Combining noninvasive risk stratification parameters improves the prediction of mortality and appropriate ICD shocks.


Journal

Annals of noninvasive electrocardiology : the official journal of the International Society for Holter and Noninvasive Electrocardiology, Inc
ISSN: 1542-474X
Titre abrégé: Ann Noninvasive Electrocardiol
Pays: United States
ID NLM: 9607443

Informations de publication

Date de publication:
01 2019
Historique:
received: 08 06 2018
revised: 06 08 2018
accepted: 10 08 2018
pubmed: 29 9 2018
medline: 10 4 2020
entrez: 29 9 2018
Statut: ppublish

Résumé

Sudden cardiac death (SCD) results from a complex interplay of abnormalities in autonomic function, myocardial substrate and vulnerability. We studied whether a combination of noninvasive risk stratification tests reflecting these key players could improve risk stratification. Patients implanted with an ICD in whom 24-hr holter recordings were available prior to implant were included. QRS fragmentation (fQRS) was selected as measure of myocardial substrate and a high ventricular premature beat count (VPB >10/hr) for arrhythmic vulnerability. From receiver operating characteristics analysis, detrended fluctuation analysis (DFA), turbulence slope, and deceleration capacity were selected for autonomic function. Adjusted Cox regression analysis with comparison of C-statistics was performed to predict first appropriate shock (AS) and total mortality. A total of 220 patients were included in the analysis with an overall follow-up of 4.3 ± 3.1 years. A model including VPB >10/hr, inferior fQRS, and abnormal nonedited DFA was the best for prediction of AS after 1 year of follow-up with a trends toward improvement of the C-statistics compared to baseline (p = 0.055). The risk increased significantly with every abnormal test (HR 1.793, 95%CI 1.255-2.564). A model including fQRS in any region and abnormal edited DFA was the best for prediction of mortality after 3 years of follow-up with significant improvement of the C-statistics (p = 0.023). Each abnormal test was associated with a significant increase in mortality (HR 5.069, 95%CI 1.978-12.994). Combining noninvasive risk stratification tests according to their physiological background can improve the risk prediction of SCD and mortality.

Sections du résumé

BACKGROUND
Sudden cardiac death (SCD) results from a complex interplay of abnormalities in autonomic function, myocardial substrate and vulnerability. We studied whether a combination of noninvasive risk stratification tests reflecting these key players could improve risk stratification.
METHODS
Patients implanted with an ICD in whom 24-hr holter recordings were available prior to implant were included. QRS fragmentation (fQRS) was selected as measure of myocardial substrate and a high ventricular premature beat count (VPB >10/hr) for arrhythmic vulnerability. From receiver operating characteristics analysis, detrended fluctuation analysis (DFA), turbulence slope, and deceleration capacity were selected for autonomic function. Adjusted Cox regression analysis with comparison of C-statistics was performed to predict first appropriate shock (AS) and total mortality.
RESULTS
A total of 220 patients were included in the analysis with an overall follow-up of 4.3 ± 3.1 years. A model including VPB >10/hr, inferior fQRS, and abnormal nonedited DFA was the best for prediction of AS after 1 year of follow-up with a trends toward improvement of the C-statistics compared to baseline (p = 0.055). The risk increased significantly with every abnormal test (HR 1.793, 95%CI 1.255-2.564). A model including fQRS in any region and abnormal edited DFA was the best for prediction of mortality after 3 years of follow-up with significant improvement of the C-statistics (p = 0.023). Each abnormal test was associated with a significant increase in mortality (HR 5.069, 95%CI 1.978-12.994).
CONCLUSION
Combining noninvasive risk stratification tests according to their physiological background can improve the risk prediction of SCD and mortality.

Identifiants

pubmed: 30265438
doi: 10.1111/anec.12604
pmc: PMC6931642
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e12604

Subventions

Organisme : European Community's Seventh Framework Program FP7: EU-CERT-ICD
ID : HEALTH-F2-2013-602299
Pays : International

Informations de copyright

© 2018 Wiley Periodicals, Inc.

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Auteurs

Bert Vandenberk (B)

Department of Cardiovascular Sciences, University of Leuven, Leuven, Belgium.
Department of Cardiology, University Hospitals Leuven, Leuven, Belgium.

M Juhani Junttila (MJ)

Research Unit of Internal Medicine, Medical Research Center, University Hospital and University of Oulu, Oulu, Finland.

Tomas Robyns (T)

Department of Cardiovascular Sciences, University of Leuven, Leuven, Belgium.
Department of Cardiology, University Hospitals Leuven, Leuven, Belgium.

Christophe Garweg (C)

Department of Cardiovascular Sciences, University of Leuven, Leuven, Belgium.
Department of Cardiology, University Hospitals Leuven, Leuven, Belgium.

Joris Ector (J)

Department of Cardiovascular Sciences, University of Leuven, Leuven, Belgium.
Department of Cardiology, University Hospitals Leuven, Leuven, Belgium.

Heikki V Huikuri (HV)

Research Unit of Internal Medicine, Medical Research Center, University Hospital and University of Oulu, Oulu, Finland.

Rik Willems (R)

Department of Cardiovascular Sciences, University of Leuven, Leuven, Belgium.
Department of Cardiology, University Hospitals Leuven, Leuven, Belgium.

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