The Association of Therapy Adherence and Thyroid Function in Adult Patients with Phenylketonuria.


Journal

Annals of nutrition & metabolism
ISSN: 1421-9697
Titre abrégé: Ann Nutr Metab
Pays: Switzerland
ID NLM: 8105511

Informations de publication

Date de publication:
2019
Historique:
received: 05 02 2019
accepted: 04 06 2019
pubmed: 27 6 2019
medline: 6 6 2020
entrez: 27 6 2019
Statut: ppublish

Résumé

The standard, lifelong therapy of phenylketonuria (PKU) is a natural protein-restricted diet complemented with phenylalanine (Phe)-free L-amino acid mixtures that provide the daily necessary micronutrients. To assess thyroid function and structure and the iodine status of early-treated adult PKU (ETPKU) patients in Hungary. Sixty-nine PKU patients (aged 18-41 years) and 50 healthy controls were enrolled in the study. Thyroid hormones, serum thyroglobulin, thyroid antibodies, urinary iodine, and selenium concentrations were measured, and thyroid ultrasound was performed. The incidence of thyroid dysfunction was infrequent (n = 2). Blood Phe was negatively correlated with thyroid-stimulating hormone (TSH), and PKU patients had higher free thyroxine and lower TSH levels than healthy controls. Although optimal iodine status was found in the entire PKU population, by dividing the patients according to their therapy compliance, we observed that lower therapy adherence was associated with mild iodine deficiency and lower urinary selenium levels. The results of this study suggest that iodine status is strongly influenced by the adherence to therapy in ETPKU patients. No or not enough medical food consumption combined with a low-Phe diet can lead to subclinical iodine deficiency.

Sections du résumé

BACKGROUND
The standard, lifelong therapy of phenylketonuria (PKU) is a natural protein-restricted diet complemented with phenylalanine (Phe)-free L-amino acid mixtures that provide the daily necessary micronutrients.
OBJECTIVE
To assess thyroid function and structure and the iodine status of early-treated adult PKU (ETPKU) patients in Hungary.
METHODS
Sixty-nine PKU patients (aged 18-41 years) and 50 healthy controls were enrolled in the study. Thyroid hormones, serum thyroglobulin, thyroid antibodies, urinary iodine, and selenium concentrations were measured, and thyroid ultrasound was performed.
RESULTS
The incidence of thyroid dysfunction was infrequent (n = 2). Blood Phe was negatively correlated with thyroid-stimulating hormone (TSH), and PKU patients had higher free thyroxine and lower TSH levels than healthy controls. Although optimal iodine status was found in the entire PKU population, by dividing the patients according to their therapy compliance, we observed that lower therapy adherence was associated with mild iodine deficiency and lower urinary selenium levels.
CONCLUSIONS
The results of this study suggest that iodine status is strongly influenced by the adherence to therapy in ETPKU patients. No or not enough medical food consumption combined with a low-Phe diet can lead to subclinical iodine deficiency.

Identifiants

pubmed: 31242484
pii: 000501301
doi: 10.1159/000501301
doi:

Substances chimiques

Phenylalanine 47E5O17Y3R
Thyrotropin 9002-71-5
Thyroglobulin 9010-34-8
Iodine 9679TC07X4
Selenium H6241UJ22B
Thyroxine Q51BO43MG4

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

16-23

Informations de copyright

© 2019 S. Karger AG, Basel.

Auteurs

Csaba Sumanszki (C)

2nd Department of Medicine, Semmelweis University, Budapest, Hungary.

Erika Kiss (E)

1st Department of Pediatrics, Semmelweis University, Budapest, Hungary.

Erika Simon (E)

1st Department of Pediatrics, Semmelweis University, Budapest, Hungary.

Erika Galgoczi (E)

Division of Endocrinology, Department of Medicine, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.

Aron Soos (A)

Institute of Food Science, Faculty of Agricultural and Food Science and Environmental Management, University of Debrecen, Debrecen, Hungary.

Attila Patocs (A)

Department of Laboratory Medicine, Semmelweis University, Budapest, Hungary.
"Lendület" Hereditary Endocrine Tumours Research Group, Hungarian Academy of Sciences and Semmelweis University, Budapest, Hungary.

Bela Kovacs (B)

Institute of Food Science, Faculty of Agricultural and Food Science and Environmental Management, University of Debrecen, Debrecen, Hungary.

Endre V Nagy (EV)

Division of Endocrinology, Department of Medicine, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.

Peter Reismann (P)

2nd Department of Medicine, Semmelweis University, Budapest, Hungary, reismann.peter@med.semmelweis-univ.hu.

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