Myocardial strain is regulated by cardiac preload in the early stage of sepsis.


Journal

BMC cardiovascular disorders
ISSN: 1471-2261
Titre abrégé: BMC Cardiovasc Disord
Pays: England
ID NLM: 100968539

Informations de publication

Date de publication:
14 Aug 2024
Historique:
received: 18 07 2023
accepted: 30 07 2024
medline: 15 8 2024
pubmed: 15 8 2024
entrez: 14 8 2024
Statut: epublish

Résumé

Owing to a lack of data, this study aimed to explore the effect of cardiac preload on myocardial strain in patients with sepsis. A total of 70 patients with sepsis in intensive care unit (ICU) of a tertiary teaching hospital in China from January 2018 to July 2019 and underwent transthoracic echocardiography were enrolled. Echocardiographic data were recorded at ICU admission and 24 h later. Patients were assigned to low left ventricular end-diastolic volume index (LVEDVI) and normal LVEDVI groups. We assessed the impact of preload on myocardial strain between the groups and analyzed the correlation of echocardiographic parameters under different preload conditions. Thirty-seven patients (53%) had a low LVEDVI and 33 (47%) a normal LVEDVI. Those in the low LVEDVI group had a faster heart rate (121.7 vs. 95.3, p < 0.001) and required a greater degree of fluid infusion (3.67 L vs. 2.62 L, P = 0.019). The left ventricular global strain (LVGLS) (-8.60% vs. -10.80%, p = 0.001), left ventricular global circumferential strain (LVGCS) (-13.83% vs. -18.26%, p = 0.006), and right ventricular global longitudinal strain (RVGLS) (-6.9% vs. -10.60%, p = 0.001) showed significant improvements in the low LVEDVI group after fluid resuscitation. However, fluid resuscitation resulted in a significantly increased cardiac afterload value (1172.00 vs. 1487.00, p = 0.009) only in the normal LVEDVI group. Multivariate backward linear regression showed that LVEDVI changes were independently associated with myocardial strain-related improvements during fluid resuscitation. The baseline LVEDVI was significantly negatively correlated with the LVGLS and RVGLS (r = -0.44 and - 0.39, respectively) but not LVGCS. LVEDVI increases during fluid resuscitation were associated with improvements in the myocardial strain degree. Myocardial strain alterations were significantly influenced by the cardiac preload during fluid resuscitation in sepsis.

Sections du résumé

BACKGROUND BACKGROUND
Owing to a lack of data, this study aimed to explore the effect of cardiac preload on myocardial strain in patients with sepsis.
METHODS METHODS
A total of 70 patients with sepsis in intensive care unit (ICU) of a tertiary teaching hospital in China from January 2018 to July 2019 and underwent transthoracic echocardiography were enrolled. Echocardiographic data were recorded at ICU admission and 24 h later. Patients were assigned to low left ventricular end-diastolic volume index (LVEDVI) and normal LVEDVI groups. We assessed the impact of preload on myocardial strain between the groups and analyzed the correlation of echocardiographic parameters under different preload conditions.
RESULTS RESULTS
Thirty-seven patients (53%) had a low LVEDVI and 33 (47%) a normal LVEDVI. Those in the low LVEDVI group had a faster heart rate (121.7 vs. 95.3, p < 0.001) and required a greater degree of fluid infusion (3.67 L vs. 2.62 L, P = 0.019). The left ventricular global strain (LVGLS) (-8.60% vs. -10.80%, p = 0.001), left ventricular global circumferential strain (LVGCS) (-13.83% vs. -18.26%, p = 0.006), and right ventricular global longitudinal strain (RVGLS) (-6.9% vs. -10.60%, p = 0.001) showed significant improvements in the low LVEDVI group after fluid resuscitation. However, fluid resuscitation resulted in a significantly increased cardiac afterload value (1172.00 vs. 1487.00, p = 0.009) only in the normal LVEDVI group. Multivariate backward linear regression showed that LVEDVI changes were independently associated with myocardial strain-related improvements during fluid resuscitation. The baseline LVEDVI was significantly negatively correlated with the LVGLS and RVGLS (r = -0.44 and - 0.39, respectively) but not LVGCS. LVEDVI increases during fluid resuscitation were associated with improvements in the myocardial strain degree.
CONCLUSIONS CONCLUSIONS
Myocardial strain alterations were significantly influenced by the cardiac preload during fluid resuscitation in sepsis.

Identifiants

pubmed: 39143461
doi: 10.1186/s12872-024-04083-8
pii: 10.1186/s12872-024-04083-8
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

426

Informations de copyright

© 2024. The Author(s).

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Auteurs

Xiaolong Huang (X)

Department of Intensive Care Unit, School of Medicine, The First Affiliated Hospital of Xiamen University, Xiamen University, Xiamen, China.
The Third Clinical Medical College, Fujian Medical University, Fuzhou, China.

Guiyang Lu (G)

Department of Intensive Care Unit, School of Medicine, The First Affiliated Hospital of Xiamen University, Xiamen University, Xiamen, China.
The Third Clinical Medical College, Fujian Medical University, Fuzhou, China.

Xiaoyang Cai (X)

Department of Intensive Care Unit, School of Medicine, The First Affiliated Hospital of Xiamen University, Xiamen University, Xiamen, China.
The Third Clinical Medical College, Fujian Medical University, Fuzhou, China.

Yingchang Xue (Y)

Department of Intensive Care Unit, People's Hospital Affiliated to Shandong First Medical University, Jinan, Shandong, China.

Xinxin Wang (X)

Department of Intensive Care Unit, School of Medicine, The First Affiliated Hospital of Xiamen University, Xiamen University, Xiamen, China.
The Third Clinical Medical College, Fujian Medical University, Fuzhou, China.

Yuanyuan Jiang (Y)

Department of Ultrasound, School of Medicine, The First Affiliated Hospital of Xiamen University, Xiamen University, Xiamen, China.

Yaogui Ning (Y)

Department of Intensive Care Unit, School of Medicine, The First Affiliated Hospital of Xiamen University, Xiamen University, Xiamen, China. nyg@xmu.edu.cn.
The Third Clinical Medical College, Fujian Medical University, Fuzhou, China. nyg@xmu.edu.cn.

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