Effect of Oxygen Therapy on Cardiovascular Outcomes in Relation to Baseline Oxygen Saturation.


Journal

JACC. Cardiovascular interventions
ISSN: 1876-7605
Titre abrégé: JACC Cardiovasc Interv
Pays: United States
ID NLM: 101467004

Informations de publication

Date de publication:
24 02 2020
Historique:
received: 19 06 2019
revised: 13 08 2019
accepted: 04 09 2019
pubmed: 16 12 2019
medline: 21 10 2020
entrez: 16 12 2019
Statut: ppublish

Résumé

The aim of this study was to determine the effect of supplemental oxygen in patients with myocardial infarction (MI) on the composite of all-cause death, rehospitalization with MI, or heart failure related to baseline oxygen saturation. A secondary objective was to investigate outcomes in patients developing hypoxemia. In the DETO2X-AMI (Determination of the Role of Oxygen in Suspected Acute Myocardial Infarction) trial, 6,629 normoxemic patients with suspected MI were randomized to oxygen at 6 l/min for 6 to 12 h or ambient air. The study population of 5,010 patients with confirmed MI was divided by baseline oxygen saturation into a low-normal (90% to 94%) and a high-normal (95% to 100%) cohort. Outcomes are reported within 1 year. To increase power, all follow-up time (between 1 and 4 years) was included post hoc, and interaction analyses were performed with oxygen saturation as a continuous covariate. The composite endpoint of all-cause death, rehospitalization with MI, or heart failure occurred significantly more often in patients in the low-normal cohort (17.3%) compared with those in the high-normal cohort (9.5%) (p < 0.001), and most often in patients developing hypoxemia (23.6%). Oxygen therapy compared with ambient air was not associated with improved outcomes regardless of baseline oxygen saturation (interaction p values: composite endpoint, p = 0.79; all-cause death, p = 0.33; rehospitalization with MI, p = 0.86; hospitalization for heart failure, p = 0.35). Irrespective of oxygen saturation at baseline, we found no clinically relevant beneficial effect of routine oxygen therapy in normoxemic patients with MI regarding cardiovascular outcomes. Low-normal baseline oxygen saturation or development of hypoxemia was identified as an independent marker of poor prognosis. (An Efficacy and Outcome Study of Supplemental Oxygen Treatment in Patients With Suspected Myocardial Infarction; NCT01787110).

Sections du résumé

OBJECTIVES
The aim of this study was to determine the effect of supplemental oxygen in patients with myocardial infarction (MI) on the composite of all-cause death, rehospitalization with MI, or heart failure related to baseline oxygen saturation. A secondary objective was to investigate outcomes in patients developing hypoxemia.
BACKGROUND
In the DETO2X-AMI (Determination of the Role of Oxygen in Suspected Acute Myocardial Infarction) trial, 6,629 normoxemic patients with suspected MI were randomized to oxygen at 6 l/min for 6 to 12 h or ambient air.
METHODS
The study population of 5,010 patients with confirmed MI was divided by baseline oxygen saturation into a low-normal (90% to 94%) and a high-normal (95% to 100%) cohort. Outcomes are reported within 1 year. To increase power, all follow-up time (between 1 and 4 years) was included post hoc, and interaction analyses were performed with oxygen saturation as a continuous covariate.
RESULTS
The composite endpoint of all-cause death, rehospitalization with MI, or heart failure occurred significantly more often in patients in the low-normal cohort (17.3%) compared with those in the high-normal cohort (9.5%) (p < 0.001), and most often in patients developing hypoxemia (23.6%). Oxygen therapy compared with ambient air was not associated with improved outcomes regardless of baseline oxygen saturation (interaction p values: composite endpoint, p = 0.79; all-cause death, p = 0.33; rehospitalization with MI, p = 0.86; hospitalization for heart failure, p = 0.35).
CONCLUSIONS
Irrespective of oxygen saturation at baseline, we found no clinically relevant beneficial effect of routine oxygen therapy in normoxemic patients with MI regarding cardiovascular outcomes. Low-normal baseline oxygen saturation or development of hypoxemia was identified as an independent marker of poor prognosis. (An Efficacy and Outcome Study of Supplemental Oxygen Treatment in Patients With Suspected Myocardial Infarction; NCT01787110).

Identifiants

pubmed: 31838113
pii: S1936-8798(19)31940-5
doi: 10.1016/j.jcin.2019.09.016
pii:
doi:

Substances chimiques

Biomarkers 0
Oxygen S88TT14065

Banques de données

ClinicalTrials.gov
['NCT01787110']

Types de publication

Journal Article Multicenter Study Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

502-513

Investigateurs

Leif Svensson (L)
Johan Herlitz (J)
Robin Hofmann (R)
Nils Witt (N)
Mats Frick (M)
Tomas Jernberg (T)
Mattias Ekström (M)
Rickard Linder (R)
Lennart Nilsson (L)
Joakim Alfredsson (J)
David Erlinge (D)
David Zughaft (D)
Ulf Ekelund (U)
Annica Ravn-Fischer (A)
Elmir Omerovic (E)
Thomas Kellerth (T)
Stefan James (S)
Bertil Lindahl (B)
Ollie Östlund (O)
Leif Svensson (L)
Johan Herlitz (J)
Robin Hofmann (R)
Nils Witt (N)
David Erlinge (D)
Tomas Jernberg (T)
Bertil Lindahl (B)
Stefan James (S)
Eva Jacobsson (E)
Ollie Östlund (O)

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2020 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved.

Auteurs

Stefan K James (SK)

Department of Medical Sciences, Cardiology, Uppsala University, Uppsala, Sweden; Uppsala Clinical Research Center, Uppsala University, Uppsala, Sweden.

David Erlinge (D)

Department of Clinical Sciences, Cardiology, Lund University, Lund, Sweden.

Johan Herlitz (J)

Department of Health Sciences, University of Borås, Borås, Sweden.

Joakim Alfredsson (J)

Department of Medical and Health Sciences and Department of Cardiology, Linköping University, Linköping, Sweden.

Sasha Koul (S)

Department of Clinical Sciences, Cardiology, Lund University, Lund, Sweden.

Ole Fröbert (O)

Department of Cardiology, Faculty of Medicine and Health, Örebro University, Örebro, Sweden.

Thomas Kellerth (T)

Department of Cardiology, Faculty of Medicine and Health, Örebro University, Örebro, Sweden.

Annica Ravn-Fischer (A)

Department of Molecular and Clinical Medicine and Sahlgrenska University Hospital, Department of Cardiology, University of Gothenburg, Gothenburg, Sweden.

Patrik Alström (P)

Department of Clinical Science and Education, Division of Cardiology, Karolinska Institutet, Södersjukhuset, Stockholm, Sweden.

Ollie Östlund (O)

Uppsala Clinical Research Center, Uppsala University, Uppsala, Sweden.

Tomas Jernberg (T)

Department of Clinical Sciences, Cardiology, Karolinska Institutet, Danderyd Hospital, Stockholm, Sweden.

Bertil Lindahl (B)

Department of Medical Sciences, Cardiology, Uppsala University, Uppsala, Sweden; Uppsala Clinical Research Center, Uppsala University, Uppsala, Sweden.

Robin Hofmann (R)

Department of Clinical Science and Education, Division of Cardiology, Karolinska Institutet, Södersjukhuset, Stockholm, Sweden. Electronic address: robin.hofmann@sll.se.

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