MCM2-7 loading-dependent ORC release ensures genome-wide origin licensing.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
24 Aug 2024
Historique:
received: 01 02 2024
accepted: 09 08 2024
medline: 26 8 2024
pubmed: 26 8 2024
entrez: 24 8 2024
Statut: epublish

Résumé

Origin recognition complex (ORC)-dependent loading of the replicative helicase MCM2-7 onto replication origins in G1-phase forms the basis of replication fork establishment in S-phase. However, how ORC and MCM2-7 facilitate genome-wide DNA licensing is not fully understood. Mapping the molecular footprints of budding yeast ORC and MCM2-7 genome-wide, we discovered that MCM2-7 loading is associated with ORC release from origins and redistribution to non-origin sites. Our bioinformatic analysis revealed that origins are compact units, where a single MCM2-7 double hexamer blocks repetitive loading through steric ORC binding site occlusion. Analyses of A-elements and an improved B2-element consensus motif uncovered that DNA shape, DNA flexibility, and the correct, face-to-face spacing of the two DNA elements are hallmarks of ORC-binding and efficient helicase loading sites. Thus, our work identified fundamental principles for MCM2-7 helicase loading that explain how origin licensing is realised across the genome.

Identifiants

pubmed: 39181881
doi: 10.1038/s41467-024-51538-9
pii: 10.1038/s41467-024-51538-9
doi:

Substances chimiques

Origin Recognition Complex 0
Saccharomyces cerevisiae Proteins 0
Minichromosome Maintenance Proteins EC 3.6.4.12
DNA, Fungal 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

7306

Subventions

Organisme : RCUK | Biotechnology and Biological Sciences Research Council (BBSRC)
ID : BB/N000323/1
Organisme : RCUK | Biotechnology and Biological Sciences Research Council (BBSRC)
ID : BB/S001387/1
Organisme : RCUK | MRC | Medical Research Foundation
ID : MC_U120085811
Organisme : Wellcome Trust (Wellcome)
ID : 107903/Z/15/Z
Organisme : Wellcome Trust (Wellcome)
ID : 107903/Z/15/Z
Organisme : Wellcome Trust (Wellcome)
ID : 107903/Z/15/Z
Organisme : Deutsche Forschungsgemeinschaft (German Research Foundation)
ID : 336963304
Organisme : Deutsche Forschungsgemeinschaft (German Research Foundation)
ID : 505087959
Organisme : Deutsche Forschungsgemeinschaft (German Research Foundation)
ID : RE 4094/2-1
Organisme : EC | Horizon 2020 Framework Programme (EU Framework Programme for Research and Innovation H2020)
ID : 101109916

Informations de copyright

© 2024. The Author(s).

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Auteurs

L Maximilian Reuter (LM)

DNA Replication Group, Institute of Clinical Sciences, Faculty of Medicine, Imperial College London, London, United Kingdom. m.reuter@imb-mainz.de.
Institute of Molecular Biology (IMB) gGmbH, Ackermannweg 4, Mainz, Germany. m.reuter@imb-mainz.de.

Sanjay P Khadayate (SP)

MRC London Institute of Medical Sciences (LMS), London, United Kingdom.

Audrey Mossler (A)

DNA Replication Group, Institute of Clinical Sciences, Faculty of Medicine, Imperial College London, London, United Kingdom.

Korbinian Liebl (K)

Department of Chemistry, Chicago Center for Theoretical Chemistry, Institute for Biophysical Dynamics, and James Franck Institute, The University of Chicago, Chicago, IL, USA.

Sarah V Faull (SV)

DNA Replication Group, Institute of Clinical Sciences, Faculty of Medicine, Imperial College London, London, United Kingdom.

Mohammad M Karimi (MM)

MRC London Institute of Medical Sciences (LMS), London, United Kingdom.
Comprehensive Cancer Centre, School of Cancer & Pharmaceutical Sciences, Faculty of Life Sciences & Medicine, King's College London, London, United Kingdom.

Christian Speck (C)

DNA Replication Group, Institute of Clinical Sciences, Faculty of Medicine, Imperial College London, London, United Kingdom. chris.speck@imperial.ac.uk.
MRC London Institute of Medical Sciences (LMS), London, United Kingdom. chris.speck@imperial.ac.uk.

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