Beneficial Effects of the Angiotensin-Converting Enzyme 2 Activator Dize in Renovascular Hypertension.


Journal

Protein and peptide letters
ISSN: 1875-5305
Titre abrégé: Protein Pept Lett
Pays: Netherlands
ID NLM: 9441434

Informations de publication

Date de publication:
2019
Historique:
received: 17 01 2019
revised: 22 03 2019
accepted: 23 03 2019
pubmed: 6 4 2019
medline: 8 10 2019
entrez: 6 4 2019
Statut: ppublish

Résumé

Angiotensin Converting Enzyme (ACE) 2 is an important modulator of the Renin Angiotensin System (RAS) and the RAS plays a central role in renovascular hypertension. Very few studies investigated the role of components of the counterregulatory RAS axis (ACE2, Ang-(1-7) and Mas receptor) in renovascular hypertension and the results are controversial. The aim of this study was to investigate the effects of Diminazene Aceturate (DIZE) administration on renal function and renal inflammation parameters in 2K1C hypertensive rats. Male Wistar rats were divided into three experimental groups: sham-operated animals, 2K1C+saline and 2K1C+DIZE orally (1 mg/kg/day). At the end of the 30 days of treatment, renal function was analyzed and kidneys from all the groups were collected and processed separately for measurement of N-acetyl-beta-D-glucosaminidase (NAG) and Myeloperoxidase (MPO) activities, cytokines, chemokines and nitric oxide levels. Oral DIZE administration for 4 weeks in hypertensive rats attenuated renal dysfunction and reduced the levels of MPO and NAG, cytokines and chemokines (IL1β, IL-6, TNF-α and MCP-1) and increased urinary nitrate/nitrite levels in 2K1C hypertensive rats. Our findings showed that ACE2 activation may effectively improve renal alterations and inflammation induced by renovascular hypertension.

Sections du résumé

BACKGROUND BACKGROUND
Angiotensin Converting Enzyme (ACE) 2 is an important modulator of the Renin Angiotensin System (RAS) and the RAS plays a central role in renovascular hypertension. Very few studies investigated the role of components of the counterregulatory RAS axis (ACE2, Ang-(1-7) and Mas receptor) in renovascular hypertension and the results are controversial.
OBJECTIVE OBJECTIVE
The aim of this study was to investigate the effects of Diminazene Aceturate (DIZE) administration on renal function and renal inflammation parameters in 2K1C hypertensive rats.
METHODS METHODS
Male Wistar rats were divided into three experimental groups: sham-operated animals, 2K1C+saline and 2K1C+DIZE orally (1 mg/kg/day). At the end of the 30 days of treatment, renal function was analyzed and kidneys from all the groups were collected and processed separately for measurement of N-acetyl-beta-D-glucosaminidase (NAG) and Myeloperoxidase (MPO) activities, cytokines, chemokines and nitric oxide levels.
RESULTS RESULTS
Oral DIZE administration for 4 weeks in hypertensive rats attenuated renal dysfunction and reduced the levels of MPO and NAG, cytokines and chemokines (IL1β, IL-6, TNF-α and MCP-1) and increased urinary nitrate/nitrite levels in 2K1C hypertensive rats.
CONCLUSION CONCLUSIONS
Our findings showed that ACE2 activation may effectively improve renal alterations and inflammation induced by renovascular hypertension.

Identifiants

pubmed: 30950337
pii: PPL-EPUB-97826
doi: 10.2174/0929866526666190405123422
doi:

Substances chimiques

Cytokines 0
Enzyme Activators 0
Peptide Fragments 0
Nitric Oxide 31C4KY9ESH
Angiotensin I 9041-90-1
Peroxidase EC 1.11.1.7
Acetylglucosaminidase EC 3.2.1.52
Peptidyl-Dipeptidase A EC 3.4.15.1
Ace2 protein, rat EC 3.4.17.23
Angiotensin-Converting Enzyme 2 EC 3.4.17.23
angiotensin I (1-7) IJ3FUK8MOF
diminazene aceturate JI8SAD85NO
Diminazene Y5G36EEA5Z

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

523-531

Informations de copyright

Copyright© Bentham Science Publishers; For any queries, please email at epub@benthamscience.net.

Auteurs

Lucas Miranda Kangussu (LM)

Department of Morphology, Institute of Biological Sciences, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil.

Tatiane Cristine S de Almeida (TCS)

Department of Morphology, Institute of Biological Sciences, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil.

Thiago Ruiz R Prestes (TRR)

Department of Pediatrics, School of Medicine, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil.

Marilda Luz de Andrade De Maria (ML)

Department of Morphology, Institute of Biological Sciences, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil.

Roberta da Silva Filha (R)

Department of Pediatrics, School of Medicine, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil.

Maria Aparecida Ribeiro Vieira (MAR)

Department of Physiology and Biophysics, Institute of Biological Sciences, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil.

Ana Cristina Simões E Silva (ACSE)

Department of Pediatrics, School of Medicine, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil.

Anderson José Ferreira (AJ)

Department of Morphology, Institute of Biological Sciences, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil.

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Classifications MeSH