Inhibition of fatty acid oxidation enables heart regeneration in adult mice.


Journal

Nature
ISSN: 1476-4687
Titre abrégé: Nature
Pays: England
ID NLM: 0410462

Informations de publication

Date de publication:
Oct 2023
Historique:
received: 05 08 2021
accepted: 30 08 2023
medline: 23 10 2023
pubmed: 28 9 2023
entrez: 27 9 2023
Statut: ppublish

Résumé

Postnatal maturation of cardiomyocytes is characterized by a metabolic switch from glycolysis to fatty acid oxidation, chromatin reconfiguration and exit from the cell cycle, instating a barrier for adult heart regeneration

Identifiants

pubmed: 37758950
doi: 10.1038/s41586-023-06585-5
pii: 10.1038/s41586-023-06585-5
pmc: PMC10584682
doi:

Substances chimiques

Carnitine O-Palmitoyltransferase EC 2.3.1.21
CPT1B protein, mouse EC 2.3.1.21
Fatty Acids 0
Histone Demethylases EC 1.14.11.-
Ketoglutaric Acids 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

619-626

Commentaires et corrections

Type : ErratumIn

Informations de copyright

© 2023. The Author(s).

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Auteurs

Xiang Li (X)

Department of Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Fan Wu (F)

Department of Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Stefan Günther (S)

Department of Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Mario Looso (M)

Department of Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Carsten Kuenne (C)

Department of Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Ting Zhang (T)

Department of Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Marion Wiesnet (M)

Department of Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Stephan Klatt (S)

Institute for Vascular Signaling, Centre for Molecular Medicine, Goethe-University, Frankfurt am Main, Germany.

Sven Zukunft (S)

Institute for Vascular Signaling, Centre for Molecular Medicine, Goethe-University, Frankfurt am Main, Germany.

Ingrid Fleming (I)

Institute for Vascular Signaling, Centre for Molecular Medicine, Goethe-University, Frankfurt am Main, Germany.

Gernot Poschet (G)

Metabolomics Core Technology Platform, Centre for Organismal Studies (COS), Heidelberg University, Heidelberg, Germany.

Astrid Wietelmann (A)

Department of Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Ann Atzberger (A)

Department of Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.

Michael Potente (M)

Angiogenesis and Metabolism Laboratory, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany.
Max Delbrück Center for Molecular Medicine, Helmholtz Association of German Research Centres, Berlin, Germany.
Berlin Institute of Health, Charité-Universitätsmedizin Berlin, Berlin, Germany.

Xuejun Yuan (X)

Department of Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany. Xuejun.Yuan@mpi-bn.mpg.de.
Instituto de Investigacion en Biomedicina de Buenos Aires (IBioBA) - CONICET - Partner Institute of the Max Planck Society, Buenos Aires, Argentina. Xuejun.Yuan@mpi-bn.mpg.de.

Thomas Braun (T)

Department of Cardiac Development and Remodeling, Max Planck Institute for Heart and Lung Research, Bad Nauheim, Germany. Thomas.Braun@mpi-bn.mpg.de.
Instituto de Investigacion en Biomedicina de Buenos Aires (IBioBA) - CONICET - Partner Institute of the Max Planck Society, Buenos Aires, Argentina. Thomas.Braun@mpi-bn.mpg.de.

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