HLA-B leader genotypes in a clinical population.


Journal

HLA
ISSN: 2059-2310
Titre abrégé: HLA
Pays: England
ID NLM: 101675570

Informations de publication

Date de publication:
07 2023
Historique:
revised: 15 02 2023
received: 25 08 2022
accepted: 15 02 2023
medline: 15 6 2023
pubmed: 18 3 2023
entrez: 17 3 2023
Statut: ppublish

Résumé

The -21 dimorphism in the leader sequences of HLA-B exon 1 is associated with risk of graft-versus-host disease (GVHD), relapse and overall survival after unrelated donor hematopoietic cell transplantation (HCT), haploidentical HCT and cord blood transplantation. Consideration of the leader dimorphism in the prospective selection of allogeneic donors for HCT may help to lower risks for patients, but requires understanding of the frequencies of the leader in patients and candidate transplant donors. We defined the frequencies of the HLA-B leader, and its association to HLA-B Bw4/Bw6 and C1/C2 KIR epitopes. Sequence variants of rs1050458 of exon 1 position -21 for 11,126 haplotypes were analyzed from high resolution HLA typing of over 5500 study subjects. HLA typing was performed by TruSight/AlloSeq NGS and analyzed using TruSight/AlloSeq Assign software. HLA-B Bw4/Bw6 and C1/C2 KIR epitopes were defined based on established sequence alignments and nomenclature. Alleles at rs1050458 of HLA-B exon 1 were validated as dimorphic: rs1050458-C or -T variants encoding threonine (T) or methionine (M) at anchor position 2 (P2) of nonameric HLA-B leader peptides, respectfully. No additional variants were observed. Among study subjects, 70% of HLA-B haplotypes encoded T-leader and 30% encoded M-leader sequences. The genotype frequencies of TT, MT, and MM were consistent among patient, related, and unrelated donor groups. The associations of M/T leader, Bw4/Bw6, and C1/C2 enhanced understanding of the Class I features involved in the innate immune response. A population of patients and transplant donors confirms the rs1050458 leader dimorphism and its association with HLA-B Bw4/Bw6 and C1/C2 KIR features.

Identifiants

pubmed: 36929133
doi: 10.1111/tan.15022
doi:

Substances chimiques

Receptors, KIR 0
HLA-B Antigens 0
Epitopes 0

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

44-51

Informations de copyright

© 2023 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd.

Références

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Auteurs

Gansuvd Balgansuren (G)

Clinical Immunogenetics Laboratory, Fred Hutchinson Cancer Center, Seattle, Washington, USA.
Department of Laboratory Medicine and Pathology, University of Washington School of Medicine, Seattle, Washington, USA.

Maggie Sprague (M)

Clinical Immunogenetics Laboratory, Fred Hutchinson Cancer Center, Seattle, Washington, USA.

Paula Peterson (P)

Clinical Immunogenetics Laboratory, Fred Hutchinson Cancer Center, Seattle, Washington, USA.

Yasaman Shenavar (Y)

Clinical Immunogenetics Laboratory, Fred Hutchinson Cancer Center, Seattle, Washington, USA.

Ada Ng (A)

Clinical Immunogenetics Laboratory, Fred Hutchinson Cancer Center, Seattle, Washington, USA.

Lois Regen (L)

Clinical Immunogenetics Laboratory, Fred Hutchinson Cancer Center, Seattle, Washington, USA.

Nakita Shelton (N)

Clinical Immunogenetics Laboratory, Fred Hutchinson Cancer Center, Seattle, Washington, USA.

Effie Petersdorf (E)

Clinical Immunogenetics Laboratory, Fred Hutchinson Cancer Center, Seattle, Washington, USA.
Department of Laboratory Medicine and Pathology, University of Washington School of Medicine, Seattle, Washington, USA.

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