Oxidative stress and activation of H2A.X in lung alveolar epithelial cells (A549) by nanoparticulate carbon black.
A549
Air particulate matter
DNA damage
Histone H2A.X
Oxidative stress
Journal
Respiratory physiology & neurobiology
ISSN: 1878-1519
Titre abrégé: Respir Physiol Neurobiol
Pays: Netherlands
ID NLM: 101140022
Informations de publication
Date de publication:
10 2023
10 2023
Historique:
received:
19
05
2023
revised:
06
08
2023
accepted:
13
08
2023
medline:
6
9
2023
pubmed:
17
8
2023
entrez:
16
8
2023
Statut:
ppublish
Résumé
Fine airborne particulate matter enter the respiratory system, induce oxidative stress and initiate DNA damage. The aim of our study was the estimation of cell viability, oxidative stress, DNA damage, cell cycle alterations and activation of histone H2A.X. Experiments were done on lung alveolar epithelial (A549) cells grown for 24 h with 200 µg mL
Identifiants
pubmed: 37586603
pii: S1569-9048(23)00128-3
doi: 10.1016/j.resp.2023.104140
pii:
doi:
Substances chimiques
Histones
0
Soot
0
RNA, Messenger
0
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
104140Informations de copyright
Copyright © 2023 The Authors. Published by Elsevier B.V. All rights reserved.