The terminal oxidase cytochrome bd-I confers carbon monoxide resistance to Escherichia coli cells.


Journal

Journal of inorganic biochemistry
ISSN: 1873-3344
Titre abrégé: J Inorg Biochem
Pays: United States
ID NLM: 7905788

Informations de publication

Date de publication:
10 2023
Historique:
received: 12 04 2023
revised: 12 07 2023
accepted: 22 07 2023
medline: 21 8 2023
pubmed: 30 7 2023
entrez: 29 7 2023
Statut: ppublish

Résumé

Carbon monoxide (CO) plays a multifaceted role in the physiology of organisms, from poison to signaling molecule. Heme proteins, including terminal oxidases, are plausible CO targets. Three quinol oxidases terminate the branched aerobic respiratory chain of Escherichia coli. These are the heme‑copper cytochrome bo

Identifiants

pubmed: 37515940
pii: S0162-0134(23)00223-4
doi: 10.1016/j.jinorgbio.2023.112341
pii:
doi:

Substances chimiques

Carbon Monoxide 7U1EE4V452
Copper 789U1901C5
Escherichia coli Proteins 0
Cytochrome b Group 0
Electron Transport Chain Complex Proteins 0
Cytochromes 0
Oxidoreductases EC 1.-

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

112341

Informations de copyright

Copyright © 2023 The Authors. Published by Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Martina R Nastasi (MR)

Department of Biochemical Sciences, Sapienza University of Rome, I-00185 Rome, Italy.

Vitaliy B Borisov (VB)

Belozersky Institute of Physico-Chemical Biology, Lomonosov Moscow State University, 119991 Moscow, Russia. Electronic address: bor@belozersky.msu.ru.

Elena Forte (E)

Department of Biochemical Sciences, Sapienza University of Rome, I-00185 Rome, Italy. Electronic address: elena.forte@uniroma1.it.

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Classifications MeSH