Sciatic nerve fractional anisotropy and neurofilament light chain protein are related to sensorimotor deficit of the upper and lower limbs in patients with type 2 diabetes.

diabetic neuropathy diffusion tensor imaging fractional anisotropy magnetic resonance neurography neurofilament light chain protein quantitative sensory testing

Journal

Frontiers in endocrinology
ISSN: 1664-2392
Titre abrégé: Front Endocrinol (Lausanne)
Pays: Switzerland
ID NLM: 101555782

Informations de publication

Date de publication:
2023
Historique:
received: 16 09 2022
accepted: 06 02 2023
medline: 4 4 2023
entrez: 3 4 2023
pubmed: 4 4 2023
Statut: epublish

Résumé

Diabetic sensorimotor polyneuropathy (DSPN) is one of the most prevalent and poorly understood diabetic microvascular complications. Recent studies have found that fractional anisotropy (FA), a marker for microstructural nerve integrity, is a sensitive parameter for the structural and functional nerve damage in DSPN. The aim of this study was to investigate the significance of proximal sciatic nerve's FA on different distal nerve fiber deficits of the upper and lower limbs and its correlation with the neuroaxonal biomarker, neurofilament light chain protein (NfL). Sixty-nine patients with type 2 diabetes (T2DM) and 30 healthy controls underwent detailed clinical and electrophysiological assessments, complete quantitative sensory testing (QST), and diffusion-weighted magnetic resonance neurography of the sciatic nerve. NfL was measured in the serum of healthy controls and patients with T2DM. Multivariate models were used to adjust for confounders of microvascular damage. Patients with DSPN showed a 17% lower sciatic microstructural integrity compared to healthy controls ( This is the first study showing that microstructural nerve integrity is associated with damage of different nerve fiber types and a neuroaxonal biomarker in DSPN. Furthermore, these findings show that proximal nerve damage is related to distal nerve function even before clinical symptoms occur. The microstructure of the proximal sciatic nerve and is also associated with functional nerve fiber deficits of the upper and lower limbs, suggesting that diabetic neuropathy involves structural changes of peripheral nerves of upper limbs too.

Sections du résumé

Background
Diabetic sensorimotor polyneuropathy (DSPN) is one of the most prevalent and poorly understood diabetic microvascular complications. Recent studies have found that fractional anisotropy (FA), a marker for microstructural nerve integrity, is a sensitive parameter for the structural and functional nerve damage in DSPN. The aim of this study was to investigate the significance of proximal sciatic nerve's FA on different distal nerve fiber deficits of the upper and lower limbs and its correlation with the neuroaxonal biomarker, neurofilament light chain protein (NfL).
Materials and methods
Sixty-nine patients with type 2 diabetes (T2DM) and 30 healthy controls underwent detailed clinical and electrophysiological assessments, complete quantitative sensory testing (QST), and diffusion-weighted magnetic resonance neurography of the sciatic nerve. NfL was measured in the serum of healthy controls and patients with T2DM. Multivariate models were used to adjust for confounders of microvascular damage.
Results
Patients with DSPN showed a 17% lower sciatic microstructural integrity compared to healthy controls (
Conclusion
This is the first study showing that microstructural nerve integrity is associated with damage of different nerve fiber types and a neuroaxonal biomarker in DSPN. Furthermore, these findings show that proximal nerve damage is related to distal nerve function even before clinical symptoms occur. The microstructure of the proximal sciatic nerve and is also associated with functional nerve fiber deficits of the upper and lower limbs, suggesting that diabetic neuropathy involves structural changes of peripheral nerves of upper limbs too.

Identifiants

pubmed: 37008917
doi: 10.3389/fendo.2023.1046690
pmc: PMC10053786
doi:

Substances chimiques

Biomarkers 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1046690

Informations de copyright

Copyright © 2023 Kender, Jende, Kurz, Tsilingiris, Schimpfle, Sulaj, von Rauchhaupt, Bartl, Mooshage, Göpfert, Nawroth, Herzig, Szendroedi, Bendszus and Kopf.

