Comorbid argyrophilic grain disease in an 87-year-old male with spinocerebellar ataxia type 31 with dementia: a case report.


Journal

BMC neurology
ISSN: 1471-2377
Titre abrégé: BMC Neurol
Pays: England
ID NLM: 100968555

Informations de publication

Date de publication:
15 Apr 2020
Historique:
received: 03 12 2019
accepted: 12 04 2020
entrez: 16 4 2020
pubmed: 16 4 2020
medline: 25 8 2020
Statut: epublish

Résumé

Spinocerebellar ataxia type 31 (SCA31) is not usually associated with dementia, and autopsy in a patient with both conditions is very rare. An 87-year-old male patient presented with ataxia and progressive dementia. Genetic testing led to a diagnosis of SCA31. Fifteen years after his initial symptoms of hearing loss and difficulty walking, he died of aspiration pneumonia. A pathological analysis showed cerebellar degeneration consistent with SCA31 and abundant argyrophilic grains in the hippocampal formation and amygdala that could explain his dementia. This is the first autopsy report on comorbid argyrophilic grain disease with SCA31.

Sections du résumé

BACKGROUND BACKGROUND
Spinocerebellar ataxia type 31 (SCA31) is not usually associated with dementia, and autopsy in a patient with both conditions is very rare.
CASE PRESENTATION METHODS
An 87-year-old male patient presented with ataxia and progressive dementia. Genetic testing led to a diagnosis of SCA31. Fifteen years after his initial symptoms of hearing loss and difficulty walking, he died of aspiration pneumonia. A pathological analysis showed cerebellar degeneration consistent with SCA31 and abundant argyrophilic grains in the hippocampal formation and amygdala that could explain his dementia.
CONCLUSIONS CONCLUSIONS
This is the first autopsy report on comorbid argyrophilic grain disease with SCA31.

Identifiants

pubmed: 32293309
doi: 10.1186/s12883-020-01723-2
pii: 10.1186/s12883-020-01723-2
pmc: PMC7158122
doi:

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

136

Références

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pubmed: 27830516
Neurology. 2005 Aug 23;65(4):629-32
pubmed: 16116133
Acta Neurol Scand. 2007 Aug;116(2):123-7
pubmed: 17661799
Mov Disord. 2007 Apr 30;22(6):857-62
pubmed: 17357132
Neuropathology. 2015 Jun;35(3):273-9
pubmed: 25495291
J Neuropathol Exp Neurol. 2004 Sep;63(9):911-8
pubmed: 15453090
J Hum Genet. 2007;52(10):848-855
pubmed: 17805477

Auteurs

Shuta Toru (S)

Department of Neurology, Nitobe Memorial Nakano General Hospital, 4-59-16 Chuo, Nakano-ku, Tokyo, 164-8607, Japan. shuta-toru@nakanosogo.or.jp.

Shoko Ishida (S)

Department of Pathology, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo, 113-8519, Japan.

Toshiki Uchihara (T)

Department of Neurology, Nitobe Memorial Nakano General Hospital, 4-59-16 Chuo, Nakano-ku, Tokyo, 164-8607, Japan.

Katsuiku Hirokawa (K)

Department of Pathology, Nitobe Memorial Nakano General Hospital, 4-59-16 Chuo, Nakano-ku, Tokyo, 164-8607, Japan.

Masanobu Kitagawa (M)

Department of Pathology, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo, 113-8519, Japan.

Kinya Ishikawa (K)

Department of Neurology, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo, 113-8519, Japan.

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