Importance of hospital and clinical factors for early mortality in Takotsubo syndrome: Insights from the Swedish Coronary Angiography and Angioplasty Registry.


Journal

BMC cardiovascular disorders
ISSN: 1471-2261
Titre abrégé: BMC Cardiovasc Disord
Pays: England
ID NLM: 100968539

Informations de publication

Date de publication:
15 Jul 2024
Historique:
received: 16 04 2024
accepted: 01 07 2024
medline: 15 7 2024
pubmed: 15 7 2024
entrez: 14 7 2024
Statut: epublish

Résumé

Takotsubo syndrome (TTS) is an acute heart failure syndrome with symptoms similar to acute myocardial infarction. TTS is often triggered by acute emotional or physical stress and is a significant cause of morbidity and mortality. Predictors of mortality in patients with TS are not well understood, and there is a need to identify high-risk patients and tailor treatment accordingly. This study aimed to assess the importance of various clinical factors in predicting 30-day mortality in TTS patients using a machine learning algorithm. We analyzed data from the nationwide Swedish Coronary Angiography and Angioplasty Registry (SCAAR) for all patients with TTS in Sweden between 2015 and 2022. Gradient boosting was used to assess the relative importance of variables in predicting 30-day mortality in TTS patients. Of 3,180 patients hospitalized with TTS, 76.0% were women. The median age was 71.0 years (interquartile range 62-77). The crude all-cause mortality rate was 3.2% at 30 days. Machine learning algorithms by gradient boosting identified treating hospitals as the most important predictor of 30-day mortality. This factor was followed in significance by the clinical indication for angiography, creatinine level, Killip class, and age. Other less important factors included weight, height, and certain medical conditions such as hyperlipidemia and smoking status. Using machine learning with gradient boosting, we analyzed all Swedish patients diagnosed with TTS over seven years and found that the treating hospital was the most significant predictor of 30-day mortality.

Sections du résumé

BACKGROUND BACKGROUND
Takotsubo syndrome (TTS) is an acute heart failure syndrome with symptoms similar to acute myocardial infarction. TTS is often triggered by acute emotional or physical stress and is a significant cause of morbidity and mortality. Predictors of mortality in patients with TS are not well understood, and there is a need to identify high-risk patients and tailor treatment accordingly. This study aimed to assess the importance of various clinical factors in predicting 30-day mortality in TTS patients using a machine learning algorithm.
METHODS METHODS
We analyzed data from the nationwide Swedish Coronary Angiography and Angioplasty Registry (SCAAR) for all patients with TTS in Sweden between 2015 and 2022. Gradient boosting was used to assess the relative importance of variables in predicting 30-day mortality in TTS patients.
RESULTS RESULTS
Of 3,180 patients hospitalized with TTS, 76.0% were women. The median age was 71.0 years (interquartile range 62-77). The crude all-cause mortality rate was 3.2% at 30 days. Machine learning algorithms by gradient boosting identified treating hospitals as the most important predictor of 30-day mortality. This factor was followed in significance by the clinical indication for angiography, creatinine level, Killip class, and age. Other less important factors included weight, height, and certain medical conditions such as hyperlipidemia and smoking status.
CONCLUSIONS CONCLUSIONS
Using machine learning with gradient boosting, we analyzed all Swedish patients diagnosed with TTS over seven years and found that the treating hospital was the most significant predictor of 30-day mortality.

Identifiants

pubmed: 39004698
doi: 10.1186/s12872-024-04023-6
pii: 10.1186/s12872-024-04023-6
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

359

Informations de copyright

© 2024. The Author(s).

Références

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Auteurs

Thorsteinn Gudmundsson (T)

Department of Cardiology, Department of Molecular and Clinical Medicine, Sahlgrenska University Hospital Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Bruna stråket 16, Gothenburg, 41345, Sweden.

Björn Redfors (B)

Department of Cardiology, Department of Molecular and Clinical Medicine, Sahlgrenska University Hospital Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Bruna stråket 16, Gothenburg, 41345, Sweden.

Truls Råmunddal (T)

Department of Cardiology, Department of Molecular and Clinical Medicine, Sahlgrenska University Hospital Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Bruna stråket 16, Gothenburg, 41345, Sweden.

Oskar Angerås (O)

Department of Cardiology, Department of Molecular and Clinical Medicine, Sahlgrenska University Hospital Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Bruna stråket 16, Gothenburg, 41345, Sweden.

Petur Petursson (P)

Department of Cardiology, Department of Molecular and Clinical Medicine, Sahlgrenska University Hospital Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Bruna stråket 16, Gothenburg, 41345, Sweden.

Araz Rawshani (A)

Department of Cardiology, Department of Molecular and Clinical Medicine, Sahlgrenska University Hospital Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Bruna stråket 16, Gothenburg, 41345, Sweden.

Henrik Hagström (H)

Department of Cardiology, Umeå University Hospital, Umeå, Sweden.

Joakim Alfredsson (J)

Department of Cardiology, University Hospital, Linköping, Sweden.

Christina Ekenbäck (C)

Department of Cardiology, Danderyd University Hospital, Stockholm, Sweden.

Loghman Henareh (L)

Department of Cardiology, Karolinska University Hospital, Stockholm, Sweden.

Kristofer Skoglund (K)

Department of Cardiology, Department of Molecular and Clinical Medicine, Sahlgrenska University Hospital Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Bruna stråket 16, Gothenburg, 41345, Sweden.

Charlotta Ljungman (C)

Department of Cardiology, Department of Molecular and Clinical Medicine, Sahlgrenska University Hospital Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Bruna stråket 16, Gothenburg, 41345, Sweden.

Moman Mohammad (M)

Department of Cardiology, Skåne University Hospital, Lund, Sweden.

Tomas Jernberg (T)

Department of Cardiology, Karolinska University Hospital, Stockholm, Sweden.

Ole Fröbert (O)

Department of Cardiology, Örebro University Hospital, Örebro, Sweden.

David Erlinge (D)

Department of Cardiology, Skåne University Hospital, Lund, Sweden.

Elmir Omerovic (E)

Department of Cardiology, Department of Molecular and Clinical Medicine, Sahlgrenska University Hospital Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Bruna stråket 16, Gothenburg, 41345, Sweden. elmir@wlab.gu.se.

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