Risk factors and diagnostic markers of bacteremia in Stevens-Johnson syndrome and toxic epidermal necrolysis: A cohort study of 176 patients.


Journal

Journal of the American Academy of Dermatology
ISSN: 1097-6787
Titre abrégé: J Am Acad Dermatol
Pays: United States
ID NLM: 7907132

Informations de publication

Date de publication:
Sep 2019
Historique:
received: 06 02 2019
revised: 10 05 2019
accepted: 29 05 2019
pubmed: 14 6 2019
medline: 24 1 2020
entrez: 14 6 2019
Statut: ppublish

Résumé

Sepsis is the main cause of death in Stevens-Johnson syndrome (SJS) and toxic epidermal necrolysis (TEN). Our aim was to identify admission risk factors predictive of bacteremia and the accompanying clinical or biochemical markers associated with positive blood cultures. A retrospective cohort study over a 14-year period (2003-2016) was performed. The study included 176 patients with SJS (n = 59), SJS-TEN overlap (n = 51), and TEN (n = 66). During hospitalization, bacteremia developed in 52 patients (29.5%), who experienced poorer outcomes, including higher intensive care unit admission (P < .0005), longer length of stay (P < .0005), and higher mortality (P < .0005). There were 112 episodes of bacteremia, and isolates included Acinetobacter baumannii (27.7%, n = 31) and Staphylococcus aureus (21.4%, n = 24). On multivariate analysis, clinical factors present at admission that were predictive of bacteremia included hemoglobin ≤10 g/dL (odds ratio [OR] 2.4, confidence interval [CI] 2.2-2.6), existing cardiovascular disease (OR 2.10, CI 2.0-2.3), and body surface area involvement ≥10% (OR 14.3, CI 13.4-15.2). The Bacteremia Risk Score was constructed with good calibration. Hypothermia (P = .03) and procalcitonin ≥1 μg/L (P = .02) concurrent with blood culture sampling were predictive of blood culture positivity. This is a retrospective study performed in a reference center. Hemoglobin ≤10 g/dL, cardiovascular disease, and body surface area involvement ≥10% on admission were risk factors for bacteremia. Hypothermia and elevated procalcitonin are useful markers for the timely detection of bacteremia.

Sections du résumé

BACKGROUND BACKGROUND
Sepsis is the main cause of death in Stevens-Johnson syndrome (SJS) and toxic epidermal necrolysis (TEN).
OBJECTIVES OBJECTIVE
Our aim was to identify admission risk factors predictive of bacteremia and the accompanying clinical or biochemical markers associated with positive blood cultures.
METHODS METHODS
A retrospective cohort study over a 14-year period (2003-2016) was performed.
RESULTS RESULTS
The study included 176 patients with SJS (n = 59), SJS-TEN overlap (n = 51), and TEN (n = 66). During hospitalization, bacteremia developed in 52 patients (29.5%), who experienced poorer outcomes, including higher intensive care unit admission (P < .0005), longer length of stay (P < .0005), and higher mortality (P < .0005). There were 112 episodes of bacteremia, and isolates included Acinetobacter baumannii (27.7%, n = 31) and Staphylococcus aureus (21.4%, n = 24). On multivariate analysis, clinical factors present at admission that were predictive of bacteremia included hemoglobin ≤10 g/dL (odds ratio [OR] 2.4, confidence interval [CI] 2.2-2.6), existing cardiovascular disease (OR 2.10, CI 2.0-2.3), and body surface area involvement ≥10% (OR 14.3, CI 13.4-15.2). The Bacteremia Risk Score was constructed with good calibration. Hypothermia (P = .03) and procalcitonin ≥1 μg/L (P = .02) concurrent with blood culture sampling were predictive of blood culture positivity.
LIMITATIONS CONCLUSIONS
This is a retrospective study performed in a reference center.
CONCLUSION CONCLUSIONS
Hemoglobin ≤10 g/dL, cardiovascular disease, and body surface area involvement ≥10% on admission were risk factors for bacteremia. Hypothermia and elevated procalcitonin are useful markers for the timely detection of bacteremia.

Identifiants

pubmed: 31195022
pii: S0190-9622(19)30901-6
doi: 10.1016/j.jaad.2019.05.096
pii:
doi:

Substances chimiques

Hemoglobins 0
Procalcitonin 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

686-693

Commentaires et corrections

Type : CommentIn
Type : CommentIn
Type : CommentIn

Informations de copyright

Copyright © 2019 American Academy of Dermatology, Inc. Published by Elsevier Inc. All rights reserved.

Auteurs

Hui Kai Koh (HK)

Yong Loo Lin School of Medicine, National University of Singapore, Singapore.

Zi Teng Chai (ZT)

Department of Dermatology, Singapore General Hospital, Singapore.

Hui Wen Tay (HW)

Yong Loo Lin School of Medicine, National University of Singapore, Singapore.

Stephanie Fook-Chong (S)

Health Services Research Unit, Division of Medicine, Singapore General Hospital, Singapore.

Karen J L Choo (KJL)

Department of Dermatology, Singapore General Hospital, Singapore.

Choon Chiat Oh (CC)

Department of Dermatology, Singapore General Hospital, Singapore.

Yi Wei Yeo (YW)

Department of Dermatology, Singapore General Hospital, Singapore.

Hong Yi Koh (HY)

Department of Dermatology, Singapore General Hospital, Singapore.

Shiu Ming Pang (SM)

Department of Dermatology, Singapore General Hospital, Singapore.

Haur Yueh Lee (HY)

Department of Dermatology, Singapore General Hospital, Singapore. Electronic address: lee.haur.yueh@singhealth.com.sg.

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Classifications MeSH