Transgenic Mice for the Translational Study of Neuropathic Pain and Dystonia.
DYT1
dystonia
gabapentin
neurodegenerative diseases
pain
torsinA
transgenic mice
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
02 Aug 2022
02 Aug 2022
Historique:
received:
24
06
2022
revised:
26
07
2022
accepted:
29
07
2022
entrez:
12
8
2022
pubmed:
13
8
2022
medline:
16
8
2022
Statut:
epublish
Résumé
Murine models are fundamental in the study of clinical conditions and the development of new drugs and treatments. Transgenic technology has started to offer advantages in oncology, encompassing all research fields related to the study of painful syndromes. Knockout mice or mice overexpressing genes encoding for proteins linked to pain development and maintenance can be produced and pain models can be applied to transgenic mice to model the most disabling neurological conditions. Due to the association of movement disorders with sensitivity and pain processing, our group focused for the first time on the role of the torsinA gene GAG deletion-responsible for DYT1 dystonia-in baseline sensitivity and neuropathic responses. The aim of the present report are to review the complex network that exists between the chaperonine-like protein torsinA and the baseline sensitivity pattern-which are fundamental in neuropathic pain-and to point at its possible role in neurodegenerative diseases.
Identifiants
pubmed: 35955710
pii: ijms23158580
doi: 10.3390/ijms23158580
pmc: PMC9368787
pii:
doi:
Substances chimiques
Dyt1 protein, mouse
0
Molecular Chaperones
0
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Ministry of Health (Rome, Italy)
ID : Progetto Finalizzato NET-2016-02361805 (WP5)
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