Pioneer and nonpioneer factor cooperation drives lineage specific chromatin opening.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
23 08 2019
Historique:
received: 15 01 2019
accepted: 30 07 2019
entrez: 25 8 2019
pubmed: 25 8 2019
medline: 27 12 2019
Statut: epublish

Résumé

Pioneer transcription factors are characterized by having the unique property of enabling the opening of closed chromatin sites, for implementation of cell fates. We previously found that the pioneer Pax7 specifies melanotrope cells through deployment of an enhancer repertoire, which allows binding of Tpit, a nonpioneer factor that determines the related lineages of melanotropes and corticotropes. Here, we investigate the relation between these two factors in the pioneer mechanism. Cell-specific gene expression and chromatin landscapes are defined by scRNAseq and chromatin accessibility profiling. We find that in vivo deployment of the melanotrope enhancer repertoire and chromatin opening requires both Pax7 and Tpit. In cells, binding of heterochromatin targets by Pax7 is independent of Tpit but Pax7-dependent chromatin opening requires Tpit. The present work shows that pioneer core properties are limited to the ability to recognize heterochromatin targets and facilitate nonpioneer binding. Chromatin opening per se may be provided through cooperation with nonpioneer factors.

Identifiants

pubmed: 31444346
doi: 10.1038/s41467-019-11791-9
pii: 10.1038/s41467-019-11791-9
pmc: PMC6707328
doi:

Substances chimiques

Heterochromatin 0
Homeodomain Proteins 0
PAX7 Transcription Factor 0
Pax7 protein, mouse 0
T-Box Domain Proteins 0
Tbx19 protein, mouse 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3807

Subventions

Organisme : Gouvernement du Canada | Canadian Institutes of Health Research (Instituts de Recherche en Santé du Canada)
ID : FDN-154297
Pays : International

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Auteurs

Alexandre Mayran (A)

Laboratoire de Génétique Moléculaire, Institut de Recherches Cliniques de Montréal (IRCM), Montreal, QC, Canada.
Department of Biochemistry, McGill University, Montreal, QC, Canada.

Kevin Sochodolsky (K)

Laboratoire de Génétique Moléculaire, Institut de Recherches Cliniques de Montréal (IRCM), Montreal, QC, Canada.

Konstantin Khetchoumian (K)

Laboratoire de Génétique Moléculaire, Institut de Recherches Cliniques de Montréal (IRCM), Montreal, QC, Canada.

Juliette Harris (J)

Laboratoire de Génétique Moléculaire, Institut de Recherches Cliniques de Montréal (IRCM), Montreal, QC, Canada.

Yves Gauthier (Y)

Laboratoire de Génétique Moléculaire, Institut de Recherches Cliniques de Montréal (IRCM), Montreal, QC, Canada.

Amandine Bemmo (A)

Laboratoire de Génétique Moléculaire, Institut de Recherches Cliniques de Montréal (IRCM), Montreal, QC, Canada.

Aurelio Balsalobre (A)

Laboratoire de Génétique Moléculaire, Institut de Recherches Cliniques de Montréal (IRCM), Montreal, QC, Canada.

Jacques Drouin (J)

Laboratoire de Génétique Moléculaire, Institut de Recherches Cliniques de Montréal (IRCM), Montreal, QC, Canada. jacques.drouin@ircm.qc.ca.
Department of Biochemistry, McGill University, Montreal, QC, Canada. jacques.drouin@ircm.qc.ca.
Département de Biochimie, Université de Montréal, Montreal, QC, Canada. jacques.drouin@ircm.qc.ca.

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Classifications MeSH