Effect of eplerenone on clinical stability of Japanese patients with acute heart failure.


Journal

International journal of cardiology
ISSN: 1874-1754
Titre abrégé: Int J Cardiol
Pays: Netherlands
ID NLM: 8200291

Informations de publication

Date de publication:
01 03 2023
Historique:
received: 20 10 2022
revised: 20 12 2022
accepted: 23 12 2022
pubmed: 1 1 2023
medline: 8 2 2023
entrez: 31 12 2022
Statut: ppublish

Résumé

In the EARLIER (Efficacy and Safety of Early Initiation of Eplerenone Treatment in Patients with Acute Heart Failure) trial, eplerenone did not reduce heart failure (HF) hospitalizations or all-cause mortality in 300 patients admitted for acute HF (AHF). However, the trial might have been underpowered for these endpoints, and a comprehensive overview of the effect of eplerenone on diuretic doses and patients' clinical stability is warranted. The EARLIER trial included Japanese patients hospitalized for AHF randomly assigned to eplerenone or placebo over 6 months. Cox proportional hazards and mixed-effects models were used for analyses. Three hundred patients were included (mean age, 67 ± 13 years; 73% males). The median furosemide equivalent dose was 40 (20-62) mg at randomization. Patients with higher furosemide-equivalent doses had more severe signs and symptoms of congestion and a higher risk of all-cause mortality or HF hospitalization during 6-month follow-up (adjusted-hazard ratio per 10 mg/day increase = 1.25, 95% confidence interval: 1.05-1.49). Eplerenone significantly decreased furosemide-equivalent diuretic doses and b-type natriuretic levels throughout the follow-up (overall-joint-p < 0.05 for both) and reduced E/e' and inferior vena cava diameter at 4 weeks (both p < 0.05). Additionally, eplerenone significantly reduced left ventricular (LV) end-diastolic diameter at 24 weeks (p < 0.05). Eplerenone treatment improved the clinical stability particularly during short period following hospitalization for AHF, translated by lower diuretic doses, natriuretic peptide levels, indirect markers of filling pressure and venous congestion, and a smaller LV volume.

Sections du résumé

BACKGROUND
In the EARLIER (Efficacy and Safety of Early Initiation of Eplerenone Treatment in Patients with Acute Heart Failure) trial, eplerenone did not reduce heart failure (HF) hospitalizations or all-cause mortality in 300 patients admitted for acute HF (AHF). However, the trial might have been underpowered for these endpoints, and a comprehensive overview of the effect of eplerenone on diuretic doses and patients' clinical stability is warranted.
METHODS
The EARLIER trial included Japanese patients hospitalized for AHF randomly assigned to eplerenone or placebo over 6 months. Cox proportional hazards and mixed-effects models were used for analyses.
RESULTS
Three hundred patients were included (mean age, 67 ± 13 years; 73% males). The median furosemide equivalent dose was 40 (20-62) mg at randomization. Patients with higher furosemide-equivalent doses had more severe signs and symptoms of congestion and a higher risk of all-cause mortality or HF hospitalization during 6-month follow-up (adjusted-hazard ratio per 10 mg/day increase = 1.25, 95% confidence interval: 1.05-1.49). Eplerenone significantly decreased furosemide-equivalent diuretic doses and b-type natriuretic levels throughout the follow-up (overall-joint-p < 0.05 for both) and reduced E/e' and inferior vena cava diameter at 4 weeks (both p < 0.05). Additionally, eplerenone significantly reduced left ventricular (LV) end-diastolic diameter at 24 weeks (p < 0.05).
CONCLUSIONS
Eplerenone treatment improved the clinical stability particularly during short period following hospitalization for AHF, translated by lower diuretic doses, natriuretic peptide levels, indirect markers of filling pressure and venous congestion, and a smaller LV volume.

Identifiants

pubmed: 36586516
pii: S0167-5273(22)01938-6
doi: 10.1016/j.ijcard.2022.12.045
pii:
doi:

Substances chimiques

Diuretics 0
Eplerenone 6995V82D0B
Furosemide 7LXU5N7ZO5
Mineralocorticoid Receptor Antagonists 0

Types de publication

Review Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

73-78

Informations de copyright

Copyright © 2022 Elsevier B.V. All rights reserved.

Auteurs

Masatake Kobayashi (M)

Department of Cardiology, Tokyo medical university, Tokyo, Japan. Electronic address: mkoba12@me.com.

João Pedro Ferreira (JP)

Université de Lorraine, INSERM, Centre d'Investigations Cliniques Plurithématique 1433, Inserm U1116, CHRU de Nancy and F-CRIN INI-CRCT, Nancy, France; Cardiovascular Research and Development Center, Department of Surgery and Physiology, Faculty of Medicine of the University of Porto, Porto, Portugal.

Yuya Matsue (Y)

Department of Cardiovascular Biology and Medicine, Juntendo University Graduate School of Medicine, Tokyo, Japan.

Taishiro Chikamori (T)

Department of Cardiology, Tokyo medical university, Tokyo, Japan.

Shin Ito (S)

Department of Clinical Research and Development, National Cerebral and Cardiovascular Center, Osaka, Japan.

Masanori Asakura (M)

Department of Cardiovascular and Renal Medicine, Hyogo Medical University, Hyogo, Japan.

Akira Yamashina (A)

Department of Cardiology, Tokyo medical university, Tokyo, Japan; Department of Nursing, Kiryu University, Gunma, Japan.

Masafumi Kitakaze (M)

Department of Clinical Research and Development, National Cerebral and Cardiovascular Center, Osaka, Japan; Hanwa Memorial Hospital, Osaka, Japan. Electronic address: kitakaze@zf6.so-net.ne.jp.

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