Déclaration de conflit d'intérêts

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

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Auteurs

Zoltan Kender (Z)

Department of Endocrinology, Diabetology and Clinical Chemistry (Internal Medicine 1), Heidelberg University Hospital, Heidelberg, Germany.
German Center of Diabetes Research [Deutsches Zentrum für Diabetesforschung (DZD)], München, Germany.

Johann M E Jende (JME)

Department of Neuroradiology, Heidelberg University Hospital, Heidelberg, Germany.

Felix T Kurz (FT)

Department of Neuroradiology, Heidelberg University Hospital, Heidelberg, Germany.
Department of Radiology, German Cancer Research Center, Heidelberg, Germany.

Dimitrios Tsilingiris (D)

Department of Endocrinology, Diabetology and Clinical Chemistry (Internal Medicine 1), Heidelberg University Hospital, Heidelberg, Germany.
German Center of Diabetes Research [Deutsches Zentrum für Diabetesforschung (DZD)], München, Germany.

Lukas Schimpfle (L)

Department of Endocrinology, Diabetology and Clinical Chemistry (Internal Medicine 1), Heidelberg University Hospital, Heidelberg, Germany.

Alba Sulaj (A)

Department of Endocrinology, Diabetology and Clinical Chemistry (Internal Medicine 1), Heidelberg University Hospital, Heidelberg, Germany.
German Center of Diabetes Research [Deutsches Zentrum für Diabetesforschung (DZD)], München, Germany.

Ekaterina von Rauchhaupt (E)

Department of Endocrinology, Diabetology and Clinical Chemistry (Internal Medicine 1), Heidelberg University Hospital, Heidelberg, Germany.
German Center of Diabetes Research [Deutsches Zentrum für Diabetesforschung (DZD)], München, Germany.

Hannelore Bartl (H)

Department of Endocrinology, Diabetology and Clinical Chemistry (Internal Medicine 1), Heidelberg University Hospital, Heidelberg, Germany.

Christoph Mooshage (C)

Department of Neuroradiology, Heidelberg University Hospital, Heidelberg, Germany.

Jens Göpfert (J)

NMI Natural and Medical Sciences Institute at the University of Tübingen, Reutlingen, Germany.

Peter Nawroth (P)

Department of Endocrinology, Diabetology and Clinical Chemistry (Internal Medicine 1), Heidelberg University Hospital, Heidelberg, Germany.
German Center of Diabetes Research [Deutsches Zentrum für Diabetesforschung (DZD)], München, Germany.
Joint-IDC Institute for Diabetes and Cancer, Heidelberg University, Heidelberg, Germany.

Stephan Herzig (S)

German Center of Diabetes Research [Deutsches Zentrum für Diabetesforschung (DZD)], München, Germany.
Joint-IDC Institute for Diabetes and Cancer, Heidelberg University, Heidelberg, Germany.
Joint-IDC Institute for Diabetes and Cancer, Helmholtz-Zentrum Munich, Munich, Germany.

Julia Szendroedi (J)

Department of Endocrinology, Diabetology and Clinical Chemistry (Internal Medicine 1), Heidelberg University Hospital, Heidelberg, Germany.
German Center of Diabetes Research [Deutsches Zentrum für Diabetesforschung (DZD)], München, Germany.
Joint-IDC Institute for Diabetes and Cancer, Heidelberg University, Heidelberg, Germany.

Martin Bendszus (M)

Department of Neuroradiology, Heidelberg University Hospital, Heidelberg, Germany.

Stefan Kopf (S)

Department of Endocrinology, Diabetology and Clinical Chemistry (Internal Medicine 1), Heidelberg University Hospital, Heidelberg, Germany.
German Center of Diabetes Research [Deutsches Zentrum für Diabetesforschung (DZD)], München, Germany.

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