Evidence of widespread endemic populations of highly multidrug resistant Klebsiella pneumoniae in hospital settings in Hanoi, Vietnam: a prospective cohort study.


Journal

The Lancet. Microbe
ISSN: 2666-5247
Titre abrégé: Lancet Microbe
Pays: England
ID NLM: 101769019

Informations de publication

Date de publication:
04 2023
Historique:
received: 27 09 2021
revised: 14 09 2022
accepted: 16 11 2022
medline: 4 4 2023
pubmed: 22 2 2023
entrez: 21 2 2023
Statut: ppublish

Résumé

Patients with prolonged hospitalisation have a significant risk of carriage of and subsequent infection with extended spectrum β-lactamase (ESBL)-producing and carbapenemase-producing Klebsiella pneumoniae. However, the distinctive roles of the community and hospital environments in the transmission of ESBL-producing or carbapenemase-producing K pneumoniae remain elusive. We aimed to investigate the prevalence and transmission of K pneumoniae within and between the two tertiary hospitals in Hanoi, Viet Nam, using whole-genome sequencing. We did a prospective cohort study of 69 patients in intensive care units (ICUs) from two hospitals in Hanoi, Viet Nam. Patients were included if they were aged 18 years or older, admitted for longer than the mean length of stay in their ICU, and cultured K pneumoniae from their clinical samples. Longitudinally collected samples from patients (collected weekly) and the ICU environment (collected monthly) were cultured on selective media, and whole-genome sequences from K pneumoniae colonies analysed. We did phylogenetic analyses and correlated phenotypic antimicrobial susceptibility testing with genotypic features of K pneumoniae isolates. We constructed transmission networks of patient samples, relating ICU admission times and locations with genetic similarity of infecting K pneumoniae. Between June 1, 2017, and Jan 31, 2018, 69 patients were in the ICUs and eligible for inclusion, and a total of 357 K pneumoniae isolates were cultured and successfully sequenced. 228 (64%) of K pneumoniae isolates carried between two and four different ESBL-encoding and carbapenemase-encoding genes, with 164 (46%) isolates carrying genes encoding both, with high minimum inhibitory concentrations. We found a novel co-occurrence of bla These results highlight the high prevalence of ESBL-positive carbapenem-resistant K pneumoniae in ICUs in Viet Nam. Through studying K pneumoniae ST15 in detail, we showed how important resistance genes are contained within these strains that are carried broadly by patients entering the two hospitals directly or through referral. Medical Research Council Newton Fund, Ministry of Science and Technology, Wellcome Trust, Academy of Medical Sciences, Health Foundation, and National Institute for Health and Care Research Cambridge Biomedical Research Centre.

Sections du résumé

BACKGROUND
Patients with prolonged hospitalisation have a significant risk of carriage of and subsequent infection with extended spectrum β-lactamase (ESBL)-producing and carbapenemase-producing Klebsiella pneumoniae. However, the distinctive roles of the community and hospital environments in the transmission of ESBL-producing or carbapenemase-producing K pneumoniae remain elusive. We aimed to investigate the prevalence and transmission of K pneumoniae within and between the two tertiary hospitals in Hanoi, Viet Nam, using whole-genome sequencing.
METHODS
We did a prospective cohort study of 69 patients in intensive care units (ICUs) from two hospitals in Hanoi, Viet Nam. Patients were included if they were aged 18 years or older, admitted for longer than the mean length of stay in their ICU, and cultured K pneumoniae from their clinical samples. Longitudinally collected samples from patients (collected weekly) and the ICU environment (collected monthly) were cultured on selective media, and whole-genome sequences from K pneumoniae colonies analysed. We did phylogenetic analyses and correlated phenotypic antimicrobial susceptibility testing with genotypic features of K pneumoniae isolates. We constructed transmission networks of patient samples, relating ICU admission times and locations with genetic similarity of infecting K pneumoniae.
FINDINGS
Between June 1, 2017, and Jan 31, 2018, 69 patients were in the ICUs and eligible for inclusion, and a total of 357 K pneumoniae isolates were cultured and successfully sequenced. 228 (64%) of K pneumoniae isolates carried between two and four different ESBL-encoding and carbapenemase-encoding genes, with 164 (46%) isolates carrying genes encoding both, with high minimum inhibitory concentrations. We found a novel co-occurrence of bla
INTERPRETATION
These results highlight the high prevalence of ESBL-positive carbapenem-resistant K pneumoniae in ICUs in Viet Nam. Through studying K pneumoniae ST15 in detail, we showed how important resistance genes are contained within these strains that are carried broadly by patients entering the two hospitals directly or through referral.
FUNDING
Medical Research Council Newton Fund, Ministry of Science and Technology, Wellcome Trust, Academy of Medical Sciences, Health Foundation, and National Institute for Health and Care Research Cambridge Biomedical Research Centre.

