The synaptic scaffold protein MPP2 interacts with GABAA receptors at the periphery of the postsynaptic density of glutamatergic synapses.


Journal

PLoS biology
ISSN: 1545-7885
Titre abrégé: PLoS Biol
Pays: United States
ID NLM: 101183755

Informations de publication

Date de publication:
03 2022
Historique:
received: 01 11 2021
accepted: 02 12 2021
revised: 31 03 2022
pubmed: 22 3 2022
medline: 5 4 2022
entrez: 21 3 2022
Statut: epublish

Résumé

Recent advances in imaging technology have highlighted that scaffold proteins and receptors are arranged in subsynaptic nanodomains. The synaptic membrane-associated guanylate kinase (MAGUK) scaffold protein membrane protein palmitoylated 2 (MPP2) is a component of α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor-associated protein complexes and also binds to the synaptic cell adhesion molecule SynCAM 1. Using superresolution imaging, we show that-like SynCAM 1-MPP2 is situated at the periphery of the postsynaptic density (PSD). In order to explore MPP2-associated protein complexes, we used a quantitative comparative proteomics approach and identified multiple γ-aminobutyric acid (GABA)A receptor subunits among novel synaptic MPP2 interactors. In line with a scaffold function for MPP2 in the assembly and/or modulation of intact GABAA receptors, manipulating MPP2 expression had effects on inhibitory synaptic transmission. We further show that GABAA receptors are found together with MPP2 in a subset of dendritic spines and thus highlight MPP2 as a scaffold that serves as an adaptor molecule, linking peripheral synaptic elements critical for inhibitory regulation to central structures at the PSD of glutamatergic synapses.

Identifiants

pubmed: 35312684
doi: 10.1371/journal.pbio.3001503
pii: PBIOLOGY-D-21-02850
pmc: PMC8970474
doi:

Substances chimiques

Membrane Proteins 0
Receptors, AMPA 0
Receptors, GABA-A 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e3001503

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

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Auteurs

Bettina Schmerl (B)

Neuroscience Research Center, Charité-Universitätsmedizin Berlin, Germany.

Niclas Gimber (N)

Advanced Medical BioImaging Core Facility-AMBIO, Charité-Universitätsmedizin Berlin, Germany.

Benno Kuropka (B)

Institute of Chemistry and Biochemistry, Freie Universität Berlin, Germany.

Alexander Stumpf (A)

Neuroscience Research Center, Charité-Universitätsmedizin Berlin, Germany.

Jakob Rentsch (J)

Institute of Chemistry and Biochemistry, Freie Universität Berlin, Germany.

Stella-Amrei Kunde (SA)

Neuroscience Research Center, Charité-Universitätsmedizin Berlin, Germany.

Judith von Sivers (J)

Neuroscience Research Center, Charité-Universitätsmedizin Berlin, Germany.

Helge Ewers (H)

Institute of Chemistry and Biochemistry, Freie Universität Berlin, Germany.

Dietmar Schmitz (D)

Neuroscience Research Center, Charité-Universitätsmedizin Berlin, Germany.
German Center for Neurodegenerative Diseases (DZNE), Berlin, Germany.
Einstein Center for Neurosciences, Berlin, Germany.

Christian Freund (C)

Institute of Chemistry and Biochemistry, Freie Universität Berlin, Germany.

Jan Schmoranzer (J)

Advanced Medical BioImaging Core Facility-AMBIO, Charité-Universitätsmedizin Berlin, Germany.

Nils Rademacher (N)

Neuroscience Research Center, Charité-Universitätsmedizin Berlin, Germany.
German Center for Neurodegenerative Diseases (DZNE), Berlin, Germany.

Sarah A Shoichet (SA)

Neuroscience Research Center, Charité-Universitätsmedizin Berlin, Germany.
Einstein Center for Neurosciences, Berlin, Germany.

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Classifications MeSH