Reduction of p11 in dorsal raphe nucleus serotonergic neurons mediates depression-like behaviors.


Journal

Translational psychiatry
ISSN: 2158-3188
Titre abrégé: Transl Psychiatry
Pays: United States
ID NLM: 101562664

Informations de publication

Date de publication:
22 Nov 2023
Historique:
received: 08 08 2023
accepted: 09 11 2023
revised: 13 10 2023
medline: 24 11 2023
pubmed: 23 11 2023
entrez: 22 11 2023
Statut: epublish

Résumé

The pathology of depression is related to the imbalance of various neurotransmitters. The dorsal raphe nucleus (DRN), the main brain region producing 5-HT, is crucially involved in the pathophysiology of depression. It contains several neuron types, in which GABAergic neurons are activated by stimuli associated with negative experiences and 5-HT neurons are activated by reward signals. However, little is known about its underlying molecular mechanisms. Here, we found that p11, a multifunctional protein associated with depression, was down-regulated by chronic social defeat stress in 5-HT

Identifiants

pubmed: 37993435
doi: 10.1038/s41398-023-02664-3
pii: 10.1038/s41398-023-02664-3
pmc: PMC10665321
doi:

Substances chimiques

Serotonin 333DO1RDJY

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

359

Subventions

Organisme : National Natural Science Foundation of China (National Science Foundation of China)
ID : 81973943
Organisme : National Natural Science Foundation of China (National Science Foundation of China)
ID : 82274643
Organisme : National Natural Science Foundation of China (National Science Foundation of China)
ID : 82104986
Organisme : National Natural Science Foundation of China (National Science Foundation of China)
ID : 82174499

Informations de copyright

© 2023. The Author(s).

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Auteurs

Wei Li (W)

Department of Acupuncture and Moxibustion, Shanghai Municipal Hospital of Traditional Chinese Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, 200071, China.
Department of Integrative Medicine and Neurobiology, School of Basic Medical Sciences, Shanghai Medical College, Fudan University, Shanghai, 200032, China.
Shanghai Key Laboratory of Acupuncture Mechanism and Acupoint Function, Fudan University, Shanghai, 200433, China.

Zuqi Shen (Z)

Department of Integrative Medicine and Neurobiology, School of Basic Medical Sciences, Shanghai Medical College, Fudan University, Shanghai, 200032, China.
Shanghai Key Laboratory of Acupuncture Mechanism and Acupoint Function, Fudan University, Shanghai, 200433, China.

Xuan Yin (X)

Department of Acupuncture and Moxibustion, Shanghai Municipal Hospital of Traditional Chinese Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, 200071, China.

Weiqi Chang (W)

Department of Integrative Medicine and Neurobiology, School of Basic Medical Sciences, Shanghai Medical College, Fudan University, Shanghai, 200032, China.
Shanghai Key Laboratory of Acupuncture Mechanism and Acupoint Function, Fudan University, Shanghai, 200433, China.

Xiaorong Chen (X)

Department of Physiology, Laboratory of Neurodegenerative diseases, Changzhi Medical College, Changzhi, Shanxi, 046000, China.

Jin Yu (J)

Department of Integrative Medicine and Neurobiology, School of Basic Medical Sciences, Shanghai Medical College, Fudan University, Shanghai, 200032, China. yujin@shmu.edu.cn.
Shanghai Key Laboratory of Acupuncture Mechanism and Acupoint Function, Fudan University, Shanghai, 200433, China. yujin@shmu.edu.cn.

Shifen Xu (S)

Department of Acupuncture and Moxibustion, Shanghai Municipal Hospital of Traditional Chinese Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, 200071, China. xu_teacher2006@126.com.

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Classifications MeSH