Promoting antigen escape from dendritic cell endosomes potentiates anti-tumoral immunity.

Accum anti-tumoral immunity antigen cross-presentation dendritic cells endosomes immune checkpoint inhibitors tumor-infiltrating lymphocytes

Journal

Cell reports. Medicine
ISSN: 2666-3791
Titre abrégé: Cell Rep Med
Pays: United States
ID NLM: 101766894

Informations de publication

Date de publication:
15 03 2022
Historique:
received: 11 06 2021
revised: 17 12 2021
accepted: 27 01 2022
entrez: 2 5 2022
pubmed: 3 5 2022
medline: 4 5 2022
Statut: epublish

Résumé

The cross-presenting capacity of dendritic cells (DCs) can be limited by non-specific degradation during endosome maturation. To bypass this limitation, we present in this study a new Accum-based formulation designed to promote endosome-to-cytosol escape. Treatment of primary DCs with Accum linked to the xenoantigen ovalbumin (OVA) triggers endosomal damages and enhances protein processing. Despite multiple challenges using ascending doses of tumor cells, DC prophylactic vaccination results in complete protection due to increased levels of effector CD4 and CD8 T cells as well as high production of pro-inflammatory mediators. When combined with anti-PD-1, therapeutic vaccination using both syngeneic and allogeneic Accum-OVA-pulsed DCs triggers potent anti-tumoral responses. The net outcome culminates in increased CD11c, CD8, and NK infiltration along with a high CD8/Treg ratio. These highly favorable therapeutic effects highlight the promising potential of Accum as a distinct and potent technology platform suitable for the design of next generation cell cancer vaccines.

Identifiants

pubmed: 35492876
doi: 10.1016/j.xcrm.2022.100534
pii: S2666-3791(22)00034-9
pmc: PMC9040180
doi:

Substances chimiques

Cancer Vaccines 0
Ovalbumin 9006-59-1

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

100534

Informations de copyright

© 2022 The Author(s).

Déclaration de conflit d'intérêts

S.B. and S.P. are current employees of Defence Therapeutics Inc. and declare competing financial interest.

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Auteurs

Jean-Pierre Bikorimana (JP)

Department of Microbiology, Infectiology and Immunology, Université de Montréal, Montréal, QC, Canada.

Natasha Salame (N)

Department of Biomedical Sciences, Université de Montréal, Montréal, QC, Canada.

Simon Beaudoin (S)

Research and Development Branch, Defence Therapeutics Inc., Vancouver, BC, Canada.

Mohammad Balood (M)

Department of Pharmacology and Physiology, Université de Montréal, Montréal, QC, Canada.

Théo Crosson (T)

Department of Pharmacology and Physiology, Université de Montréal, Montréal, QC, Canada.

Jamilah Abusarah (J)

Department of Pharmacology and Physiology, Université de Montréal, Montréal, QC, Canada.

Sebastien Talbot (S)

Department of Pharmacology and Physiology, Université de Montréal, Montréal, QC, Canada.

Raimar Löbenberg (R)

Department of Pharmacy and Pharmaceutical Sciences, University of Alberta, Edmonton, AB, Canada.

Sebastien Plouffe (S)

Research and Development Branch, Defence Therapeutics Inc., Vancouver, BC, Canada.

Moutih Rafei (M)

Department of Microbiology, Infectiology and Immunology, Université de Montréal, Montréal, QC, Canada.
Department of Pharmacology and Physiology, Université de Montréal, Montréal, QC, Canada.
Molecular Biology Program, Université de Montréal, Montréal, QC, Canada.

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Classifications MeSH