Design, molecular modelling and synthesis of novel benzothiazole derivatives as BCL-2 inhibitors.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
20 09 2023
Historique:
received: 09 03 2023
accepted: 31 08 2023
medline: 22 9 2023
pubmed: 21 9 2023
entrez: 20 9 2023
Statut: epublish

Résumé

Apoptosis plays a crucial role in cancer pathogenesis and drug resistance. BCL-2 family of enzymes is considered as one of the key enzymes which is involved in apoptosis. When there is disruption in the balance between anti-apoptotic and pro-apoptotic members of the BCL-2 family apoptosis is dysregulated in the affected cells. Herein, 33 novel benzothiazole-based molecules 7a-i, 8a-f, 9a-b, 12a-e, 13a-d, 14a,b, and 17a-j were designed, synthesized and tested for their BCL-2 inhibitory activity. Scaffold hopping strategy was applied in designing of the target compounds. Compounds 13c and 13d showed the highest activity with IC

Identifiants

pubmed: 37730790
doi: 10.1038/s41598-023-41783-1
pii: 10.1038/s41598-023-41783-1
pmc: PMC10511702
doi:

Substances chimiques

Benzothiazoles 0
Antineoplastic Agents 0
Proto-Oncogene Proteins c-bcl-2 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

15554

Informations de copyright

© 2023. Springer Nature Limited.

