt, D. W. & Klevit, R. E. A UbcH5/ubiquitin noncovalent complex is required for processive BRCA1-directed ubiquitination. Mol. Cell 21, 873–880 (2006).
pubmed: 16543155 doi: 10.1016/j.molcel.2006.02.008
Sakata, E. et al. Crystal structure of UbcH5b~ubiquitin intermediate: insight into the formation of the self-assembled E2~Ub Conjugates. Structure 18, 138–147 (2010).
pubmed: 20152160 doi: 10.1016/j.str.2009.11.007
Patel, A., Sibbet, G. J. & Huang, D. T. Structural insights into non-covalent ubiquitin activation of the cIAP1-UbcH5B approximately ubiquitin complex. J. Biol. Chem. 294, 1240 (2019).
pubmed: 30523153 doi: 10.1074/jbc.RA118.006045
Buetow, L. et al. Activation of a primed RING E3-E2-ubiquitin complex by non-covalent ubiquitin. Mol. Cell 58, 297–310 (2015).
Hibbert, R. G. & Sixma, T. K. Intrinsic flexibility of ubiquitin on proliferating cell nuclear antigen (PCNA) in translesion synthesis. J. Biol. Chem. 287, 39216–39223 (2012).
pubmed: 22989887 pmcid: 3493961 doi: 10.1074/jbc.M112.389890
Kiss, L. et al. A tri-ionic anchor mechanism drives Ube2N-specific recruitment and K63-chain ubiquitination in TRIM ligases. Nat. Commun. 10, 1–13 (2019).
doi: 10.1038/s41467-019-12388-y
Magnussen, H. M. et al. Structural basis for DNA damage-induced phosphoregulation of MDM2 RING domain. Nat. Commun. 11, 1–15 (2020).
doi: 10.1038/s41467-020-15783-y
Nomura, K. et al. Structural analysis of MDM2 RING separates degradation from regulation of p53 transcription activity. Nat. Struct. Mol. Biol. 62, 578–587 (2017).
doi: 10.1038/nsmb.3414
Middleton, A. J., Zhu, J. & Day, C. L. The RING domain of RING finger 12 efficiently builds degradative ubiquitin chains. J. Mol. Biol. 432, 3790 (2020).
pubmed: 32416094 doi: 10.1016/j.jmb.2020.05.001
Behera, A. P. et al. Structural insights into the nanomolar affinity of RING E3 ligase ZNRF1 for Ube2N and its functional implications. Biochem. J. 475, 1569–1582 (2018).
pubmed: 29626159 doi: 10.1042/BCJ20170909
Khago, D., Fucci, I. J. & Byrd, R. A. The role of conformational dynamics in the recognition and regulation of ubiquitination. Molecules 25, 5933 (2020).
pmcid: 7765195 doi: 10.3390/molecules25245933
Pruneda, J. N., Stoll, K. E., Bolton, L. J., Brzovic, P. S. & Klevit, R. E. Ubiquitin in motion: structural studies of the ubiquitin-conjugating enzyme∼ubiquitin conjugate. Biochemistry 50, 1624–1633 (2011).
pubmed: 21226485 doi: 10.1021/bi101913m
Soss, S. E., Klevit, R. E. & Chazin, W. J. Activation of UbcH5c∼Ub is the result of a shift in interdomain motions of the conjugate bound to U-box E3 ligase E4B. Biochemistry 52, 2991–2999 (2013).
pubmed: 23550736 doi: 10.1021/bi3015949
Ye, Y. & Rape, M. Building ubiquitin chains: E2 enzymes at work. Nat. Rev. Mol. Cell Biol. 10, 755–764 (2009).
pubmed: 19851334 pmcid: 3107738 doi: 10.1038/nrm2780
Stewart, M. D., Ritterhoff, T., Klevit, R. E. & Brzovic, P. S. E2 enzymes: more than just middle men. Cell Res. 26, 423–440 (2016).
pubmed: 27002219 pmcid: 4822130 doi: 10.1038/cr.2016.35
Eddins, M. J., Carlile, C. M., Gomez, K. M., Pickart, C. M. & Wolberger, C. Mms2-Ubc13 covalently bound to ubiquitin reveals the structural basis of linkage-specific polyubiquitin chain formation. Nat. Struct. Mol. Biol. 13, 915–920 (2006).
pubmed: 16980971 doi: 10.1038/nsmb1148
Middleton, A. J. & Day, C. L. The molecular basis of lysine 48 ubiquitin chain synthesis by Ube2K. Sci. Rep. 5, 16793 (2015).
pubmed: 26592444 pmcid: 4655369 doi: 10.1038/srep16793
