Serum markers of microbial translocation and intestinal damage in assessment of gastrointestinal tract involvement in systemic sclerosis.


Journal

Clinical and experimental medicine
ISSN: 1591-9528
Titre abrégé: Clin Exp Med
Pays: Italy
ID NLM: 100973405

Informations de publication

Date de publication:
19 Sep 2024
Historique:
received: 04 07 2024
accepted: 14 08 2024
medline: 19 9 2024
pubmed: 19 9 2024
entrez: 18 9 2024
Statut: epublish

Résumé

Gastrointestinal (GI) tract involvement affects up to 90% of Systemic sclerosis (SSc) patients. The presence of GI symptoms is assessed by the University of California, Los Angeles, and Scleroderma Clinical Trials Consortium Gastrointestinal Scale (UCLA SCTC GIT 2.0). Microbial translocation (MT) is reported in SSc patients consequently to increased intestinal permeability due to intestinal damage (ID) and dysbiosis. Aim of this study was to assess circulating levels of LBP and EndoCab IgM (markers of MT), IL-6 (marker of inflammation), I-FABP and Zonulin (markers of ID) in a cohort of SSc patients and healthy controls (HC). Moreover, we aimed to correlate these parameters with severity of GI symptoms. UCLA SCTC GIT 2.0 questionnaire was administered to 60 consecutive SSc patients. Markers of MT, inflammation and ID were evaluated in SSc patients and HC. SSc patients had higher median value of markers of MT, inflammation and ID than HC. The logistic regression analysis showed LBP as the only variable associated with an UCLA total score "moderate-to-very severe" [OR 1.001 (CI 95%: 1.001-1.002), p < 0.001]. The logistic regression analysis showed LBP [OR 1.002 (CI 95%: 1.001-1.003), p < 0.01] and disease duration [OR 1.242 (CI 95%: 1.023-1.506), p < 0.05] as variables associated with UCLA distension/bloating "moderate-to-very severe". The logistic regression analysis showed LBP as the only variable associated with UCLA diarrhea "moderate-to-very severe" [OR 1.002 (CI 95%: 1.001-1.003), p < 0.01]. SSc patients with dysregulation gut mucosal integrity expressed by high levels of MT and ID biomarkers had more severe GI symptoms.

Identifiants

pubmed: 39294494
doi: 10.1007/s10238-024-01466-1
pii: 10.1007/s10238-024-01466-1
doi:

Substances chimiques

Biomarkers 0
zonulin 0
Haptoglobins 0
Cholera Toxin 9012-63-9
Interleukin-6 0
Protein Precursors 0
Fatty Acid-Binding Proteins 0
lipopolysaccharide-binding protein 0
Carrier Proteins 0
Acute-Phase Proteins 0
Immunoglobulin M 0
FABP2 protein, human 0
Membrane Glycoproteins 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

225

Informations de copyright

© 2024. The Author(s).

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Auteurs

Chiara Pellicano (C)

Department of Translational and Precision Medicine, Sapienza University of Rome, Viale dell'Università 37, 00185, Rome, Italy.

Alessandra Oliva (A)

Department of Public Health and Infectious Diseases, Sapienza University of Rome, 00185, Rome, Italy.

Amalia Colalillo (A)

Department of Translational and Precision Medicine, Sapienza University of Rome, Viale dell'Università 37, 00185, Rome, Italy.

Antonietta Gigante (A)

Department of Translational and Precision Medicine, Sapienza University of Rome, Viale dell'Università 37, 00185, Rome, Italy.

Elisa D'Aliesio (E)

Department of Public Health and Infectious Diseases, Sapienza University of Rome, 00185, Rome, Italy.

Dania Al Ismail (D)

Department of Public Health and Infectious Diseases, Sapienza University of Rome, 00185, Rome, Italy.

Maria Claudia Miele (MC)

Department of Public Health and Infectious Diseases, Sapienza University of Rome, 00185, Rome, Italy.

Rosario Cianci (R)

Department of Translational and Precision Medicine, Sapienza University of Rome, Viale dell'Università 37, 00185, Rome, Italy.

Claudio Maria Mastroianni (CM)

Department of Public Health and Infectious Diseases, Sapienza University of Rome, 00185, Rome, Italy.

Edoardo Rosato (E)

Department of Translational and Precision Medicine, Sapienza University of Rome, Viale dell'Università 37, 00185, Rome, Italy. edoardo.rosato@uniroma1.it.

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