Myelin water imaging in relapsing multiple sclerosis treated with ocrelizumab and interferon beta-1a.


Journal

NeuroImage. Clinical
ISSN: 2213-1582
Titre abrégé: Neuroimage Clin
Pays: Netherlands
ID NLM: 101597070

Informations de publication

Date de publication:
2022
Historique:
received: 22 04 2022
revised: 27 06 2022
accepted: 10 07 2022
pubmed: 26 7 2022
medline: 25 8 2022
entrez: 25 7 2022
Statut: ppublish

Résumé

Myelin water imaging is a magnetic resonance imaging (MRI) technique that quantifies myelin damage and repair in multiple sclerosis (MS) via the myelin water fraction (MWF). In this substudy of a phase 3 therapeutic trial, OPERA II, MWF was assessed in relapsing MS participants assigned to interferon beta-1a (IFNb-1a) or ocrelizumab (OCR) during a two-year double-blind period (DBP) followed by a two-year open label extension (OLE) with ocrelizumab treatment. MWF in normal appearing white matter (NAWM), including both whole brain NAWM and 5 white matter structures, and chronic lesions, was assessed in 29 OCR and 26 IFNb-1a treated participants at weeks 0, 24, 48 and 96 (DBP), and weeks 144 and 192 (OLE), and in white matter for 23 healthy control participants at weeks 0, 48 and 96. Linear mixed-effects models of data from baseline to week 96 showed a difference in the change in MWF over time favouring ocrelizumab in all NAWM regions. At week 192, lesion MWF was lower for participants originally randomised to IFNb-1a compared to those originally randomised to OCR. Controls showed no change in MWF over 96 weeks in any region. Ocrelizumab appears to protect against demyelination in MS NAWM and chronic lesions and may allow for a more permissive micro environment for remyelination to occur in focal and diffusely damaged tissue.

Sections du résumé

BACKGROUND
Myelin water imaging is a magnetic resonance imaging (MRI) technique that quantifies myelin damage and repair in multiple sclerosis (MS) via the myelin water fraction (MWF).
OBJECTIVE
In this substudy of a phase 3 therapeutic trial, OPERA II, MWF was assessed in relapsing MS participants assigned to interferon beta-1a (IFNb-1a) or ocrelizumab (OCR) during a two-year double-blind period (DBP) followed by a two-year open label extension (OLE) with ocrelizumab treatment.
METHODS
MWF in normal appearing white matter (NAWM), including both whole brain NAWM and 5 white matter structures, and chronic lesions, was assessed in 29 OCR and 26 IFNb-1a treated participants at weeks 0, 24, 48 and 96 (DBP), and weeks 144 and 192 (OLE), and in white matter for 23 healthy control participants at weeks 0, 48 and 96.
RESULTS
Linear mixed-effects models of data from baseline to week 96 showed a difference in the change in MWF over time favouring ocrelizumab in all NAWM regions. At week 192, lesion MWF was lower for participants originally randomised to IFNb-1a compared to those originally randomised to OCR. Controls showed no change in MWF over 96 weeks in any region.
CONCLUSION
Ocrelizumab appears to protect against demyelination in MS NAWM and chronic lesions and may allow for a more permissive micro environment for remyelination to occur in focal and diffusely damaged tissue.

Identifiants

pubmed: 35878575
pii: S2213-1582(22)00174-7
doi: 10.1016/j.nicl.2022.103109
pmc: PMC9421448
pii:
doi:

Substances chimiques

Antibodies, Monoclonal, Humanized 0
Water 059QF0KO0R
ocrelizumab A10SJL62JY
Interferon beta-1a XRO4566Q4R

Types de publication

Clinical Trial, Phase III Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

103109

Informations de copyright

Copyright © 2022 The Author(s). Published by Elsevier Inc. All rights reserved.

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Auteurs

Shannon Kolind (S)

Department of Medicine (Neurology), University of British Columbia, 2775 Laurel Street, Vancouver V5Z 1M9, Canada; Department of Radiology, University of British Columbia, 2775 Laurel Street, Vancouver V5Z 1M9, Canada; Department of Physics and Astronomy, University of British Columbia, 6224 Agricultural Road, Vancouver V6T 1Z1, Canada; International Collaboration on Repair Discoveries (ICORD), University of British Columbia, 818 West 10th Avenue, Vancouver, BC V5Z 1M9, Canada. Electronic address: Shannon.Kolind@ubc.ca.

Shawna Abel (S)

Department of Medicine (Neurology), University of British Columbia, 2775 Laurel Street, Vancouver V5Z 1M9, Canada.

Carolyn Taylor (C)

Applied Statistics and Data Science Group (ASDa), Department of Statistics, University of British Columbia, 3178-2207 Main Mall, Vancouver, V6T 1Z4, Canada.

Roger Tam (R)

Department of Radiology, University of British Columbia, 2775 Laurel Street, Vancouver V5Z 1M9, Canada; School of Biomedical Engineering, University of British Columbia, 251 - 2222 Heath Sciences Mall, Vancouver V6T 1Z3, Canada.

Cornelia Laule (C)

Department of Radiology, University of British Columbia, 2775 Laurel Street, Vancouver V5Z 1M9, Canada; Department of Physics and Astronomy, University of British Columbia, 6224 Agricultural Road, Vancouver V6T 1Z1, Canada; International Collaboration on Repair Discoveries (ICORD), University of British Columbia, 818 West 10th Avenue, Vancouver, BC V5Z 1M9, Canada; Department of Pathology & Laboratory Medicine, University of British Columbia, 2211 West Mall, Vancouver V6T 2B5, Canada.

David K B Li (DKB)

Department of Radiology, University of British Columbia, 2775 Laurel Street, Vancouver V5Z 1M9, Canada.

Hideki Garren (H)

Prothena Biosciences, 331 Oyster Point Boulevard, South San Francisco, CA 94080, USA.

Laura Gaetano (L)

F. Hoffmann-La Roche Ltd, Grenzacherstrasse 124, Basel 4070, Switzerland.

Corrado Bernasconi (C)

F. Hoffmann-La Roche Ltd, Grenzacherstrasse 124, Basel 4070, Switzerland.

David Clayton (D)

Genentech, Inc., 1 DNA Way, South San Francisco, CA 94080, USA.

Irene Vavasour (I)

Department of Radiology, University of British Columbia, 2775 Laurel Street, Vancouver V5Z 1M9, Canada.

Anthony Traboulsee (A)

Department of Medicine (Neurology), University of British Columbia, 2775 Laurel Street, Vancouver V5Z 1M9, Canada.

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Classifications MeSH