Hill, S., Harrison, J. S., Lewis, S. M., Kuhlman, B. & Kleiger, G. Mechanism of lysine 48 selectivity during polyubiquitin chain formation by the Ube2R1/2 ubiquitin-conjugating enzyme. Mol. Cell Biol. 36, 1720–1732 (2016).
pubmed: 27044868 pmcid: 4959314 doi: 10.1128/MCB.00097-16
Middleton, A. J., Budhidarmo, R. & Day, C. L. Use of E2~ubiquitin conjugates for the characterization of ubiquitin transfer by RING E3 ligases such as the inhibitor of apoptosis proteins. Methods Enzymol. 545, 243–263 (2014).
pubmed: 25065893 doi: 10.1016/B978-0-12-801430-1.00010-X
Cowieson, N. P. et al. MX1: a bending-magnet crystallography beamline serving both chemical and macromolecular crystallography communities at the Australian Synchrotron. J. Synchrotron Radiat. 22, 187–190 (2015).
pubmed: 25537608 pmcid: 4294030 doi: 10.1107/S1600577514021717
Aragao, D. et al. MX2: a high-flux undulator microfocus beamline serving both the chemical and macromolecular crystallography communities at the Australian Synchrotron. J. Synchrotron Radiat. 25, 885–891 (2018).
pubmed: 29714201 pmcid: 5929359 doi: 10.1107/S1600577518003120
Kabsch, W. Xds. Acta Crystallogr. D Biol. Crystallogr. 66, 125–132 (2010).
pubmed: 20124692 pmcid: 2815665 doi: 10.1107/S0907444909047337
Evans, P. R. & Murshudov, G. N. How good are my data and what is the resolution? Acta Crystallogr. D Biol. Crystallogr. 69, 1204–1214 (2013).
pubmed: 23793146 pmcid: 3689523 doi: 10.1107/S0907444913000061
McCoy, A. J. et al. Phaser crystallographic software. J. Appl. Crystallogr. 40, 658–674 (2007).
pubmed: 19461840 pmcid: 2483472 doi: 10.1107/S0021889807021206
Adams, P. D. et al. PHENIX: a comprehensive Python-based system for macromolecular structure solution. Acta Crystallogr. D Biol. Crystallogr. 66, 213–221 (2010).
pubmed: 20124702 pmcid: 2815670 doi: 10.1107/S0907444909052925
Pettersen, E. F. et al. UCSF ChimeraX: structure visualization for researchers, educators, and developers. Protein Sci. 30, 70–82 (2021).
doi: 10.1002/pro.3943 pubmed: 32881101
Krissinel, E. & Henrick, K. Inference of macromolecular assemblies from crystalline state. J. Mol. Biol. 372, 774–797 (2007).
pubmed: 17681537 doi: 10.1016/j.jmb.2007.05.022
Gotze, M. et al. StavroX—a software for analyzing crosslinked products in protein interaction studies. J. Am. Soc. Mass Spectrom. 23, 76–87 (2012).
pubmed: 22038510 doi: 10.1007/s13361-011-0261-2

Auteurs

Andrej Paluda (A)

Biochemistry Department, School of Biomedical Sciences, University of Otago, Dunedin, 9054, New Zealand.
TMDU Advanced Research Institute, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo, 113-8510, Japan.

Adam J Middleton (AJ)

Biochemistry Department, School of Biomedical Sciences, University of Otago, Dunedin, 9054, New Zealand.

Claudia Rossig (C)

Biochemistry Department, School of Biomedical Sciences, University of Otago, Dunedin, 9054, New Zealand.

Peter D Mace (PD)

Biochemistry Department, School of Biomedical Sciences, University of Otago, Dunedin, 9054, New Zealand.

Catherine L Day (CL)

Biochemistry Department, School of Biomedical Sciences, University of Otago, Dunedin, 9054, New Zealand. catherine.day@otago.ac.nz.

Articles similaires

Glycine max Photoperiod Ubiquitin-Protein Ligases Flowers Gene Expression Regulation, Plant
Porphyromonas gingivalis MicroRNAs Humans Periodontitis Adhesins, Bacterial
Autophagy Humans Neoplasms Ubiquitination Animals
Humans Ferroptosis Stomach Neoplasms Cisplatin Mitophagy

Classifications MeSH