Exercise, Mitohormesis, and Mitochondrial ORF of the 12S rRNA Type-C (MOTS-c).

Exercise Hormesis MOTS-c peptide, human Mitochondria Mitochondrial proteins Obesity

Journal

Diabetes & metabolism journal
ISSN: 2233-6087
Titre abrégé: Diabetes Metab J
Pays: Korea (South)
ID NLM: 101556588

Informations de publication

Date de publication:
05 2022
Historique:
received: 21 03 2022
accepted: 27 04 2022
entrez: 3 6 2022
pubmed: 4 6 2022
medline: 7 6 2022
Statut: ppublish

Résumé

Low levels of mitochondrial stress are beneficial for organismal health and survival through a process known as mitohormesis. Mitohormetic responses occur during or after exercise and may mediate some salutary effects of exercise on metabolism. Exercise-related mitohormesis involves reactive oxygen species production, mitochondrial unfolded protein response (UPRmt), and release of mitochondria-derived peptides (MDPs). MDPs are a group of small peptides encoded by mitochondrial DNA with beneficial metabolic effects. Among MDPs, mitochondrial ORF of the 12S rRNA type-c (MOTS-c) is the most associated with exercise. MOTS-c expression levels increase in skeletal muscles, systemic circulation, and the hypothalamus upon exercise. Systemic MOTS-c administration increases exercise performance by boosting skeletal muscle stress responses and by enhancing metabolic adaptation to exercise. Exogenous MOTS-c also stimulates thermogenesis in subcutaneous white adipose tissues, thereby enhancing energy expenditure and contributing to the anti-obesity effects of exercise training. This review briefly summarizes the mitohormetic mechanisms of exercise with an emphasis on MOTS-c.

Identifiants

pubmed: 35656563
pii: dmj.2022.0092
doi: 10.4093/dmj.2022.0092
pmc: PMC9171157
doi:

Substances chimiques

RNA, Ribosomal 0
RNA, ribosomal, 12S 0
Transcription Factors 0

Types de publication

Journal Article Review Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

402-413

Subventions

Organisme : NCIRD CDC HHS
ID : H23 IP000855
Pays : United States

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Auteurs

Tae Kwan Yoon (TK)

Division of Endocrinology and Metabolism, Department of Internal Medicine, H+ Yangji Hospital, Seoul, Korea.

Chan Hee Lee (CH)

Department of of Biomedical Science & Program of Material Science for Medicine and Pharmaceutics, Hallym University, Chuncheon, Korea.

Obin Kwon (O)

Department of Biomedical Sciences, Seoul National University College of Medicine, Seoul, Korea.

Min-Seon Kim (MS)

Division of Endocrinology and Metabolism, Department of Internal Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Korea.

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Classifications MeSH