Grey matter correlates of dystonic soft signs in essential tremor.


Journal

Parkinsonism & related disorders
ISSN: 1873-5126
Titre abrégé: Parkinsonism Relat Disord
Pays: England
ID NLM: 9513583

Informations de publication

Date de publication:
Jul 2023
Historique:
received: 20 02 2023
revised: 15 05 2023
accepted: 22 05 2023
medline: 3 7 2023
pubmed: 28 5 2023
entrez: 28 5 2023
Statut: ppublish

Résumé

Questionable signs of dystonia are a common finding in patients with essential tremor (ET). Brain structural alterations in ET patients plus dystonic soft signs (ET + ds) in comparison to ET patients without dystonic soft signs (ET-ds) or patients with tremor associated with manifest dystonia (TAWD) have not been examined yet. Therefore, our study aims to explore alterations of brain grey matter in patients with ET + ds. A total of 68 elderly patients with ET-ds (n = 32), ET + ds (n = 20) or idiopathic cervical dystonia with dystonia associated action tremor of the upper limbs (TAWD, n = 16) and 42 age-matched healthy controls underwent a clinical and electrophysiological assessment and 3T MRI. For grey matter alterations T1 MRI images were analysed by voxel-based morphometry. Additionally, regression analyses with clinical parameters (tremor frequency, severity and disease duration) were performed. VBM showed a significant increase of grey matter in the right lentiform nucleus in ET + ds and TAWD compared to HC and ET-ds. Further, an increase of cortical grey matter in the middle frontal gyrus in ET + ds was shown. The hypertrophy of the lentiform nucleus in ET + ds was correlated with disease severity and duration. Patients with ET + ds showed grey matter brain structural alterations similar to TAWD. Our findings suggest an involvement of the basal ganglia-cortical loop in ET + ds which may indicate a pathophysiological similarity with TAWD rather than ET.

Sections du résumé

BACKGROUND BACKGROUND
Questionable signs of dystonia are a common finding in patients with essential tremor (ET). Brain structural alterations in ET patients plus dystonic soft signs (ET + ds) in comparison to ET patients without dystonic soft signs (ET-ds) or patients with tremor associated with manifest dystonia (TAWD) have not been examined yet. Therefore, our study aims to explore alterations of brain grey matter in patients with ET + ds.
METHODS METHODS
A total of 68 elderly patients with ET-ds (n = 32), ET + ds (n = 20) or idiopathic cervical dystonia with dystonia associated action tremor of the upper limbs (TAWD, n = 16) and 42 age-matched healthy controls underwent a clinical and electrophysiological assessment and 3T MRI. For grey matter alterations T1 MRI images were analysed by voxel-based morphometry. Additionally, regression analyses with clinical parameters (tremor frequency, severity and disease duration) were performed.
RESULTS RESULTS
VBM showed a significant increase of grey matter in the right lentiform nucleus in ET + ds and TAWD compared to HC and ET-ds. Further, an increase of cortical grey matter in the middle frontal gyrus in ET + ds was shown. The hypertrophy of the lentiform nucleus in ET + ds was correlated with disease severity and duration.
CONCLUSION CONCLUSIONS
Patients with ET + ds showed grey matter brain structural alterations similar to TAWD. Our findings suggest an involvement of the basal ganglia-cortical loop in ET + ds which may indicate a pathophysiological similarity with TAWD rather than ET.

Identifiants

pubmed: 37245277
pii: S1353-8020(23)00180-3
doi: 10.1016/j.parkreldis.2023.105457
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

105457

Informations de copyright

Copyright © 2023 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest None.

Auteurs

Dana Brinker (D)

Department of Neurology, UKSH, Christian-Albrechts University Kiel, Germany.

Oliver Granert (O)

Department of Neurology, UKSH, Christian-Albrechts University Kiel, Germany.

Felix Gövert (F)

Department of Neurology, UKSH, Christian-Albrechts University Kiel, Germany.

Inken Tödt (I)

Department of Neurology, UKSH, Christian-Albrechts University Kiel, Germany.

Alexander Baumann (A)

Department of Neurology, UKSH, Christian-Albrechts University Kiel, Germany.

Kirsten E Zeuner (KE)

Department of Neurology, UKSH, Christian-Albrechts University Kiel, Germany.

Robin Wolke (R)

Department of Neurology, UKSH, Christian-Albrechts University Kiel, Germany.

Günther Deuschl (G)

Department of Neurology, UKSH, Christian-Albrechts University Kiel, Germany.

Jos S Becktepe (JS)

Department of Neurology, UKSH, Christian-Albrechts University Kiel, Germany. Electronic address: j.becktepe@neurologie.uni-kiel.de.

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