Experimental infection of ringtail possums (Pseudocheirus peregrinus) with Mycobacterium ulcerans, the agent of Buruli ulcer.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
25 Oct 2024
Historique:
received: 03 06 2024
accepted: 17 10 2024
medline: 26 10 2024
pubmed: 26 10 2024
entrez: 25 10 2024
Statut: epublish

Résumé

Buruli ulcer (BU) is a necrotizing disease of skin and soft tissue caused by the bacterium Mycobacterium ulcerans (MU). In Australia, where the disease is emerging in new geographic areas and human case numbers are increasing, native possum species act as reservoir hosts. To better understand the life history of MU in one of its natural hosts, we conducted intra-dermal challenge of six wild caught, MU-naïve common ringtail possums (Pseudocheirus peregrinus). All six animals developed BU disease consistent with that observed in naturally infected ringtail possums. Time to ulceration varied between 49 and 77 days (mean = 61.8 days). Molecular evidence of systemic infection was detected in five animals and was supported by consistent histopathological findings in four animals. Pathological findings included random, multifocal, granulomatous hepatitis in four possums, one of which also had a mild, multifocal, interstitial granulomatous pneumonia. Acid-fast bacilli were only evident in inflammatory foci beyond the primary inoculation site in one possum. The ringtail possum model of MU infection is an important tool for the investigation of bacterial transmission dynamics, pathogenesis and immune response in a natural host. Data from this model may improve disease risk modelling and help identify intervention points to stop zoonotic transmission and disease spread.

Identifiants

pubmed: 39455716
doi: 10.1038/s41598-024-76857-1
pii: 10.1038/s41598-024-76857-1
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

25352

Informations de copyright

© 2024. Crown.

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Auteurs

Kim R Blasdell (KR)

Health and Biosecurity, Commonwealth Scientific and Industrial Research Organisation, Geelong, Australia. kim.blasdell@csiro.au.

Richard J Ploeg (RJ)

Australian Animal Health Laboratory, Commonwealth Scientific and Industrial Research Organisation, Geelong, Australia.

Emma C Hobbs (EC)

Department of Veterinary Biosciences, University of Melbourne, Melbourne, Australia.

Stephen Muhi (S)

Peter Doherty Institute for Infection and Immunity, University of Melbourne, Melbourne, Australia.

Sarah J Riddell (SJ)

Australian Animal Health Laboratory, Commonwealth Scientific and Industrial Research Organisation, Geelong, Australia.

Alexandra Cunneen (A)

Australian Animal Health Laboratory, Commonwealth Scientific and Industrial Research Organisation, Geelong, Australia.

Michael L Kelly (ML)

Australian Animal Health Laboratory, Commonwealth Scientific and Industrial Research Organisation, Geelong, Australia.

Kate Maynard (K)

Australian Animal Health Laboratory, Commonwealth Scientific and Industrial Research Organisation, Geelong, Australia.

Tess R Malcolm (TR)

Health and Biosecurity, Commonwealth Scientific and Industrial Research Organisation, Geelong, Australia.

Md Tanjir Islam (MT)

Health and Biosecurity, Commonwealth Scientific and Industrial Research Organisation, Geelong, Australia.

Victoria Boyd (V)

Health and Biosecurity, Commonwealth Scientific and Industrial Research Organisation, Geelong, Australia.

Timothy P Stinear (TP)

Peter Doherty Institute for Infection and Immunity, University of Melbourne, Melbourne, Australia.

Sacha J Pidot (SJ)

Peter Doherty Institute for Infection and Immunity, University of Melbourne, Melbourne, Australia.

Eugene Athan (E)

Department of Infectious Diseases, Barwon Health, Geelong, VIC, Australia.

Daniel P O'Brien (DP)

Department of Infectious Diseases, Barwon Health, Geelong, VIC, Australia.

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