Inflammatory Arthritis as a Possible Feature of Coffin-Siris Syndrome.


Journal

Pediatrics
ISSN: 1098-4275
Titre abrégé: Pediatrics
Pays: United States
ID NLM: 0376422

Informations de publication

Date de publication:
07 2019
Historique:
accepted: 14 03 2019
pubmed: 28 6 2019
medline: 18 12 2019
entrez: 28 6 2019
Statut: ppublish

Résumé

Coffin-Siris syndrome (CSS) and Nicolaides-Baraitser syndrome (NBS) are 2 overlapping syndromes caused by mutations in genes of the barrier-to-autointegration factor chromatin-remodeling complex, presenting with multiple malformations and intellectual disability. Musculoskeletal changes such as noninflammatory prominence of interphalangeal joints in hands, feet, and, to a lesser extent, knee joints are common in NBS (up to 85%) and also reported in CSS. We present the case of a 7-year-old boy with polyarthritis of several years' duration (without uveitis), developmental delay, microcephaly, and dysmorphic features reminiscent of NBS. Sanger sequencing of the

Identifiants

pubmed: 31243159
pii: peds.2018-1741
doi: 10.1542/peds.2018-1741
pii:
doi:

Substances chimiques

ARID1B protein, human 0
Anti-Inflammatory Agents, Non-Steroidal 0
Codon, Nonsense 0
DNA-Binding Proteins 0
Transcription Factors 0
Etanercept OP401G7OJC
Methotrexate YL5FZ2Y5U1

Types de publication

Case Reports Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Wellcome Trust
ID : 209568/Z/17/Z
Pays : United Kingdom

Informations de copyright

Copyright © 2019 by the American Academy of Pediatrics.

Déclaration de conflit d'intérêts

POTENTIAL CONFLICT OF INTEREST: Dr Brogan has received a grant from Novartis, consulting fees from Roche, speaker fees from UCB, committee fees and an institutional grant from Swedish Orphan Biovitrum for gene hunting in autoinflammation, an institutional grant from Novimmune for a clinical trial, and other institutional fees from ChemoCentryx; the other authors have indicated they have no potential conflicts of interest to disclose.

Auteurs

Sonia Melo Gomes (S)

Department of Infection, Immunity, and Inflammation, University College London Great Ormond Street Institute of Child Health, London, United Kingdom; s.melogomes@ucl.ac.uk.
Departments of Rheumatology.

Cristina Dias (C)

Clinical Genetics, and.
Department of Medical and Molecular Genetics, School of Basic and Medical Biosciences, King's College London, London, United Kingdom; and.
The Francis Crick Institute, London, United Kingdom.

Ebun Omoyinmi (E)

Department of Infection, Immunity, and Inflammation, University College London Great Ormond Street Institute of Child Health, London, United Kingdom.

Sandrine Compeyrot-Lacassagne (S)

Departments of Rheumatology.

Nigel Klein (N)

Department of Infection, Immunity, and Inflammation, University College London Great Ormond Street Institute of Child Health, London, United Kingdom.

Neil J Sebire (NJ)

Histopathology, Great Ormond Street Hospital, London, United Kingdom.

Paul Brogan (P)

Department of Infection, Immunity, and Inflammation, University College London Great Ormond Street Institute of Child Health, London, United Kingdom.
Departments of Rheumatology.

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Classifications MeSH