Long-term results of desensitization protocol with and without rituximab in sensitized kidney transplant recipients.


Journal

Clinical transplantation
ISSN: 1399-0012
Titre abrégé: Clin Transplant
Pays: Denmark
ID NLM: 8710240

Informations de publication

Date de publication:
06 2019
Historique:
received: 05 08 2018
revised: 27 12 2018
accepted: 14 03 2019
pubmed: 4 4 2019
medline: 5 8 2020
entrez: 4 4 2019
Statut: ppublish

Résumé

Desensitization protocols have been developed in order to overcome the immunological barrier of donor-specific anti-HLA antibodies (DSA). During 2006-2012, we implemented a program for desensitizing sensitized (positive DSA, negative NIH-CDC crossmatch) living-donor recipients. The long-term outcome of 36 sensitized recipients, treated with IVIG and plasmapheresis (PP), with or without rituximab (added when > 7500 MFI), was compared to 252 non-sensitized living-donor recipients. Median peak DSA level before desensitization was 7223 (range 3567-16 000) MFI. During a mean follow-up of 121.9 months, graft loss occurred in 6/36 (17%) of the sensitized and 15/251 (6%) of the non-sensitized recipients (P = 0.021). Five-year and 10-year death-censored graft survival rates were 85% and 81% compared to 95% and 92%, respectively, for the non-sensitized recipients. There was no difference in recipients' survival. Slightly more episodes of acute rejection occurred in the sensitized group but had not influence on graft survival. At the last follow-up, 28 recipients had functioning graft; seventeen (47%) did not have detectable DSA. Eleven recipients had excellent graft function despite having detectable DSA. The long-term outcomes of sensitized recipients who underwent desensitization are encouraging. Adding rituximab to PP + IVIG in candidates with very high titers may result in improved outcome.

Sections du résumé

BACKGROUND
Desensitization protocols have been developed in order to overcome the immunological barrier of donor-specific anti-HLA antibodies (DSA).
METHODS
During 2006-2012, we implemented a program for desensitizing sensitized (positive DSA, negative NIH-CDC crossmatch) living-donor recipients. The long-term outcome of 36 sensitized recipients, treated with IVIG and plasmapheresis (PP), with or without rituximab (added when > 7500 MFI), was compared to 252 non-sensitized living-donor recipients.
RESULTS
Median peak DSA level before desensitization was 7223 (range 3567-16 000) MFI. During a mean follow-up of 121.9 months, graft loss occurred in 6/36 (17%) of the sensitized and 15/251 (6%) of the non-sensitized recipients (P = 0.021). Five-year and 10-year death-censored graft survival rates were 85% and 81% compared to 95% and 92%, respectively, for the non-sensitized recipients. There was no difference in recipients' survival. Slightly more episodes of acute rejection occurred in the sensitized group but had not influence on graft survival. At the last follow-up, 28 recipients had functioning graft; seventeen (47%) did not have detectable DSA. Eleven recipients had excellent graft function despite having detectable DSA.
CONCLUSION
The long-term outcomes of sensitized recipients who underwent desensitization are encouraging. Adding rituximab to PP + IVIG in candidates with very high titers may result in improved outcome.

Identifiants

pubmed: 30941818
doi: 10.1111/ctr.13562
doi:

Substances chimiques

HLA Antigens 0
Immunologic Factors 0
Isoantibodies 0
Rituximab 4F4X42SYQ6

Types de publication

Comparative Study Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e13562

Informations de copyright

© 2019 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd.

Auteurs

Hefziba Green (H)

Department of Medicine B, Rabin Medical Center, Petah-Tikva, Israel.
Nephrology and Hypertension, Rabin Medical Center, Petah-Tikva, Israel.
Sackler School of Medicine, Tel-Aviv University, Tel-aviv, Israel.

Eviatar Nesher (E)

Department of Transplantation, Rabin Medical Center, Petah-Tikva, Israel.

Sigal Aizner (S)

Department of Transplantation, Rabin Medical Center, Petah-Tikva, Israel.

Moshe Israeli (M)

Sackler School of Medicine, Tel-Aviv University, Tel-aviv, Israel.
Tissue Typing Laboratory, Rabin Medical Center, Petah-Tikva, Israel.

Tirza Klein (T)

Sackler School of Medicine, Tel-Aviv University, Tel-aviv, Israel.
Tissue Typing Laboratory, Rabin Medical Center, Petah-Tikva, Israel.

Hana Zakai (H)

Sackler School of Medicine, Tel-Aviv University, Tel-aviv, Israel.
Tissue Typing Laboratory, Rabin Medical Center, Petah-Tikva, Israel.

Ruth Rahamimov (R)

Nephrology and Hypertension, Rabin Medical Center, Petah-Tikva, Israel.
Sackler School of Medicine, Tel-Aviv University, Tel-aviv, Israel.

Benaya Rozen-Zvi (B)

Nephrology and Hypertension, Rabin Medical Center, Petah-Tikva, Israel.
Sackler School of Medicine, Tel-Aviv University, Tel-aviv, Israel.

Eytan Mor (E)

Sackler School of Medicine, Tel-Aviv University, Tel-aviv, Israel.
Transplant Center, Department of Surgery B, Sheba Medical Center, Ramat-Gan, Israel.

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