Références

Avendaño, C. & Menéndez, J. C. Medicinal Chemistry of Anticancer Drugs 2nd edn, 1–740 (Elsevier, 2015).
doi: 10.1016/B978-0-444-62649-3.00001-6
Hanahan, D. & Weinberg, R. A. Hallmarks of cancer: The next generation. Cell 144(5), 646–674 (2011).
doi: 10.1016/j.cell.2011.02.013 pubmed: 21376230
Renehan, A. G., Booth, C. & Potten, C. S. What is apoptosis, and why is it important?. BMJ 322(7301), 1536–1538 (2001).
doi: 10.1136/bmj.322.7301.1536 pubmed: 11420279 pmcid: 1120576
Soliman, A. M. et al. Induction of apoptosis, cytotoxicity and radiosensitization by novel 3,4-dihydroquinazolinone derivatives. Bioorg. Med. Chem. Lett. 49, 128308 (2021).
doi: 10.1016/j.bmcl.2021.128308 pubmed: 34363937
Wong, R. S. Y. Apoptosis in cancer: From pathogenesis to treatment. J. Exp. Clin. Cancer Res. 30(1), 87 (2011).
doi: 10.1186/1756-9966-30-87 pubmed: 21943236 pmcid: 3197541
Kasibhatla, S. & Tseng, B. Why target apoptosis in cancer treatment?. Mol. Cancer Ther. 2(6), 573–580 (2003).
pubmed: 12813137
Boice, A. & Bouchier-Hayes, L. Targeting apoptotic caspases in cancer. Biochim. Biophys. Acta Mol. Cell Res. 1867(6), 118688 (2020).
doi: 10.1016/j.bbamcr.2020.118688 pubmed: 32087180 pmcid: 7155770
Van Opdenbosch, N. & Lamkanfi, M. Caspases in cell death, inflammation, and disease. Immunity 50(6), 1352–1364 (2019).
doi: 10.1016/j.immuni.2019.05.020 pubmed: 31216460 pmcid: 6611727
Garrido, C. et al. Mechanisms of cytochrome c release from mitochondria. Cell Death Differ. 13(9), 1423–1433 (2006).
doi: 10.1038/sj.cdd.4401950 pubmed: 16676004
Pfeffer, C. M. & Singh, A. T. K. Apoptosis: A target for anticancer therapy. Int. J. Mol. Sci. 19, E448. https://doi.org/10.3390/ijms19020448 (2018).
doi: 10.3390/ijms19020448
Reed, J. C. & Pellecchia, M. Apoptosis-based therapies for hematologic malignancies. Blood 106(2), 408–418 (2005).
doi: 10.1182/blood-2004-07-2761 pubmed: 15797997
Adams, J. M. & Cory, S. The Bcl-2 apoptotic switch in cancer development and therapy. Oncogene 26(9), 1324–1337 (2007).
doi: 10.1038/sj.onc.1210220 pubmed: 17322918 pmcid: 2930981
Kale, J., Osterlund, E. J. & Andrews, D. W. BCL-2 family proteins: Changing partners in the dance towards death. Cell Death Differ. 25(1), 65–80 (2018).
doi: 10.1038/cdd.2017.186 pubmed: 29149100
Warren, C. F. A., Wong-Brown, M. W. & Bowden, N. A. BCL-2 family isoforms in apoptosis and cancer. Cell Death Dis. 10(3), 177 (2019).
doi: 10.1038/s41419-019-1407-6 pubmed: 30792387 pmcid: 6384907
Kroemer, G., Galluzzi, L. & Brenner, C. Mitochondrial membrane permeabilization in cell death. Physiol. Rev. 87(1), 99–163 (2007).
doi: 10.1152/physrev.00013.2006 pubmed: 17237344
Hata, A. N., Engelman, J. A. & Faber, A. C. The BCL2 family: Key mediators of the apoptotic response to targeted anticancer therapeutics. Cancer Discov. 5(5), 475–487 (2015).
doi: 10.1158/2159-8290.CD-15-0011 pubmed: 25895919 pmcid: 4727530
Kvansakul, M. & Hinds, M. G. The Bcl-2 family: Structures, interactions and targets for drug discovery. Apoptosis 20(2), 136–150 (2015).
doi: 10.1007/s10495-014-1051-7 pubmed: 25398535
Chan, S. L. & Yu, V. C. Proteins of the bcl-2 family in apoptosis signalling: From mechanistic insights to therapeutic opportunities. Clin. Exp. Pharmacol. Physiol. 31(3), 119–128 (2004).
doi: 10.1111/j.1440-1681.2004.03975.x pubmed: 15008953
Youle, R. J. & Strasser, A. The BCL-2 protein family: Opposing activities that mediate cell death. Nat. Rev. Mol. Cell Biol. 9(1), 47–59 (2008).
doi: 10.1038/nrm2308 pubmed: 18097445
Obeng, E. Apoptosis (programmed cell death) and its signals—A review. Braz J Biol 81(4), 1133–1143 (2021).
doi: 10.1590/1519-6984.228437 pubmed: 33111928
Shimizu, S. et al. Role of Bcl-2 family proteins in a non-apoptotic programmed cell death dependent on autophagy genes. Nat. Cell Biol. 6(12), 1221–1228 (2004).
doi: 10.1038/ncb1192 pubmed: 15558033
Reed, J. S. et al. The role of MHC class I allele Mamu-A*07 during SIV(mac)239 infection. Immunogenetics 63(12), 789–807 (2011).
doi: 10.1007/s00251-011-0541-9 pubmed: 21732180 pmcid: 3706270
Khan, K. H., Blanco-Codesido, M. & Molife, L. R. Cancer therapeutics: Targeting the apoptotic pathway. Crit. Rev. Oncol. Hematol. 90(3), 200–219 (2014).
