Genetic variants associated with Hermansky-Pudlak syndrome.
Aminocaproic Acid
/ pharmacology
Antifibrinolytic Agents
/ pharmacology
Blood Platelets
/ metabolism
Carrier Proteins
/ genetics
Contusions
/ genetics
Deamino Arginine Vasopressin
/ therapeutic use
Hemorrhage
/ genetics
Hermanski-Pudlak Syndrome
/ drug therapy
Humans
Hypopigmentation
/ genetics
Lysosomes
/ genetics
Nerve Tissue Proteins
/ genetics
Proteins
/ genetics
Tranexamic Acid
/ pharmacology
Hermansky-Pudlak syndrome
platelet delta granule
Journal
Platelets
ISSN: 1369-1635
Titre abrégé: Platelets
Pays: England
ID NLM: 9208117
Informations de publication
Date de publication:
18 May 2020
18 May 2020
Historique:
entrez:
22
5
2020
pubmed:
22
5
2020
medline:
26
11
2020
Statut:
ppublish
Résumé
Hermansky-Pudlak syndrome (HPS) is a rare autosomal recessive disorder characterized by defective biogenesis of lysosome-related organelles. Clinical manifestations include a bleeding diathesis due to a platelet delta storage pool deficiency, oculocutaneous albinism, inflammatory bowel disease, neutropenia, and pulmonary fibrosis. Ten genes associated with HPS are identified to date, and each gene encodes a protein subunit of either Biogenesis of Lysosome-related Organelles Complex (BLOC)-1, BLOC-2, BLOC-3, or the Adaptor Protein-3 complex. Several genetic variants and phenotypic heterogeneities are reported in individuals with HPS, who generally exhibit easy bruisability and increased bleeding. Desmopressin, pro-coagulants, or platelet transfusion may be used as prophylaxis or treatment for excessive bleeding in patients with HPS. However, response to desmopressin can be variable. Platelets are effective in preventing or treating bleeding in individuals with HPS, but platelets should be transfused judiciously to limit alloimmunization in patients with HPS who are at risk of developing pulmonary fibrosis and may be potential candidates for lung transplantation. The discovery of new genes associated with HPS in people with excessive bleeding and hypopigmentation of unknown etiology may be facilitated by the use of next-generation sequencing or panel-based genetic testing.
Identifiants
pubmed: 32436471
doi: 10.1080/09537104.2019.1663810
pmc: PMC8336198
mid: NIHMS1725972
doi:
Substances chimiques
Antifibrinolytic Agents
0
BLOC1S1 protein, human
0
BLOC1S2 protein, human
0
BLOC1S3 protein, human
0
Carrier Proteins
0
Nerve Tissue Proteins
0
Proteins
0
Tranexamic Acid
6T84R30KC1
Deamino Arginine Vasopressin
ENR1LLB0FP
Aminocaproic Acid
U6F3787206
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
544-547Subventions
Organisme : Intramural NIH HHS
ID : Z01 HG000215
Pays : United States
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