Drp1 Overexpression Decreases Insulin Content in Pancreatic MIN6 Cells.

MIN6 cell bioenergetics dynamin-related protein 1 glucose-stimulated insulin secretion insulin content overexpression

Journal

International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791

Informations de publication

Date de publication:
15 Oct 2022
Historique:
received: 05 08 2022
revised: 03 10 2022
accepted: 12 10 2022
entrez: 27 10 2022
pubmed: 28 10 2022
medline: 29 10 2022
Statut: epublish

Résumé

Mitochondrial dynamics and bioenergetics are central to glucose-stimulated insulin secretion by pancreatic beta cells. Previously, we demonstrated that a disturbance in glucose-invoked fission impairs insulin secretion by compromising glucose catabolism. Here, we investigated whether the overexpression of mitochondrial fission regulator

Identifiants

pubmed: 36293194
pii: ijms232012338
doi: 10.3390/ijms232012338
pmc: PMC9604375
pii:
doi:

Substances chimiques

Insulin 0
Glucose IY9XDZ35W2
Insulin, Regular, Human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Novo Nordisk Foundation
ID : 0059646

Références

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Auteurs

Uma D Kabra (UD)

Division of Metabolic Diseases, Technische Universität München, 80333 Munich, Germany.
Department of Pharmaceutical Chemistry, Parul Institute of Pharmacy, Parul University, Vadodara 391760, Gujarat, India.

Noah Moruzzi (N)

The Rolf Luft Research Center for Diabetes and Endocrinology, Karolinska Institutet, Karolinska University Hospital, SE-171 76 Stockholm, Sweden.

Per-Olof Berggren (PO)

The Rolf Luft Research Center for Diabetes and Endocrinology, Karolinska Institutet, Karolinska University Hospital, SE-171 76 Stockholm, Sweden.

Martin Jastroch (M)

Department of Molecular Biosciences, The Wenner-Gren Institute, Stockholm University, SE-106 91 Stockholm, Sweden.

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Classifications MeSH