A Novel BaEVRless-Pseudotyped γ-Globin Lentiviral Vector Drives High and Stable Fetal Hemoglobin Expression and Improves Thalassemic Erythropoiesis


Journal

Human gene therapy
ISSN: 1557-7422
Titre abrégé: Hum Gene Ther
Pays: United States
ID NLM: 9008950

Informations de publication

Date de publication:
05 2019
Historique:
pubmed: 17 10 2018
medline: 13 3 2020
entrez: 17 10 2018
Statut: ppublish

Résumé

It has previously been demonstrated that the self-inactivating γ-globin lentiviral vector GGHI can significantly increase fetal hemoglobin (HbF) in erythroid cells from thalassemia patients and thus improve the disease phenotype

Identifiants

pubmed: 30324804
doi: 10.1089/hum.2018.022
doi:

Substances chimiques

gamma-Globins 0
Fetal Hemoglobin 9034-63-3

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

601-617

Auteurs

Ekati Drakopoulou (E)

1 Laboratory of Cell and Gene Therapy, Centre for Basic Research, Biomedical Research Foundation of the Academy of Athens (BRFAA), Athens, Greece.
2 Laboratory of Biology, University of Athens School of Medicine, Athens, Greece.

Maria Georgomanoli (M)

1 Laboratory of Cell and Gene Therapy, Centre for Basic Research, Biomedical Research Foundation of the Academy of Athens (BRFAA), Athens, Greece.
2 Laboratory of Biology, University of Athens School of Medicine, Athens, Greece.

Carsten W Lederer (CW)

3 Department of Molecular Genetics Thalassemia, The Cyprus Institute of Neurology and Genetics, Nicosia, Cyprus.
4 Cyprus School of Molecular Medicine, Nicosia, Cyprus.

Marina Kleanthous (M)

3 Department of Molecular Genetics Thalassemia, The Cyprus Institute of Neurology and Genetics, Nicosia, Cyprus.
4 Cyprus School of Molecular Medicine, Nicosia, Cyprus.

Caroline Costa (C)

5 CIRI-International Center for Infectiology Research, Inserm, U1111, Université Claude Bernard Lyon 1, CNRS, UMR 5308, Ecole Normale Supérieure de Lyon, Lyon, France.

Ornellie Bernadin (O)

5 CIRI-International Center for Infectiology Research, Inserm, U1111, Université Claude Bernard Lyon 1, CNRS, UMR 5308, Ecole Normale Supérieure de Lyon, Lyon, France.

François-Loïc Cosset (FL)

5 CIRI-International Center for Infectiology Research, Inserm, U1111, Université Claude Bernard Lyon 1, CNRS, UMR 5308, Ecole Normale Supérieure de Lyon, Lyon, France.

Ersi Voskaridou (E)

6 Thalassemia and Sickle Cell Disease Centre, Laikon General Hospital, Athens, Greece.

Els Verhoeyen (E)

5 CIRI-International Center for Infectiology Research, Inserm, U1111, Université Claude Bernard Lyon 1, CNRS, UMR 5308, Ecole Normale Supérieure de Lyon, Lyon, France.
7 Inserm, U1065, Centre Méditerranéen de Médecine Moléculaire (C3M), Equipe Contrôle Métabolique des Morts Cellulaires, Nice, France.

Eleni Papanikolaou (E)

1 Laboratory of Cell and Gene Therapy, Centre for Basic Research, Biomedical Research Foundation of the Academy of Athens (BRFAA), Athens, Greece.
2 Laboratory of Biology, University of Athens School of Medicine, Athens, Greece.

Nicholas P Anagnou (NP)

1 Laboratory of Cell and Gene Therapy, Centre for Basic Research, Biomedical Research Foundation of the Academy of Athens (BRFAA), Athens, Greece.
2 Laboratory of Biology, University of Athens School of Medicine, Athens, Greece.

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Classifications MeSH