doi: 10.1016/j.critrevonc.2013.12.012 pubmed: 24507955
Kapoor, I. et al. Targeting BCL-2 in B-cell malignancies and overcoming therapeutic resistance. Cell Death Dis. 11(11), 941 (2020).
doi: 10.1038/s41419-020-03144-y pubmed: 33139702 pmcid: 7608616
Ishikawa, M. & Hashimoto, Y. Improvement in aqueous solubility in small molecule drug discovery programs by disruption of molecular planarity and symmetry. J. Med. Chem. 54(6), 1539–1554 (2011).
doi: 10.1021/jm101356p pubmed: 21344906
Sleebs, B. E. et al. Quinazoline sulfonamides as dual binders of the proteins B-cell lymphoma 2 and B-cell lymphoma extra long with potent proapoptotic cell-based activity. J. Med. Chem. 54(6), 1914–1926 (2011).
doi: 10.1021/jm101596e pubmed: 21366295
Perez, H. L. et al. Identification of a phenylacylsulfonamide series of dual Bcl-2/Bcl-xL antagonists. Bioorg. Med. Chem. Lett. 22(12), 3946–3950 (2012).
doi: 10.1016/j.bmcl.2012.04.103 pubmed: 22608961
Schroeder, G. M. et al. Pyrazole and pyrimidine phenylacylsulfonamides as dual Bcl-2/Bcl-xL antagonists. Bioorg. Med. Chem. Lett. 22(12), 3951–3956 (2012).
doi: 10.1016/j.bmcl.2012.04.106 pubmed: 22608393
Touré, B. B. et al. The role of the acidity of N-heteroaryl sulfonamides as inhibitors of bcl-2 family protein-protein interactions. ACS Med. Chem. Lett. 4(2), 186–190 (2013).
doi: 10.1021/ml300321d pubmed: 24900652 pmcid: 4027142
Filippakopoulos, P. et al. Histone recognition and large-scale structural analysis of the human bromodomain family. Cell 149(1), 214–231 (2012).
doi: 10.1016/j.cell.2012.02.013 pubmed: 22464331 pmcid: 3326523
Berman, H. M. et al. The protein data bank. Nucleic Acids Res. 28(1), 235–242 (2000).
doi: 10.1093/nar/28.1.235 pubmed: 10592235 pmcid: 102472
Pettersen, E. F. et al. UCSF Chimera–a visualization system for exploratory research and analysis. J. Comput. Chem. 25(13), 1605–1612 (2004).
doi: 10.1002/jcc.20084 pubmed: 15264254
Gibson, C. L. et al. Diversity oriented syntheses of fused pyrimidines designed as potential antifolates. Org. Biomol. Chem. 7(9), 1829–1842 (2009).
doi: 10.1039/b818339b pubmed: 19590778
Leow, M. L. et al. Benzofuran-based estrogen receptor α modulators as anti-cancer therapeutics: In silico and experimental studies. Curr. Med. Chem. 20(22), 2820–2837 (2013).
doi: 10.2174/0929867311320220007 pubmed: 23531218
González-Alvarez, M. et al. Development of novel copper(II) complexes of benzothiazole- N-sulfonamides as protective agents against superoxide anion. Crystal structures of [Cu( N-2-(4-methylbenzothiazole)benzenesulfonamidate)(2)(py)(2)] and [Cu( N-2-(6-nitrobenzothiazole)naphthalenesulfonamidate)(2)(py)(2)]. J. Biol. Inorg. Chem. 8(12), 112–20 (2003).
doi: 10.1007/s00775-002-0394-7 pubmed: 12459905
Manoharan, D. et al. Synthesis, characterization and evaluation of antidiabetic activity of novel indoline derivatives. Bangladesh J. Pharmacol. 12, 20 (2017).
doi: 10.3329/bjp.v12i2.30872
Shoemaker, R. H. The NCI60 human tumour cell line anticancer drug screen. Nat. Rev. Cancer 6(10), 813–823 (2006).
doi: 10.1038/nrc1951 pubmed: 16990858
Wu, G. et al. Detailed analysis of grid-based molecular docking: A case study of CDOCKER-A CHARMm-based MD docking algorithm. J. Comput. Chem. 24(13), 1549–1562 (2003).
doi: 10.1002/jcc.10306 pubmed: 12925999

Auteurs

Hoda S Ismail (HS)

Pharmaceutical Chemistry Department, Faculty of Pharmacy, Ain Shams University, Cairo, Egypt.

Amira Khalil (A)

Pharmaceutical Chemistry Department, Faculty of Pharmacy, The British University in Egypt (BUE), El-Sherouk City, Cairo, 11837, Egypt. Amira.Khalil@bue.edu.eg.

Rabah A Taha (RA)

Pharmaceutical Chemistry Department, Faculty of Pharmacy, Ain Shams University, Cairo, Egypt.

Deena S Lasheen (DS)

Pharmaceutical Chemistry Department, Faculty of Pharmacy, Ain Shams University, Cairo, Egypt.

Dalal A Abou El Ella (DA)

Pharmaceutical Chemistry Department, Faculty of Pharmacy, Ain Shams University, Cairo, Egypt. dalal@pharma.asu.edu.eg.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs
Animals Hemiptera Insect Proteins Phylogeny Insecticides
Humans Male Female Health Knowledge, Attitudes, Practice Middle Aged
STAT3 Transcription Factor Respiratory Syncytial Virus Infections Humans Animals Mice

Classifications MeSH