Extra Virgin Olive Oil Reduces Gut Permeability and Metabolic Endotoxemia in Diabetic Patients.

Glucagon-like peptide1(GLP1) Impaired fasting glucose (IFG) extra virgin olive oil (EVOO) gut permeability lipopolysaccharides (LPS) oleuropein zonulin

Journal

Nutrients
ISSN: 2072-6643
Titre abrégé: Nutrients
Pays: Switzerland
ID NLM: 101521595

Informations de publication

Date de publication:
21 May 2022
Historique:
received: 22 04 2022
revised: 17 05 2022
accepted: 19 05 2022
entrez: 28 5 2022
pubmed: 29 5 2022
medline: 1 6 2022
Statut: epublish

Résumé

Extra virgin olive oil (EVOO) improves post-prandial glycemia, but the underlying mechanism has not been fully elucidated. We tested the hypothesis that EVOO improves post-prandial glycemia by reducing gut permeability-derived low-grade endotoxemia. Serum levels of lipopolysaccharides (LPS), zonulin, a marker of gut permeability, glucose, insulin and glucagon-like peptide 1 (GLP1) were measured in 20 patients with impaired fasting glucose (IFG) and 20 healthy subjects (HS) matched for sex and age. The same variables were measured in IFG patients ( Compared to HS, IFG had higher levels of LPS and zonulin. In HS, meal intake was associated with a significant increase of blood glucose, insulin, and GLP1 with no changes of blood LPS and zonulin. Two hours after a meal intake containing EVOO, IFG patients showed a less significant increase of blood glucose, a more marked increase of blood insulin and GLP1 and a significant reduction of LPS and zonulin compared to IFG patients not given EVOO. Correlation analysis showed that LPS directly correlated with blood glucose and zonulin and inversely with blood insulin. Similar findings were detected in IFG patients given a chocolate added or without EVOO. Addition of EVOO to a Mediterranean diet or chocolate improves gut permeability and low-grade endotoxemia.

Sections du résumé

BACKGROUND BACKGROUND
Extra virgin olive oil (EVOO) improves post-prandial glycemia, but the underlying mechanism has not been fully elucidated. We tested the hypothesis that EVOO improves post-prandial glycemia by reducing gut permeability-derived low-grade endotoxemia.
METHODS METHODS
Serum levels of lipopolysaccharides (LPS), zonulin, a marker of gut permeability, glucose, insulin and glucagon-like peptide 1 (GLP1) were measured in 20 patients with impaired fasting glucose (IFG) and 20 healthy subjects (HS) matched for sex and age. The same variables were measured in IFG patients (
RESULTS RESULTS
Compared to HS, IFG had higher levels of LPS and zonulin. In HS, meal intake was associated with a significant increase of blood glucose, insulin, and GLP1 with no changes of blood LPS and zonulin. Two hours after a meal intake containing EVOO, IFG patients showed a less significant increase of blood glucose, a more marked increase of blood insulin and GLP1 and a significant reduction of LPS and zonulin compared to IFG patients not given EVOO. Correlation analysis showed that LPS directly correlated with blood glucose and zonulin and inversely with blood insulin. Similar findings were detected in IFG patients given a chocolate added or without EVOO.
CONCLUSION CONCLUSIONS
Addition of EVOO to a Mediterranean diet or chocolate improves gut permeability and low-grade endotoxemia.

Identifiants

pubmed: 35631294
pii: nu14102153
doi: 10.3390/nu14102153
pmc: PMC9145083
pii:
doi:

Substances chimiques

Blood Glucose 0
Insulin 0
Lipopolysaccharides 0
Olive Oil 0
Glucagon-Like Peptide 1 89750-14-1

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Références

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Auteurs

Simona Bartimoccia (S)

Department of Medical-Surgical Sciences and Biotechnologies, Sapienza University of Rome, Corso della Repubblica 79, 40100 Latina, Italy.

Vittoria Cammisotto (V)

Department of Clinical Internal, Anaesthesiological and Cardiovascular Sciences, Sapienza University of Rome, Viale del Policlinico, 155, 00161 Rome, Italy.

Cristina Nocella (C)

Department of Clinical Internal, Anaesthesiological and Cardiovascular Sciences, Sapienza University of Rome, Viale del Policlinico, 155, 00161 Rome, Italy.

Maria Del Ben (M)

Department of Clinical Internal, Anaesthesiological and Cardiovascular Sciences, Sapienza University of Rome, Viale del Policlinico, 155, 00161 Rome, Italy.

Alessandra D'Amico (A)

Department of Movement, Human and Health Sciences, University of Rome "Foro Italico", 00135 Rome, Italy.

Valentina Castellani (V)

Department of General Surgery and Surgical Speciality Paride Stefanini, Sapienza University of Rome, Viale del Policlinico, 155, 00161 Rome, Italy.

Francesco Baratta (F)

Department of Clinical Internal, Anaesthesiological and Cardiovascular Sciences, Sapienza University of Rome, Viale del Policlinico, 155, 00161 Rome, Italy.

Pasquale Pignatelli (P)

Department of Clinical Internal, Anaesthesiological and Cardiovascular Sciences, Sapienza University of Rome, Viale del Policlinico, 155, 00161 Rome, Italy.
Mediterranea Cardiocentro-Napoli, Via Orazio, 2, 80122 Naples, Italy.

Lorenzo Loffredo (L)

Department of Clinical Internal, Anaesthesiological and Cardiovascular Sciences, Sapienza University of Rome, Viale del Policlinico, 155, 00161 Rome, Italy.

Francesco Violi (F)

Mediterranea Cardiocentro-Napoli, Via Orazio, 2, 80122 Naples, Italy.

Roberto Carnevale (R)

Department of Medical-Surgical Sciences and Biotechnologies, Sapienza University of Rome, Corso della Repubblica 79, 40100 Latina, Italy.
Mediterranea Cardiocentro-Napoli, Via Orazio, 2, 80122 Naples, Italy.

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Classifications MeSH