Histones with an unconventional DNA-binding mode in vitro are major chromatin constituents in the bacterium Bdellovibrio bacteriovorus.


Journal

Nature microbiology
ISSN: 2058-5276
Titre abrégé: Nat Microbiol
Pays: England
ID NLM: 101674869

Informations de publication

Date de publication:
Nov 2023
Historique:
received: 03 05 2023
accepted: 08 09 2023
medline: 8 11 2023
pubmed: 10 10 2023
entrez: 9 10 2023
Statut: ppublish

Résumé

Histone proteins bind DNA and organize the genomes of eukaryotes and most archaea, whereas bacteria rely on different nucleoid-associated proteins. Homology searches have detected putative histone-fold domains in a few bacteria, but whether these function like archaeal/eukaryotic histones is unknown. Here we report that histones are major chromatin components in the bacteria Bdellovibrio bacteriovorus and Leptospira interrogans. Patterns of sequence evolution suggest important roles for histones in additional bacterial clades. Crystal structures (<2.0 Å) of the B. bacteriovorus histone (Bd0055) dimer and the histone-DNA complex confirm conserved histone-fold topology but indicate a distinct DNA-binding mode. Unlike known histones in eukaryotes, archaea and viruses, Bd0055 binds DNA end-on, forming a sheath of dimers encasing straight DNA rather than wrapping DNA around their outer surface. Our results demonstrate that histones are present across the tree of life and highlight potential evolutionary innovation in how they associate with DNA.

Identifiants

pubmed: 37814071
doi: 10.1038/s41564-023-01492-x
pii: 10.1038/s41564-023-01492-x
pmc: PMC10627809
doi:

Substances chimiques

Histones 0
Chromatin 0
DNA 9007-49-2

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2006-2019

Subventions

Organisme : Wellcome Trust
Pays : United Kingdom

Informations de copyright

© 2023. The Author(s).

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Auteurs

Antoine Hocher (A)

Medical Research Council London Institute of Medical Sciences, London, UK. a.hocher@lms.mrc.ac.uk.
Institute of Clinical Sciences, Faculty of Medicine, Imperial College London, London, UK. a.hocher@lms.mrc.ac.uk.

Shawn P Laursen (SP)

Department of Molecular, Cellular, and Developmental Biology, University of Colorado Boulder, Boulder, CO, USA.

Paul Radford (P)

School of Life Sciences, Medical School, Queen's Medical Centre, University of Nottingham, Nottingham, UK.

Jess Tyson (J)

School of Life Sciences, Medical School, Queen's Medical Centre, University of Nottingham, Nottingham, UK.

Carey Lambert (C)

School of Life Sciences, Medical School, Queen's Medical Centre, University of Nottingham, Nottingham, UK.

Kathryn M Stevens (KM)

Medical Research Council London Institute of Medical Sciences, London, UK.
Institute of Clinical Sciences, Faculty of Medicine, Imperial College London, London, UK.

Alex Montoya (A)

Medical Research Council London Institute of Medical Sciences, London, UK.
Institute of Clinical Sciences, Faculty of Medicine, Imperial College London, London, UK.

Pavel V Shliaha (PV)

Medical Research Council London Institute of Medical Sciences, London, UK.
Institute of Clinical Sciences, Faculty of Medicine, Imperial College London, London, UK.

Mathieu Picardeau (M)

Institut Pasteur, Université Paris Cité, CNRS UMR 6047, Biology of Spirochetes Unit, Paris, France.

R Elizabeth Sockett (RE)

School of Life Sciences, Medical School, Queen's Medical Centre, University of Nottingham, Nottingham, UK.

Karolin Luger (K)

Department of Biochemistry, University of Colorado Boulder, Boulder, CO, USA. karolin.luger@colorado.edu.
Howard Hughes Medical Institute, Chevy Chase, MD, USA. karolin.luger@colorado.edu.

Tobias Warnecke (T)

Medical Research Council London Institute of Medical Sciences, London, UK. tobias.warnecke@lms.mrc.ac.uk.
Institute of Clinical Sciences, Faculty of Medicine, Imperial College London, London, UK. tobias.warnecke@lms.mrc.ac.uk.

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