An RNA pseudoknot mediates toxin translation and antitoxin inhibition.


Journal

Proceedings of the National Academy of Sciences of the United States of America
ISSN: 1091-6490
Titre abrégé: Proc Natl Acad Sci U S A
Pays: United States
ID NLM: 7505876

Informations de publication

Date de publication:
02 Jul 2024
Historique:
medline: 27 6 2024
pubmed: 27 6 2024
entrez: 27 6 2024
Statut: ppublish

Résumé

Type I toxin-antitoxin systems (T1TAs) are bipartite bacterial loci encoding a growth-inhibitory toxin and an antitoxin small RNA (sRNA). In many of these systems, the transcribed toxin mRNA is translationally inactive, but becomes translation-competent upon ribonucleolytic processing. The antitoxin sRNA targets the processed mRNA to inhibit its translation. This two-level control mechanism prevents cotranscriptional translation of the toxin and allows its synthesis only when the antitoxin is absent. Contrary to this, we found that the

Identifiants

pubmed: 38935561
doi: 10.1073/pnas.2403063121
doi:

Substances chimiques

RNA, Bacterial 0
RNA, Messenger 0
Bacterial Toxins 0
Antitoxins 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e2403063121

Subventions

Organisme : Vetenskapsr&ådet (VR)
ID : 2021-04657
Organisme : Uppsala Antibiotic Center
ID : 2021

Déclaration de conflit d'intérêts

Competing interests statement:The authors declare no competing interest.

Auteurs

Athina Eleftheraki (A)

Department of Cell and Molecular Biology, Biomedical Center, Uppsala University, Uppsala 75124, Sweden.
Uppsala Antibiotic Center, Uppsala University, Uppsala 75123, Sweden.

Erik Holmqvist (E)

Department of Cell and Molecular Biology, Biomedical Center, Uppsala University, Uppsala 75124, Sweden.
Uppsala Antibiotic Center, Uppsala University, Uppsala 75123, Sweden.

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Classifications MeSH