Hypertrichotic patches as a mosaic manifestation of Proteus syndrome.


Journal

Journal of the American Academy of Dermatology
ISSN: 1097-6787
Titre abrégé: J Am Acad Dermatol
Pays: United States
ID NLM: 7907132

Informations de publication

Date de publication:
Feb 2021
Historique:
received: 15 08 2019
revised: 28 01 2020
accepted: 31 01 2020
pubmed: 10 2 2020
medline: 30 7 2021
entrez: 10 2 2020
Statut: ppublish

Résumé

Proteus syndrome is an overgrowth disorder caused by a mosaic activating AKT1 variant. Hair abnormalities in Proteus syndrome have rarely been reported, and frequencies of such findings have not been elucidated. To define the types and frequencies of hair findings in individuals with Proteus syndrome. A cross-sectional study was conducted of individuals with clinical features of Proteus syndrome and a confirmed pathogenic variant in AKT1 evaluated between November 1996 and June 2019 at the National Institutes of Health Clinical Center. Medical records were reviewed for patterning, density, and color of hair on the body and scalp. Of 45 individuals evaluated, 29 (64%) had asymmetric hypertrichosis on the body. This included unilateral blaschkoid hypertrichotic patches overlying normal skin or epidermal nevi in 16 (36%), unilateral nonblaschkoid hypertrichotic patches in 11 (24%), and unilateral limb hypertrichosis in 10 (22%). Diffuse, scattered, or patchy changes in scalp hair density or color were present in 11 individuals (24%). The retrospective, observational design, and limited longitudinal follow-up. Asymmetric variations in hair distribution, thickness, length, and color contribute to the overall mosaic appearance of the skin in Proteus syndrome, an observation that provides novel insights into the role of phosphoinositide 3-kinase (PI3K)-protein kinase B (AKT) signaling in skin appendage development.

Sections du résumé

BACKGROUND BACKGROUND
Proteus syndrome is an overgrowth disorder caused by a mosaic activating AKT1 variant. Hair abnormalities in Proteus syndrome have rarely been reported, and frequencies of such findings have not been elucidated.
OBJECTIVE OBJECTIVE
To define the types and frequencies of hair findings in individuals with Proteus syndrome.
METHODS METHODS
A cross-sectional study was conducted of individuals with clinical features of Proteus syndrome and a confirmed pathogenic variant in AKT1 evaluated between November 1996 and June 2019 at the National Institutes of Health Clinical Center. Medical records were reviewed for patterning, density, and color of hair on the body and scalp.
RESULTS RESULTS
Of 45 individuals evaluated, 29 (64%) had asymmetric hypertrichosis on the body. This included unilateral blaschkoid hypertrichotic patches overlying normal skin or epidermal nevi in 16 (36%), unilateral nonblaschkoid hypertrichotic patches in 11 (24%), and unilateral limb hypertrichosis in 10 (22%). Diffuse, scattered, or patchy changes in scalp hair density or color were present in 11 individuals (24%).
LIMITATIONS CONCLUSIONS
The retrospective, observational design, and limited longitudinal follow-up.
CONCLUSIONS CONCLUSIONS
Asymmetric variations in hair distribution, thickness, length, and color contribute to the overall mosaic appearance of the skin in Proteus syndrome, an observation that provides novel insights into the role of phosphoinositide 3-kinase (PI3K)-protein kinase B (AKT) signaling in skin appendage development.

Identifiants

pubmed: 32035943
pii: S0190-9622(20)30167-5
doi: 10.1016/j.jaad.2020.01.078
pmc: PMC7415737
mid: NIHMS1568923
pii:
doi:

Substances chimiques

AKT1 protein, human EC 2.7.11.1
Proto-Oncogene Proteins c-akt EC 2.7.11.1

Types de publication

Journal Article Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

415-424

Subventions

Organisme : Intramural NIH HHS
ID : ZIA HG200388
Pays : United States

Informations de copyright

Published by Elsevier Inc.

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Auteurs

Deeti J Pithadia (DJ)

Department of Dermatology, Uniformed Services University, Bethesda, Maryland.

John W Roman (JW)

Department of Dermatology, Uniformed Services University, Bethesda, Maryland.

Julie C Sapp (JC)

National Human Genome Research Institute, National Institutes of Health, Bethesda, Maryland.

Leslie G Biesecker (LG)

National Human Genome Research Institute, National Institutes of Health, Bethesda, Maryland.

Thomas N Darling (TN)

Department of Dermatology, Uniformed Services University, Bethesda, Maryland. Electronic address: thomas.darling@usuhs.edu.

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Classifications MeSH