Different inflammatory profile in young and elderly STEMI patients undergoing primary percutaneous coronary intervention (PPCI): Its influence on no-reflow and mortality.


Journal

International journal of cardiology
ISSN: 1874-1754
Titre abrégé: Int J Cardiol
Pays: Netherlands
ID NLM: 8200291

Informations de publication

Date de publication:
01 09 2019
Historique:
received: 07 03 2019
revised: 12 04 2019
accepted: 02 05 2019
pubmed: 14 5 2019
medline: 8 5 2020
entrez: 14 5 2019
Statut: ppublish

Résumé

Coronary no-reflow phenomenon in ST-segment elevation myocardial infarction (STEMI) is associated with a poor clinical prognosis. Although its pathophysiology is not fully elucidated, a deregulated systemic inflammatory response plays an important role. Specifically, the relationship between age-associated differences in inflammatory markers and either no-reflow or mortality in STEMI patients undergoing primary percutaneous coronary intervention (pPCI) has never been investigated. We retrospectively enrolled 625 consecutive STEMI patients undergoing pPCI for whom a complete laboratory inflammatory pattern was available. Routinely blood measured laboratory parameters were collected at the moment of admission. No reflow was defined as Thrombolysis in Myocardial Infarction (TIMI) flow-grade lower than 3. The population was divided into two groups using a cut-off centered at 65 years. Compared to younger patients, elderly patients had higher mean values of fibrinogen, brain natriuretic peptide (BNP), leukocytes, neutrophil-to-lymphocyte ratio (NLR), C reactive protein/albumin ratio (CAR). Conversely, lymphocyte count and albumin levels were higher in young patients. In elderly patients, the values of NLR, CAR as well as leukocytes, fibrinogen and neutrophils were associated with no-reflow, while in young patients only BNP value was associated. At multivariate Cox regression analysis, only BNP and NLR resulted as independent predictors of all-cause mortality in the whole population and in elderly patients. Elderly STEMI patients on admission had a higher acute pro-inflammatory profile than young patients, associated to coronary no-reflow and mortality outcome. These results suggest that a different therapeutic approach between elderly and young STEMI patients should be agreed.

Sections du résumé

BACKGROUND
Coronary no-reflow phenomenon in ST-segment elevation myocardial infarction (STEMI) is associated with a poor clinical prognosis. Although its pathophysiology is not fully elucidated, a deregulated systemic inflammatory response plays an important role. Specifically, the relationship between age-associated differences in inflammatory markers and either no-reflow or mortality in STEMI patients undergoing primary percutaneous coronary intervention (pPCI) has never been investigated.
METHODS AND RESULTS
We retrospectively enrolled 625 consecutive STEMI patients undergoing pPCI for whom a complete laboratory inflammatory pattern was available. Routinely blood measured laboratory parameters were collected at the moment of admission. No reflow was defined as Thrombolysis in Myocardial Infarction (TIMI) flow-grade lower than 3. The population was divided into two groups using a cut-off centered at 65 years. Compared to younger patients, elderly patients had higher mean values of fibrinogen, brain natriuretic peptide (BNP), leukocytes, neutrophil-to-lymphocyte ratio (NLR), C reactive protein/albumin ratio (CAR). Conversely, lymphocyte count and albumin levels were higher in young patients. In elderly patients, the values of NLR, CAR as well as leukocytes, fibrinogen and neutrophils were associated with no-reflow, while in young patients only BNP value was associated. At multivariate Cox regression analysis, only BNP and NLR resulted as independent predictors of all-cause mortality in the whole population and in elderly patients.
CONCLUSIONS
Elderly STEMI patients on admission had a higher acute pro-inflammatory profile than young patients, associated to coronary no-reflow and mortality outcome. These results suggest that a different therapeutic approach between elderly and young STEMI patients should be agreed.

Identifiants

pubmed: 31079969
pii: S0167-5273(19)31196-9
doi: 10.1016/j.ijcard.2019.05.002
pii:
doi:

Substances chimiques

Biomarkers 0
Inflammation Mediators 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

34-39

Commentaires et corrections

Type : CommentIn
Type : CommentIn

Informations de copyright

Copyright © 2019 Elsevier B.V. All rights reserved.

Auteurs

Serena Del Turco (S)

CNR, Institute of Clinical Physiology, Via G. Moruzzi, 1, 56124 Pisa, Italy. Electronic address: serena@ifc.cnr.it.

Giuseppina Basta (G)

CNR, Institute of Clinical Physiology, Via G. Moruzzi, 1, 56124 Pisa, Italy.

Alberto Ranieri De Caterina (AR)

Fondazione G. Monasterio CNR-Regione Toscana Pisa, Massa, Italy.

Silverio Sbrana (S)

CNR, Institute of Clinical Physiology, Via G. Moruzzi, 1, 56124 Pisa, Italy.

Umberto Paradossi (U)

Fondazione G. Monasterio CNR-Regione Toscana Pisa, Massa, Italy.

Alessandro Taddei (A)

Fondazione G. Monasterio CNR-Regione Toscana Pisa, Massa, Italy.

Giuseppe Trianni (G)

Fondazione G. Monasterio CNR-Regione Toscana Pisa, Massa, Italy.

Marcello Ravani (M)

Fondazione G. Monasterio CNR-Regione Toscana Pisa, Massa, Italy.

Cataldo Palmieri (C)

Fondazione G. Monasterio CNR-Regione Toscana Pisa, Massa, Italy.

Sergio Berti (S)

Fondazione G. Monasterio CNR-Regione Toscana Pisa, Massa, Italy.

Annamaria Mazzone (A)

Fondazione G. Monasterio CNR-Regione Toscana Pisa, Massa, Italy.

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Classifications MeSH