The Impact of Ethinyl Estradiol on Metformin Action on Prolactin Levels in Women with Hyperprolactinemia.


Journal

Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association
ISSN: 1439-3646
Titre abrégé: Exp Clin Endocrinol Diabetes
Pays: Germany
ID NLM: 9505926

Informations de publication

Date de publication:
Jan 2021
Historique:
pubmed: 25 6 2019
medline: 5 10 2021
entrez: 25 6 2019
Statut: ppublish

Résumé

Metformin reduced prolactin levels only in women with hyperprolactinemia. The purpose of this case-control study was to compare metformin action on lactoctrope function between women receiving oral contraceptive pills and women not using hormonal contraception. The study included two groups of matched women with elevated prolactin levels and new-onset prediabetes or diabetes. The first group consisted of 20 women using oral contraceptive pills for at least 12 months before entering the study, while the second group included 20 patients not using any hormonal contraception. Over the whole study period, all women were treated with metformin (1.7-3 g daily). Circulating levels of glucose, insulin, prolactin, thyrotropin, free thyroid hormones, adrenocorticotropic hormone, gonadotropins and insulin-like growth factor-1 were measured at the beginning and at the end of the study (16 weeks later). Thirty-eight patients completed the study. Metformin reduced plasma glucose levels and improved insulin sensitivity but the latter effect was stronger in women receiving oral contraceptive pills than in women not using any contraception. Although metformin treatment decreased plasma prolactin levels in both study groups, this effect was stronger in women taking oral contraceptive pills. Only in this group of women, metformin increased plasma luteinizing hormone levels. The changes in plasma prolactin correlated with their baseline insulin sensitivity and the effect of metformin on insulin sensitivity. Metformin did not affect plasma levels of thyrotropin, free thyroxine, free triiodothyronine, follicle-stimulating hormone, adrenocorticotropic hormone and insulin-like growth factor-1. The obtained results suggest that the effect of metformin on overactive lactotropes depends on estrogen levels.

Sections du résumé

BACKGROUND BACKGROUND
Metformin reduced prolactin levels only in women with hyperprolactinemia.
OBJECTIVE OBJECTIVE
The purpose of this case-control study was to compare metformin action on lactoctrope function between women receiving oral contraceptive pills and women not using hormonal contraception.
METHODS METHODS
The study included two groups of matched women with elevated prolactin levels and new-onset prediabetes or diabetes. The first group consisted of 20 women using oral contraceptive pills for at least 12 months before entering the study, while the second group included 20 patients not using any hormonal contraception. Over the whole study period, all women were treated with metformin (1.7-3 g daily). Circulating levels of glucose, insulin, prolactin, thyrotropin, free thyroid hormones, adrenocorticotropic hormone, gonadotropins and insulin-like growth factor-1 were measured at the beginning and at the end of the study (16 weeks later).
RESULTS RESULTS
Thirty-eight patients completed the study. Metformin reduced plasma glucose levels and improved insulin sensitivity but the latter effect was stronger in women receiving oral contraceptive pills than in women not using any contraception. Although metformin treatment decreased plasma prolactin levels in both study groups, this effect was stronger in women taking oral contraceptive pills. Only in this group of women, metformin increased plasma luteinizing hormone levels. The changes in plasma prolactin correlated with their baseline insulin sensitivity and the effect of metformin on insulin sensitivity. Metformin did not affect plasma levels of thyrotropin, free thyroxine, free triiodothyronine, follicle-stimulating hormone, adrenocorticotropic hormone and insulin-like growth factor-1.
CONCLUSIONS CONCLUSIONS
The obtained results suggest that the effect of metformin on overactive lactotropes depends on estrogen levels.

Identifiants

pubmed: 31234219
doi: 10.1055/a-0921-6420
doi:

Substances chimiques

Blood Glucose 0
Contraceptives, Oral, Hormonal 0
Hypoglycemic Agents 0
Ethinyl Estradiol 423D2T571U
Prolactin 9002-62-4
Luteinizing Hormone 9002-67-9
Metformin 9100L32L2N

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

22-28

Informations de copyright

Thieme. All rights reserved.

Déclaration de conflit d'intérêts

The authors declare no conflict of interest.

Auteurs

Robert Krysiak (R)

Department of Internal Medicine and Clinical Pharmacology, Medical University of Silesia, Katowice, Poland.

Witold Szkróbka (W)

Department of Internal Medicine and Clinical Pharmacology, Medical University of Silesia, Katowice, Poland.

Bogusław Okopień (B)

Department of Internal Medicine and Clinical Pharmacology, Medical University of Silesia, Katowice, Poland.

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Classifications MeSH