The influence of HLA genetic variation on plasma protein expression.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
31 Jul 2024
Historique:
received: 18 08 2023
accepted: 15 07 2024
medline: 1 8 2024
pubmed: 1 8 2024
entrez: 31 7 2024
Statut: epublish

Résumé

Genetic variation in the human leukocyte antigen (HLA) loci is associated with risk of immune-mediated diseases, but the molecular effects of HLA polymorphism are unclear. Here we examined the effects of HLA genetic variation on the expression of 2940 plasma proteins across 45,330 Europeans in the UK Biobank, with replication analyses across multiple ancestry groups. We detected 504 proteins affected by HLA variants (HLA-pQTL), including widespread trans effects by autoimmune disease risk alleles. More than 80% of the HLA-pQTL fine-mapped to amino acid positions in the peptide binding groove. HLA-I and II affected proteins expressed in similar cell types but in different pathways of both adaptive and innate immunity. Finally, we investigated potential HLA-pQTL effects on disease by integrating HLA-pQTL with fine-mapped HLA-disease signals in the UK Biobank. Our data reveal the diverse effects of HLA genetic variation and aid the interpretation of associations between HLA alleles and immune-mediated diseases.

Identifiants

pubmed: 39085222
doi: 10.1038/s41467-024-50583-8
pii: 10.1038/s41467-024-50583-8
doi:

Substances chimiques

HLA Antigens 0
Blood Proteins 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

6469

Informations de copyright

© 2024. The Author(s).

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Auteurs

Chirag Krishna (C)

Pfizer Research and Development, Pfizer Inc., Cambridge, MA, USA. Chirag.Krishna@pfizer.com.

Joshua Chiou (J)

Pfizer Research and Development, Pfizer Inc., Cambridge, MA, USA.

Saori Sakaue (S)

Center for Data Sciences, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.
Divisions of Genetics and Rheumatology, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.
Program in Medical and Population Genetics, Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Department of Biomedical Informatics, Harvard Medical School, Boston, MA, USA.

Joyce B Kang (JB)

Center for Data Sciences, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.
Divisions of Genetics and Rheumatology, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.
Program in Medical and Population Genetics, Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Department of Biomedical Informatics, Harvard Medical School, Boston, MA, USA.

Stephen M Christensen (SM)

Pfizer Research and Development, Pfizer Inc., Cambridge, MA, USA.

Isac Lee (I)

Pfizer Research and Development, Pfizer Inc., Cambridge, MA, USA.

Melis Atalar Aksit (MA)

Pfizer Research and Development, Pfizer Inc., Cambridge, MA, USA.

Hye In Kim (HI)

Pfizer Research and Development, Pfizer Inc., Cambridge, MA, USA.

David von Schack (D)

Pfizer Research and Development, Pfizer Inc., Cambridge, MA, USA.

Soumya Raychaudhuri (S)

Center for Data Sciences, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.
Divisions of Genetics and Rheumatology, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.
Program in Medical and Population Genetics, Broad Institute of MIT and Harvard, Cambridge, MA, USA.
Department of Biomedical Informatics, Harvard Medical School, Boston, MA, USA.

Daniel Ziemek (D)

Pfizer Research and Development, Pfizer Inc., Cambridge, MA, USA.

Xinli Hu (X)

Pfizer Research and Development, Pfizer Inc., Cambridge, MA, USA. xinli.hu@pfizer.com.

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