Digital Gait Biomarkers Allow to Capture 1-Year Longitudinal Change in Spinocerebellar Ataxia Type 3.


Journal

Movement disorders : official journal of the Movement Disorder Society
ISSN: 1531-8257
Titre abrégé: Mov Disord
Pays: United States
ID NLM: 8610688

Informations de publication

Date de publication:
Nov 2022
Historique:
revised: 15 07 2022
received: 14 03 2022
accepted: 10 08 2022
pubmed: 1 9 2022
medline: 16 11 2022
entrez: 31 8 2022
Statut: ppublish

Résumé

Measures of step variability and body sway during gait have shown to correlate with clinical ataxia severity in several cross-sectional studies. However, to serve as a valid progression biomarker, these gait measures have to prove their sensitivity to robustly capture longitudinal change, ideally within short time frames (eg, 1 year). We present the first multicenter longitudinal gait analysis study in spinocerebellar ataxias. We performed a combined cross-sectional (n = 28) and longitudinal (1-year interval, n = 17) analysis in Spinocerebellar Ataxia type 3 subjects (including seven preataxic mutation carriers). Longitudinal analysis showed significant change in gait measures between baseline and 1-year follow-up, with high effect sizes (stride length variability: P = 0.01, effect size r

Identifiants

pubmed: 36043376
doi: 10.1002/mds.29206
doi:

Substances chimiques

Biomarkers 0

Types de publication

Multicenter Study Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2295-2301

Informations de copyright

© 2022 The Authors. Movement Disorders published by Wiley Periodicals LLC on behalf of International Parkinson and Movement Disorder Society.

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Auteurs

Winfried Ilg (W)

Section Computational Sensomotorics, Hertie Institute for Clinical Brain Research, Tübingen, Germany.
German Center for Neurodegenerative Diseases (DZNE), Tübingen, Germany.

Björn Müller (B)

Section Computational Sensomotorics, Hertie Institute for Clinical Brain Research, Tübingen, Germany.

Jennifer Faber (J)

Department of Neurology, University Hospital Bonn, Bonn, Germany.
German Center for Neurodegenerative Diseases (DZNE), Bonn, Germany.

Judith van Gaalen (J)

Department of Neurology, Donders Institute for Brain, Cognition, and Behaviour, Radboud University Medical Center, Nijmegen, the Netherlands.

Holger Hengel (H)

German Center for Neurodegenerative Diseases (DZNE), Tübingen, Germany.
Department of Neurodegeneration, Hertie Institute for Clinical Brain Research and Centre of Neurology, Tübingen, Germany.

Ina R Vogt (IR)

German Center for Neurodegenerative Diseases (DZNE), Bonn, Germany.

Guido Hennes (G)

German Center for Neurodegenerative Diseases (DZNE), Bonn, Germany.

Bart van de Warrenburg (B)

Department of Neurology, Donders Institute for Brain, Cognition, and Behaviour, Radboud University Medical Center, Nijmegen, the Netherlands.

Thomas Klockgether (T)

Department of Neurology, University Hospital Bonn, Bonn, Germany.
German Center for Neurodegenerative Diseases (DZNE), Bonn, Germany.

Ludger Schöls (L)

German Center for Neurodegenerative Diseases (DZNE), Tübingen, Germany.
Department of Neurodegeneration, Hertie Institute for Clinical Brain Research and Centre of Neurology, Tübingen, Germany.

Matthis Synofzik (M)

German Center for Neurodegenerative Diseases (DZNE), Tübingen, Germany.
Department of Neurodegeneration, Hertie Institute for Clinical Brain Research and Centre of Neurology, Tübingen, Germany.

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