Neutrophilic granulocyte-derived B-cell activating factor supports B cells in skin lesions in hidradenitis suppurativa.

Acne inversa B cell Cibersort G-CSF IL-17 RNA sequencing TNF-α TNFSF13B granulocyte plasma cell tertiary lymphoid structures

Journal

The Journal of allergy and clinical immunology
ISSN: 1097-6825
Titre abrégé: J Allergy Clin Immunol
Pays: United States
ID NLM: 1275002

Informations de publication

Date de publication:
04 2023
Historique:
received: 25 03 2022
revised: 17 09 2022
accepted: 20 10 2022
medline: 11 4 2023
pubmed: 9 12 2022
entrez: 8 12 2022
Statut: ppublish

Résumé

Hidradenitis suppurativa (HS) is a chronic inflammatory disease characterized by painful inflamed nodules, abscesses, and pus-draining tunnels appearing in axillary, inguinal, and perianal skin areas. HS lesions contain various types of immigrated immune cells. This study aimed to characterize mediators that support lesional B/plasma cell persistence in HS. Skin samples from several cohorts of HS patients and control cohorts were assessed by mRNA sequencing, quantitative PCR on reverse-transcribed RNA, flow cytometry, and immunohistofluorescence. Blood plasma and cultured skin biopsy samples, keratinocytes, dermal fibroblasts, neutrophilic granulocytes (neutrophils), monocytes, and B cells were analyzed. Complex systems biology approaches were used to evaluate bulk and single-cell RNA sequencing data. Proportions of B/plasma cells, neutrophils, CD8 Neutrophil-derived BAFF supports B/plasma cell persistence and function in HS lesions.

Sections du résumé

BACKGROUND
Hidradenitis suppurativa (HS) is a chronic inflammatory disease characterized by painful inflamed nodules, abscesses, and pus-draining tunnels appearing in axillary, inguinal, and perianal skin areas. HS lesions contain various types of immigrated immune cells.
OBJECTIVE
This study aimed to characterize mediators that support lesional B/plasma cell persistence in HS.
METHODS
Skin samples from several cohorts of HS patients and control cohorts were assessed by mRNA sequencing, quantitative PCR on reverse-transcribed RNA, flow cytometry, and immunohistofluorescence. Blood plasma and cultured skin biopsy samples, keratinocytes, dermal fibroblasts, neutrophilic granulocytes (neutrophils), monocytes, and B cells were analyzed. Complex systems biology approaches were used to evaluate bulk and single-cell RNA sequencing data.
RESULTS
Proportions of B/plasma cells, neutrophils, CD8
CONCLUSION
Neutrophil-derived BAFF supports B/plasma cell persistence and function in HS lesions.

Identifiants

pubmed: 36481267
pii: S0091-6749(22)01621-9
doi: 10.1016/j.jaci.2022.10.034
pii:
doi:

Substances chimiques

B-Cell Activating Factor 0
Interleukin-4 207137-56-2

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1015-1026

Informations de copyright

Copyright © 2022. Published by Elsevier Inc.

Auteurs

Robert Sabat (R)

Psoriasis Research and Treatment Centre, Charité-Universitätsmedizin Berlin, Berlin, Germany; Interdisciplinary Group Molecular Immunopathology, Dermatology/Medical Immunology, Charité-Universitätsmedizin Berlin, Berlin, Germany. Electronic address: robert.sabat@charite.de.

Deimantė Šimaitė (D)

Data and Data Sciences, Sanofi-Aventis Deutschland GmbH, Frankfurt, Germany.

Johann Eli Gudjonsson (JE)

Department of Dermatology, University of Michigan, and Taubman Medical Research Institute, University of Michigan Medical School, Ann Arbor, Mich.

Theresa-Charlotte Brembach (TC)

Psoriasis Research and Treatment Centre, Charité-Universitätsmedizin Berlin, Berlin, Germany; Interdisciplinary Group Molecular Immunopathology, Dermatology/Medical Immunology, Charité-Universitätsmedizin Berlin, Berlin, Germany; Department of Food Chemistry, Institute of Nutritional Science, University of Potsdam, Potsdam, Germany.

Katrin Witte (K)

Psoriasis Research and Treatment Centre, Charité-Universitätsmedizin Berlin, Berlin, Germany; Interdisciplinary Group Molecular Immunopathology, Dermatology/Medical Immunology, Charité-Universitätsmedizin Berlin, Berlin, Germany; Inflammation and Regeneration of the Skin, BIH Center for Regenerative Therapies, Charité-Universitätsmedizin Berlin, Berlin, Germany.

Torben Krause (T)

Psoriasis Research and Treatment Centre, Charité-Universitätsmedizin Berlin, Berlin, Germany.

Georgios Kokolakis (G)

Psoriasis Research and Treatment Centre, Charité-Universitätsmedizin Berlin, Berlin, Germany.

Eckart Bartnik (E)

Immunology & Inflammation Research TA, Sanofi-Aventis Deutschland GmbH, Frankfurt, Germany.

Christos Nikolaou (C)

Psoriasis Research and Treatment Centre, Charité-Universitätsmedizin Berlin, Berlin, Germany; Interdisciplinary Group Molecular Immunopathology, Dermatology/Medical Immunology, Charité-Universitätsmedizin Berlin, Berlin, Germany.

Natascha Rill (N)

Psoriasis Research and Treatment Centre, Charité-Universitätsmedizin Berlin, Berlin, Germany; Interdisciplinary Group Molecular Immunopathology, Dermatology/Medical Immunology, Charité-Universitätsmedizin Berlin, Berlin, Germany.

Béma Coulibaly (B)

Molecular Histopathology & Bio-Imaging, R&D, Sanofi-Aventis, Vitry-sur-Seine, France.

Clément Levin (C)

Molecular Histopathology & Bio-Imaging, R&D, Sanofi-Aventis, Vitry-sur-Seine, France.

Matthias Herrmann (M)

Immunology & Inflammation Research TA, Sanofi-Aventis Deutschland GmbH, Frankfurt, Germany.

Gabriela Salinas (G)

NGS-Integrative Genomics Core Unit, Institute of Human Genetics, University Medical Center Göttingen, Göttingen, Germany.

Thomas Leeuw (T)

Immunology & Inflammation Research TA, Sanofi-Aventis Deutschland GmbH, Frankfurt, Germany.

Hans-Dieter Volk (HD)

BIH Center for Regenerative Therapies, Charité-Universitätsmedizin Berlin, Berlin, Germany; Institute of Medical Immunology, Charité-Universitätsmedizin Berlin, Berlin, Germany.

Kamran Ghoreschi (K)

Department of Dermatology, Venereology and Allergology, Charité-Universitätsmedizin Berlin, Berlin, Germany.

Kerstin Wolk (K)

Psoriasis Research and Treatment Centre, Charité-Universitätsmedizin Berlin, Berlin, Germany; Interdisciplinary Group Molecular Immunopathology, Dermatology/Medical Immunology, Charité-Universitätsmedizin Berlin, Berlin, Germany; Inflammation and Regeneration of the Skin, BIH Center for Regenerative Therapies, Charité-Universitätsmedizin Berlin, Berlin, Germany. Electronic address: kerstin.wolk@charite.de.

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