Low-frequency oscillation suppression in dystonia: Implications for adaptive deep brain stimulation.


Journal

Parkinsonism & related disorders
ISSN: 1873-5126
Titre abrégé: Parkinsonism Relat Disord
Pays: England
ID NLM: 9513583

Informations de publication

Date de publication:
10 2020
Historique:
received: 07 04 2020
revised: 18 08 2020
accepted: 21 08 2020
pubmed: 13 9 2020
medline: 3 2 2022
entrez: 12 9 2020
Statut: ppublish

Résumé

Low-frequency oscillations (LFO) detected in the internal globus pallidus of dystonia patients have been identified as a physiomarker for adaptive Deep Brain Stimulation (aDBS), since LFO correlate with dystonic symptoms and are rapidly suppressed by continuous DBS (cDBS). However, it is as yet unclear how LFO should be incorporated as feedback for aDBS. to test the acute effects of aDBS, using the amplitude of short-lived LFO-bursts to titrate stimulation, to explore the immediate effects of cDBS on LFO-modulation and dystonic symptoms, and to investigate whether a difference in the resting-state LFO is present between DBS-naïve patients and patients with chronic DBS. seven patients were assessed during either DBS-implantation (n = 2) or battery replacement surgery (n = 5), and pseudorandomized in three conditions: no stimulation, cDBS, and aDBS. Additionally, resting-state LFP-recordings from patients undergoing battery replacement were compared to those obtained during DBS-implantation; LFP-recordings from a previous cohort of six dystonia patients undergoing DBS-implantation were incorporated into this analysis (total n = 8 newly implanted patients). we corroborated that a mild LFO-suppression rapidly occurs during cDBS. However, no acute changes in clinical symptoms were observed after cDBS or aDBS. Remarkably, we observed that resting-state LFO were significantly lower in patients who had been effectively treated with chronic cDBS compared to those of newly implanted patients, even when stimulation was suspended. our results indicate that LFO-suppression in dystonia, similar to symptom response to cDBS, might be gradual, and remain after stimulation is suspended. Therefore, tracking gradual changes in LFO may be required for aDBS implementation.

Sections du résumé

BACKGROUND
Low-frequency oscillations (LFO) detected in the internal globus pallidus of dystonia patients have been identified as a physiomarker for adaptive Deep Brain Stimulation (aDBS), since LFO correlate with dystonic symptoms and are rapidly suppressed by continuous DBS (cDBS). However, it is as yet unclear how LFO should be incorporated as feedback for aDBS.
OBJECTIVES
to test the acute effects of aDBS, using the amplitude of short-lived LFO-bursts to titrate stimulation, to explore the immediate effects of cDBS on LFO-modulation and dystonic symptoms, and to investigate whether a difference in the resting-state LFO is present between DBS-naïve patients and patients with chronic DBS.
METHODS
seven patients were assessed during either DBS-implantation (n = 2) or battery replacement surgery (n = 5), and pseudorandomized in three conditions: no stimulation, cDBS, and aDBS. Additionally, resting-state LFP-recordings from patients undergoing battery replacement were compared to those obtained during DBS-implantation; LFP-recordings from a previous cohort of six dystonia patients undergoing DBS-implantation were incorporated into this analysis (total n = 8 newly implanted patients).
RESULTS
we corroborated that a mild LFO-suppression rapidly occurs during cDBS. However, no acute changes in clinical symptoms were observed after cDBS or aDBS. Remarkably, we observed that resting-state LFO were significantly lower in patients who had been effectively treated with chronic cDBS compared to those of newly implanted patients, even when stimulation was suspended.
CONCLUSIONS
our results indicate that LFO-suppression in dystonia, similar to symptom response to cDBS, might be gradual, and remain after stimulation is suspended. Therefore, tracking gradual changes in LFO may be required for aDBS implementation.

Identifiants

pubmed: 32919097
pii: S1353-8020(20)30696-9
doi: 10.1016/j.parkreldis.2020.08.030
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

105-109

Informations de copyright

Copyright © 2020 The Authors. Published by Elsevier Ltd.. All rights reserved.

Auteurs

D Piña-Fuentes (D)

University of Groningen, University Medical Center Groningen, Department of Neurosurgery, Groningen, the Netherlands; Expertise Center Movement Disorders Groningen, University of Groningen, University Medical Center Groningen, Department of Neurology, Groningen, the Netherlands; Amsterdam UMC, University of Amsterdam, Department of Neurology, Amsterdam Neuroscience, Meibergdreef 9, Amsterdam, the Netherlands. Electronic address: d.a.i.pina.fuentes@umcg.nl.

M Beudel (M)

Expertise Center Movement Disorders Groningen, University of Groningen, University Medical Center Groningen, Department of Neurology, Groningen, the Netherlands; Amsterdam UMC, University of Amsterdam, Department of Neurology, Amsterdam Neuroscience, Meibergdreef 9, Amsterdam, the Netherlands.

J C Van Zijl (JC)

Expertise Center Movement Disorders Groningen, University of Groningen, University Medical Center Groningen, Department of Neurology, Groningen, the Netherlands.

M E Van Egmond (ME)

Expertise Center Movement Disorders Groningen, University of Groningen, University Medical Center Groningen, Department of Neurology, Groningen, the Netherlands; Ommelander Ziekenhuis Groningen, Department of Neurology, Scheemda, the Netherlands.

D L M Oterdoom (DLM)

University of Groningen, University Medical Center Groningen, Department of Neurosurgery, Groningen, the Netherlands.

J M C Van Dijk (JMC)

University of Groningen, University Medical Center Groningen, Department of Neurosurgery, Groningen, the Netherlands.

M A J Tijssen (MAJ)

Expertise Center Movement Disorders Groningen, University of Groningen, University Medical Center Groningen, Department of Neurology, Groningen, the Netherlands.

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