Lasmiditan promotes recovery from acute kidney injury through induction of mitochondrial biogenesis.


Journal

American journal of physiology. Renal physiology
ISSN: 1522-1466
Titre abrégé: Am J Physiol Renal Physiol
Pays: United States
ID NLM: 100901990

Informations de publication

Date de publication:
01 01 2023
Historique:
pmc-release: 01 01 2024
pubmed: 4 11 2022
medline: 20 12 2022
entrez: 3 11 2022
Statut: ppublish

Résumé

Acute kidney injury (AKI) involves rapid loss of renal function and occurs in 8-16% of hospitalized patients. AKI can be induced by drugs, sepsis, and ischemia-reperfusion (I/R). Hallmarks of AKI include mitochondrial and microvasculature dysfunction as well as renal tubular injury. There is currently no available therapeutic for AKI. Previously, our group identified that serotonin (5-HT)

Identifiants

pubmed: 36326468
doi: 10.1152/ajprenal.00249.2022
pmc: PMC9762961
doi:

Substances chimiques

lasmiditan 760I9WM792
Creatinine AYI8EX34EU

Types de publication

Journal Article Research Support, U.S. Gov't, Non-P.H.S. Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

F56-F63

Subventions

Organisme : BLRD VA
ID : I01 BX000851
Pays : United States
Organisme : NIEHS NIH HHS
ID : T32 ES007091
Pays : United States
Organisme : NIGMS NIH HHS
ID : T32 GM139779
Pays : United States

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Auteurs

Kevin A Hurtado (KA)

Department of Pharmacology and Toxicology, College of Pharmacy, University of Arizona, Tucson, Arizona.

Jaroslav Janda (J)

Department of Pharmacology and Toxicology, College of Pharmacy, University of Arizona, Tucson, Arizona.

Rick G Schnellmann (RG)

Department of Pharmacology and Toxicology, College of Pharmacy, University of Arizona, Tucson, Arizona.
Southern Arizona Veterans Affairs Health Care System, Tucson, Arizona.
Southwest Environmental Health Science Center, University of Arizona, Tucson, Arizona.

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Classifications MeSH