Avoiding entry into intracellular protein degradation pathways by signal mutations increases protein secretion in Pichia pastoris.


Journal

Microbial biotechnology
ISSN: 1751-7915
Titre abrégé: Microb Biotechnol
Pays: United States
ID NLM: 101316335

Informations de publication

Date de publication:
09 2022
Historique:
revised: 23 03 2022
received: 03 09 2021
accepted: 31 03 2022
pubmed: 4 6 2022
medline: 9 9 2022
entrez: 3 6 2022
Statut: ppublish

Résumé

In our previous study, we serendipitously discovered that protein secretion in the methylotrophic yeast Pichia pastoris is enhanced by a mutation (V50A) in the mating factor alpha (MFα) prepro-leader signal derived from Saccharomyces cerevisiae. In the present study, we investigated 20 single-amino-acid substitutions, including V50A, located within the MFα signal peptide, indicating that V50A and several single mutations alone provided significant increase in production of the secreted proteins. In addition to hydrophobicity index analysis, both an unfolded protein response (UPR) biosensor analysis and a microscopic observation showed a clear difference on the levels of UPR induction and mis-sorting of secretory protein into vacuoles among the wild-type and mutated MFα signal peptides. This work demonstrates the importance of avoiding entry of secretory proteins into the intracellular protein degradation pathways, an observation that is expected to contribute to the engineering of strains with increased production of recombinant secreted proteins.

Identifiants

pubmed: 35656803
doi: 10.1111/1751-7915.14061
pmc: PMC9437885
doi:

Substances chimiques

Fungal Proteins 0
Protein Sorting Signals 0
Recombinant Proteins 0
Mating Factor 61194-02-3

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2364-2378

Informations de copyright

© 2022 The Authors. Microbial Biotechnology published by Society for Applied Microbiology and John Wiley & Sons Ltd.

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Auteurs

Yoichiro Ito (Y)

Engineering Biology Research Center, Kobe University, 1-1 Rokkodai, Nada, Kobe, 657-8501, Japan.
Graduate School of Science, Technology and Innovation, Kobe University, 1-1 Rokkodai, Nada, Kobe, 657-8501, Japan.

Misa Ishigami (M)

Technology Research Association of Highly Efficient Gene Design (TRAHED), Kobe, Japan.

Noriko Hashiba (N)

Technology Research Association of Highly Efficient Gene Design (TRAHED), Kobe, Japan.

Yasuyuki Nakamura (Y)

Engineering Biology Research Center, Kobe University, 1-1 Rokkodai, Nada, Kobe, 657-8501, Japan.
Graduate School of Science, Technology and Innovation, Kobe University, 1-1 Rokkodai, Nada, Kobe, 657-8501, Japan.

Goro Terai (G)

Department of Computational Biology and Medical Sciences, Graduate School of Frontier Sciences, The University of Tokyo, Chiba, Japan.

Tomohisa Hasunuma (T)

Engineering Biology Research Center, Kobe University, 1-1 Rokkodai, Nada, Kobe, 657-8501, Japan.
Graduate School of Science, Technology and Innovation, Kobe University, 1-1 Rokkodai, Nada, Kobe, 657-8501, Japan.

Jun Ishii (J)

Engineering Biology Research Center, Kobe University, 1-1 Rokkodai, Nada, Kobe, 657-8501, Japan.
Graduate School of Science, Technology and Innovation, Kobe University, 1-1 Rokkodai, Nada, Kobe, 657-8501, Japan.

Akihiko Kondo (A)

Engineering Biology Research Center, Kobe University, 1-1 Rokkodai, Nada, Kobe, 657-8501, Japan.
Graduate School of Science, Technology and Innovation, Kobe University, 1-1 Rokkodai, Nada, Kobe, 657-8501, Japan.
Department of Chemical Science and Engineering, Graduate School of Engineering, Kobe University, Kobe, Japan.
Center for Sustainable Resource Science, RIKEN, Yokohama, Japan.

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Classifications MeSH