Identifiants

pubmed: 36801013
pii: S2666-5247(22)00338-X
doi: 10.1016/S2666-5247(22)00338-X
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e255-e263

Subventions

Organisme : Medical Research Council
ID : MC_UU_00031/7
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 206194
Pays : United Kingdom
Organisme : Department of Health
Pays : United Kingdom

Informations de copyright

Copyright © 2023 The Author(s). Published by Elsevier Ltd. This is an Open Access article under the CC BY 4.0 license. Published by Elsevier Ltd.. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of interests JP is a paid consultant for Next Gen Diagnostics. All other authors declare no competing interests.

Auteurs

My H Pham (MH)

Wellcome Sanger Institute, Hinxton, UK; Oxford University Clinical Research Unit, Hanoi, Viet Nam.

Le Thi Hoi (LT)

National Hospital for Tropical Diseases, Hanoi, Viet Nam; Hanoi Medical University, Hanoi, Viet Nam.

Mathew A Beale (MA)

Wellcome Sanger Institute, Hinxton, UK.

Fahad A Khokhar (FA)

Department of Medicine, University of Cambridge, Cambridge, UK; Cambridge Institute for Therapeutic Immunology and Infectious Disease, Cambridge, UK.

Nguyen Thi Hoa (NT)

National Hospital for Tropical Diseases, Hanoi, Viet Nam; National Lung Hospital, Department of Microbiology and National Tuberculosis Reference Laboratory, Hanoi, Viet Nam.

Patrick Musicha (P)

Wellcome Sanger Institute, Hinxton, UK.

Grace A Blackwell (GA)

Wellcome Sanger Institute, Hinxton, UK; EMBL-EBI, Wellcome Genome Campus, Hinxton, Cambridge, UK.

Hoang Bao Long (HB)

Oxford University Clinical Research Unit, Hanoi, Viet Nam.

Dang Thi Huong (DT)

National Hospital for Tropical Diseases, Hanoi, Viet Nam.

Nguyen Gia Binh (NG)

Bach Mai Hospital, Hanoi, Viet Nam.

Dao Xuan Co (DX)

Bach Mai Hospital, Hanoi, Viet Nam.

Tran Giang (T)

National Hospital for Tropical Diseases, Hanoi, Viet Nam.

Cuong Bui (C)

Bach Mai Hospital, Hanoi, Viet Nam.

Hai Ninh Tran (HN)

National Hospital for Tropical Diseases, Hanoi, Viet Nam.

James Bryan (J)

Department of Medicine, University of Cambridge, Cambridge, UK.

Archie Herrick (A)

Department of Medicine, University of Cambridge, Cambridge, UK.

Theresa Feltwell (T)

Department of Medicine, University of Cambridge, Cambridge, UK.

Behzad Nadjm (B)

Oxford University Clinical Research Unit, Hanoi, Viet Nam; MRC Unit The Gambia at London School of Hygiene & Tropical Medicine, Fajara, The Gambia.

Julian Parkhill (J)

Department of Veterinary Medicine, University of Cambridge, Cambridge, UK.

Hindrik Rogier van Doorn (HR)

Oxford University Clinical Research Unit, Hanoi, Viet Nam; Nuffield Department of Medicine, University of Oxford, Oxford, UK.

Nguyen Vu Trung (NV)

National Hospital for Tropical Diseases, Hanoi, Viet Nam; Hanoi Medical University, Hanoi, Viet Nam.

Nguyen Van Kinh (N)

National Hospital for Tropical Diseases, Hanoi, Viet Nam.

Mili Estée Török (ME)

Department of Medicine, University of Cambridge, Cambridge, UK; Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK. Electronic address: et317@cam.ac.uk.

Nicholas R Thomson (NR)

Wellcome Sanger Institute, Hinxton, UK; London School of Hygiene and Tropical Medicine, London, UK.

Articles similaires

Genome, Chloroplast Phylogeny Genetic Markers Base Composition High-Throughput Nucleotide Sequencing

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C

Classifications MeSH