A case of novel DYT6 dystonia variant with serious complications after deep brain stimulation therapy: a case report.


Journal

BMC neurology
ISSN: 1471-2377
Titre abrégé: BMC Neurol
Pays: England
ID NLM: 100968555

Informations de publication

Date de publication:
12 Sep 2022
Historique:
received: 03 06 2022
accepted: 02 09 2022
entrez: 12 9 2022
pubmed: 13 9 2022
medline: 15 9 2022
Statut: epublish

Résumé

DYT6 dystonia belongs to a group of isolated, genetically determined, generalized dystonia associated with mutations in the THAP1 gene. We present the case of a young patient with DYT6 dystonia associated with a newly discovered c14G>A (p.Cys5Tyr) mutation in the THAP1 gene. We describe the clinical phenotype of this new mutation, effect of pallidal deep brain stimulation (DBS), which was accompanied by two rare postimplantation complications: an early intracerebral hemorrhage and delayed epileptic seizures. Among the published case reports of patients with DYT6 dystonia, the mentioned complications have not been described so far. DBS in the case of DYT6 dystonia is a challenge to thoroughly consider possible therapeutic benefits and potential risks associated with surgery. Genetic heterogeneity of the disease may also play an important role in predicting the development of the clinical phenotype as well as the effect of treatment including DBS. Therefore, it is beneficial to analyze the genetic and clinical relationships of DYT6 dystonia.

Sections du résumé

BACKGROUND BACKGROUND
DYT6 dystonia belongs to a group of isolated, genetically determined, generalized dystonia associated with mutations in the THAP1 gene.
CASE PRESENTATION METHODS
We present the case of a young patient with DYT6 dystonia associated with a newly discovered c14G>A (p.Cys5Tyr) mutation in the THAP1 gene. We describe the clinical phenotype of this new mutation, effect of pallidal deep brain stimulation (DBS), which was accompanied by two rare postimplantation complications: an early intracerebral hemorrhage and delayed epileptic seizures. Among the published case reports of patients with DYT6 dystonia, the mentioned complications have not been described so far.
CONCLUSIONS CONCLUSIONS
DBS in the case of DYT6 dystonia is a challenge to thoroughly consider possible therapeutic benefits and potential risks associated with surgery. Genetic heterogeneity of the disease may also play an important role in predicting the development of the clinical phenotype as well as the effect of treatment including DBS. Therefore, it is beneficial to analyze the genetic and clinical relationships of DYT6 dystonia.

Identifiants

pubmed: 36096774
doi: 10.1186/s12883-022-02871-3
pii: 10.1186/s12883-022-02871-3
pmc: PMC9465909
doi:

Substances chimiques

Apoptosis Regulatory Proteins 0
DNA-Binding Proteins 0
Nuclear Proteins 0
THAP1 protein, human 0

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

344

Subventions

Organisme : lékařská fakulta Univerzity Karlovy
ID : 825575
Organisme : Jessenius Faculty of Medicine (SK)
ID : 313011W875

Commentaires et corrections

Type : ErratumIn

Informations de copyright

© 2022. The Author(s).

Références

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Auteurs

M Grofik (M)

Department of Neurology, Jessenius Faculty of Medicine in Martin, Comenius University in Bratislava and University Hospital Martin, Martin, Slovakia.

M Cibulka (M)

Biomedical Centre Martin, Jessenius Faculty of Medicine in Martin, Comenius University in Bratislava, Bratislava, Slovakia. michal.cibulka@uniba.sk.

J Olekšáková (J)

Department of Neurology, Jessenius Faculty of Medicine in Martin, Comenius University in Bratislava and University Hospital Martin, Martin, Slovakia.

M Turčanová Koprušáková (M)

Department of Neurology, Jessenius Faculty of Medicine in Martin, Comenius University in Bratislava and University Hospital Martin, Martin, Slovakia.

T Galanda (T)

Department of Neurosurgery, Slovak Medical University and Roosevelt Hospital, Banska Bystrica, Slovakia.

J Necpál (J)

Department of Neurology, Zvolen Hospital, Zvolen, Slovakia.

P Jungová (P)

Department of Molecular and Biochemical Genetics - Centre of Rare Genetic Diseases, Faculty of Medicine & Comenius University, University Hospital Bratislava, Bratislava, Slovakia.

E Kurča (E)

Department of Neurology, Jessenius Faculty of Medicine in Martin, Comenius University in Bratislava and University Hospital Martin, Martin, Slovakia.

J Winkelmann (J)

Institute of Neurogenomics, Helmholtz Centrum, Munich, Germany.
Institute of Human Genetics, Technical University of Munich, Munich, Germany.

M Zech (M)

Institute of Neurogenomics, Helmholtz Centrum, Munich, Germany.
Institute of Human Genetics, Technical University of Munich, Munich, Germany.

R Jech (R)

Department of Neurology, Charles University, 1st Faculty of Medicine and General University Hospital, Prague, Czech Republic.

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