Role of dopamine and selective dopamine receptor agonists on mouse ductus arteriosus tone and responsiveness.


Journal

Pediatric research
ISSN: 1530-0447
Titre abrégé: Pediatr Res
Pays: United States
ID NLM: 0100714

Informations de publication

Date de publication:
05 2020
Historique:
received: 24 06 2019
accepted: 23 11 2019
revised: 19 11 2019
pubmed: 10 12 2019
medline: 1 6 2021
entrez: 10 12 2019
Statut: ppublish

Résumé

Indomethacin treatment for patent ductus arteriosus (PDA) is associated with acute kidney injury (AKI). Fenoldopam, a dopamine (DA) DA DA receptor expression in CD-1 mouse vessels was analyzed by qPCR and immunohistochemistry. Concentration-response curves were established using pressure myography. Pretreatment with SCH23390 (DA DA DA receptors are present in the ductus but have limited physiologic effects. DA-induced ductus vasoconstriction is mediated via α-adrenergic pathways. The absence of DA

Sections du résumé

BACKGROUND
Indomethacin treatment for patent ductus arteriosus (PDA) is associated with acute kidney injury (AKI). Fenoldopam, a dopamine (DA) DA
METHODS
DA receptor expression in CD-1 mouse vessels was analyzed by qPCR and immunohistochemistry. Concentration-response curves were established using pressure myography. Pretreatment with SCH23390 (DA
RESULTS
DA
CONCLUSION(S)
DA receptors are present in the ductus but have limited physiologic effects. DA-induced ductus vasoconstriction is mediated via α-adrenergic pathways. The absence of DA

Identifiants

pubmed: 31816622
doi: 10.1038/s41390-019-0716-x
pii: 10.1038/s41390-019-0716-x
pmc: PMC7196482
mid: NIHMS1544972
doi:

Substances chimiques

Dopamine Agonists 0
Drd1 protein, mouse 0
Receptors, Dopamine D1 0
Vasodilator Agents 0
Fenoldopam INU8H2KAWG
Oxygen S88TT14065
Dopamine VTD58H1Z2X
Indomethacin XXE1CET956

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

991-997

Subventions

Organisme : NHLBI NIH HHS
ID : R01 HL109199
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL128386
Pays : United States
Organisme : NIDDK NIH HHS
ID : R21 DK113073
Pays : United States
Organisme : NHLBI NIH HHS
ID : R21 HL132805
Pays : United States

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Auteurs

Stacey L Crockett (SL)

Department of Pediatrics, Vanderbilt University Medical Center, Nashville, TN, USA.

Micah Harris (M)

Department of Pediatrics, Vanderbilt University Medical Center, Nashville, TN, USA.

Naoko Boatwright (N)

Department of Pediatrics, Vanderbilt University Medical Center, Nashville, TN, USA.

Rachel L Su (RL)

Department of Pediatrics, Vanderbilt University Medical Center, Nashville, TN, USA.

Michael T Yarboro (MT)

Department of Cell and Developmental Biology, Vanderbilt University, Nashville, TN, USA.

Courtney D Berger (CD)

Department of Pediatrics, Vanderbilt University Medical Center, Nashville, TN, USA.

Elaine L Shelton (EL)

Department of Pediatrics, Vanderbilt University Medical Center, Nashville, TN, USA.
Department of Pharmacology, Vanderbilt University, Nashville, TN, USA.

Jeff Reese (J)

Department of Pediatrics, Vanderbilt University Medical Center, Nashville, TN, USA.
Department of Cell and Developmental Biology, Vanderbilt University, Nashville, TN, USA.

Jeffrey L Segar (JL)

Department of Pediatrics, Medical College of Wisconsin, Milwaukee, WI, USA. jsegar@mcw.edu.

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Classifications MeSH